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Human cerebral organoids as a therapeutic drug screening model for Creutzfeldt–Jakob disease

Scientific reports, 2021-03, Vol.11 (1), p.5165-5165, Article 5165

2021

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Lack of Transmission of Chronic Wasting Disease Prions to Human Cerebral Organoids

Emerging infectious diseases, 2024-06, Vol.30 (6), p.1193-1202

2024

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α-Synuclein seeding activity in duodenum biopsies from Parkinson's disease patients

PLoS pathogens, 2023-06, Vol.19 (6), p.e1011456-e1011456

2023

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Reduced SOD2 expression does not influence prion disease course or pathology in mice

PloS one, 2021-11, Vol.16 (11), p.e0259597-e0259597

2021

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The Prion Protein N1 and N2 Cleavage Fragments Bind to Phosphatidylserine and Phosphatidic Acid; Relevance to Stress-Protection Responses

PloS one, 2015-08, Vol.10 (8), p.e0134680-e0134680

2015

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Neuronal maturation reduces the type I IFN response to orthobunyavirus infection and leads to increased apoptosis of human neurons

Journal of neuroinflammation, 2019-11, Vol.16 (1), p.229-229, Article 229

2019

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Neural cell engraftment therapy for sporadic Creutzfeldt-Jakob disease restores neuroelectrophysiological parameters in a cerebral organoid model

Stem cell research & therapy, 2023-12, Vol.14 (1), p.348-348, Article 348

2023

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Temporary alteration of neuronal network communication is a protective response to redox imbalance that requires GPI-anchored prion protein

Redox biology, 2023-07, Vol.63, p.102733-102733, Article 102733

2023

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Sporadic Creutzfeldt-Jakob disease infected human cerebral organoids retain the original human brain subtype features following transmission to humanized transgenic mice

Acta neuropathologica communications, 2023-02, Vol.11 (1), p.28-28, Article 28

2023

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Neutron Reflectometry Studies Define Prion Protein N-terminal Peptide Membrane Binding

Biophysical journal, 2014-11, Vol.107 (10), p.2313-2324

2014

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The lymphatic route of TAU

Science translational medicine, 2019-03, Vol.11 (483)

Haigh, Cathryn L.

2019

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Endoproteolytic cleavage as a molecular switch regulating and diversifying prion protein function

Neural regeneration research, 2016-02, Vol.11 (2), p.238-239

2016

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A 3D cell culture approach for studying neuroinflammation

Journal of neuroscience methods, 2021-07, Vol.358, p.109201-109201, Article 109201

2021

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Cellular Analysis of Adult Neural Stem Cells for Investigating Prion Biology

Methods in molecular biology (Clifton, N.J.), 2017, Vol.1658, p.133

Haigh, Cathryn L

2017

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Cytosolic caspases mediate mislocalised SOD2 depletion in an in vitro model of chronic prion infection

Disease models & mechanisms, 2013-07, Vol.6 (4), p.952-963

2013

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A Functional Role for A[beta] in Metal Homeostasis? N-Truncation and High-Affinity Copper Binding

Angewandte Chemie International Edition, 2015-09, Vol.54 (36), p.10460

International ed. in English, 2015

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A PrP EGFR signaling axis controls neural stem cell senescence through modulating cellular energy pathways

The Journal of biological chemistry, 2023-11, Vol.299 (11), p.105319-105319, Article 105319

2023

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Prion protein cleavage fragments regulate adult neural stem cell quiescence through redox modulation of mitochondrial fission and SOD2 expression

Cellular and molecular life sciences : CMLS, 2018-09, Vol.75 (17), p.3231-3249

2018

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Prion subcellular fractionation reveals infectivity spectrum, with a high titre-low PrPres level disparity

Molecular neurodegeneration, 2012-04, Vol.7 (1), p.18-18

2012

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Simplified Murine 3D Neuronal Cultures for Investigating Neuronal Activity and Neurodegeneration

Cell biochemistry and biophysics, 2017-03, Vol.75 (1), p.3-13

2017

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Viruses in neurodegenerative diseases: More than just suspects in crimes

PLoS pathogens, 2022-08, Vol.18 (8), p.e1010670-e1010670

2022

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Cleavage site-directed antibodies reveal the prion protein in humans is shed by ADAM10 at Y226 and associates with misfolded protein deposits in neurodegenerative diseases

Acta neuropathologica, 2024-07, Vol.148 (1), p.2, Article 2

2024

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A Functional Role for Aβ in Metal Homeostasis? N-Truncation and High-Affinity Copper Binding

Angewandte Chemie (International ed.), 2015-09, Vol.54 (36), p.10460-10464

2015

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Cavitation during the protein misfolding cyclic amplification (PMCA) method – The trigger for de novo prion generation?

Biochemical and biophysical research communications, 2015-06, Vol.461 (3), p.494-500

2015

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Regulation of Prion Protein Expression by Noncoding Regions of the Prnp Gene

Journal of molecular biology, 2007-05, Vol.368 (4), p.915-927

2007

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Human cerebral organoids as a therapeutic drug screening model for Creutzfeldt–Jakob disease

Lack of Transmission of Chronic Wasting Disease Prions to Human Cerebral Organoids

α-Synuclein seeding activity in duodenum biopsies from Parkinson's disease patients

Reduced SOD2 expression does not influence prion disease course or pathology in mice

The Prion Protein N1 and N2 Cleavage Fragments Bind to Phosphatidylserine and Phosphatidic Acid; Relevance to Stress-Protection Responses

Neuronal maturation reduces the type I IFN response to orthobunyavirus infection and leads to increased apoptosis of human neurons

Neural cell engraftment therapy for sporadic Creutzfeldt-Jakob disease restores neuroelectrophysiological parameters in a cerebral organoid model

Temporary alteration of neuronal network communication is a protective response to redox imbalance that requires GPI-anchored prion protein

Sporadic Creutzfeldt-Jakob disease infected human cerebral organoids retain the original human brain subtype features following transmission to humanized transgenic mice

Neutron Reflectometry Studies Define Prion Protein N-terminal Peptide Membrane Binding

The lymphatic route of TAU

Endoproteolytic cleavage as a molecular switch regulating and diversifying prion protein function

A 3D cell culture approach for studying neuroinflammation

Cellular Analysis of Adult Neural Stem Cells for Investigating Prion Biology

Cytosolic caspases mediate mislocalised SOD2 depletion in an in vitro model of chronic prion infection

A Functional Role for A[beta] in Metal Homeostasis? N-Truncation and High-Affinity Copper Binding

A PrP EGFR signaling axis controls neural stem cell senescence through modulating cellular energy pathways

Prion protein cleavage fragments regulate adult neural stem cell quiescence through redox modulation of mitochondrial fission and SOD2 expression

Prion subcellular fractionation reveals infectivity spectrum, with a high titre-low PrPres level disparity

Simplified Murine 3D Neuronal Cultures for Investigating Neuronal Activity and Neurodegeneration

Viruses in neurodegenerative diseases: More than just suspects in crimes

Cleavage site-directed antibodies reveal the prion protein in humans is shed by ADAM10 at Y226 and associates with misfolded protein deposits in neurodegenerative diseases

A Functional Role for Aβ in Metal Homeostasis? N-Truncation and High-Affinity Copper Binding

Cavitation during the protein misfolding cyclic amplification (PMCA) method – The trigger for de novo prion generation?

Regulation of Prion Protein Expression by Noncoding Regions of the Prnp Gene

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