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CaM Kinase: Still Inspiring at 40

Neuron (Cambridge, Mass.), 2019-08, Vol.103 (3), p.380-394

2019

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CaMKII regulation in information processing and storage

Trends in neurosciences (Regular ed.), 2012-10, Vol.35 (10), p.607-618

2012

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Role of the CaMKII/NMDA receptor complex in the maintenance of synaptic strength

The Journal of neuroscience, 2011-06, Vol.31 (25), p.9170-9178

2011

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NMDA Receptor Activation Strengthens Weak Electrical Coupling in Mammalian Brain

Neuron (Cambridge, Mass.), 2014-03, Vol.81 (6), p.1375-1388

2014

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Autonomous CaMKII Mediates Both LTP and LTD Using a Mechanism for Differential Substrate Site Selection

Cell reports (Cambridge), 2014-02, Vol.6 (3), p.431-437

2014

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Interaction with the NMDA receptor locks CaMKII in an active conformation

Nature (London), 2001-06, Vol.411 (6839), p.801-805

2001

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Induction of LTP mechanisms in dually innervated dendritic spines

Scientific reports, 2024-07, Vol.14 (1), p.15855-9, Article 15855

2024

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Multiple CaMKII Binding Modes to the Actin Cytoskeleton Revealed by Single-Molecule Imaging

Biophysical journal, 2016-07, Vol.111 (2), p.395-408

2016

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Decreased nitrosylation of CaMKII causes aging-associated impairments in memory and synaptic plasticity in mice

Science signaling, 2023-07, Vol.16 (795), p.eade5892

2023

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Nitric Oxide Induces Ca2+-independent Activity of the Ca2+/Calmodulin-dependent Protein Kinase II (CaMKII)

The Journal of biological chemistry, 2014-07, Vol.289 (28), p.19458-19465

2014

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Excitotoxic glutamate insults block autophagic flux in hippocampal neurons

Brain research, 2014-01, Vol.1542, p.12-19

2014

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The CaMKII/GluN2B Protein Interaction Maintains Synaptic Strength

The Journal of biological chemistry, 2016-07, Vol.291 (31), p.16082-16089

2016

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The CaMKII K42M and K42R mutations are equivalent in suppressing kinase activity and targeting

PloS one, 2020-07, Vol.15 (7), p.e0236478-e0236478

2020

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The CaMKII holoenzyme structure in activation-competent conformations

Nature communications, 2017-06, Vol.8 (1), p.15742-15742, Article 15742

2017

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CaMKII T286 phosphorylation has distinct essential functions in three forms of long-term plasticity

The Journal of biological chemistry, 2022-09, Vol.298 (9), p.102299-102299, Article 102299

2022

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Targeting sphingosine-1-phosphate lyase as an anabolic therapy for bone loss

Nature medicine, 2018-05, Vol.24 (5), p.667

2018

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CaMKII "autonomy" is required for initiating but not for maintaining neuronal long-term information storage

The Journal of neuroscience, 2010-06, Vol.30 (24), p.8214-8220

2010

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DAPK1 Mediates LTD by Making CaMKII/GluN2B Binding LTP Specific

Cell reports (Cambridge), 2017-06, Vol.19 (11), p.2231-2243

2017

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LTP induction by structural rather than enzymatic functions of CaMKII

Nature (London), 2023-09, Vol.621 (7977), p.146

2023

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Stimulating β-adrenergic receptors promotes synaptic potentiation by switching CaMKII movement from LTD to LTP mode

The Journal of biological chemistry, 2023-06, Vol.299 (6), p.104706, Article 104706

2023

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Simultaneous Live Imaging of Multiple Endogenous Proteins Reveals a Mechanism for Alzheimer’s-Related Plasticity Impairment

Cell reports (Cambridge), 2019-04, Vol.27 (3), p.658-665.e4

2019

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Young DAPK1 knockout mice have altered presynaptic function

Journal of neurophysiology, 2021-05, Vol.125 (5), p.1973-1981

2021

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Transition from Reversible to Persistent Binding of CaMKII to Postsynaptic Sites and NR2B

The Journal of neuroscience, 2006-01, Vol.26 (4), p.1164-1174

2006

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Distinct synaptic pools of DAPK1 differentially regulate activity-dependent synaptic CaMKII accumulation

iScience, 2023-05, Vol.26 (5), p.106723-106723, Article 106723

2023

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Calcium/Calmodulin-Dependent Kinase (CaMKII) Inhibition Protects Against Purkinje Cell Damage Following CA/CPR in Mice

Molecular neurobiology, 2020-01, Vol.57 (1), p.150-158

2020

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CaM Kinase: Still Inspiring at 40

CaMKII regulation in information processing and storage

Role of the CaMKII/NMDA receptor complex in the maintenance of synaptic strength

NMDA Receptor Activation Strengthens Weak Electrical Coupling in Mammalian Brain

Autonomous CaMKII Mediates Both LTP and LTD Using a Mechanism for Differential Substrate Site Selection

Interaction with the NMDA receptor locks CaMKII in an active conformation

Induction of LTP mechanisms in dually innervated dendritic spines

Multiple CaMKII Binding Modes to the Actin Cytoskeleton Revealed by Single-Molecule Imaging

Decreased nitrosylation of CaMKII causes aging-associated impairments in memory and synaptic plasticity in mice

Nitric Oxide Induces Ca2+-independent Activity of the Ca2+/Calmodulin-dependent Protein Kinase II (CaMKII)

Excitotoxic glutamate insults block autophagic flux in hippocampal neurons

The CaMKII/GluN2B Protein Interaction Maintains Synaptic Strength

The CaMKII K42M and K42R mutations are equivalent in suppressing kinase activity and targeting

The CaMKII holoenzyme structure in activation-competent conformations

CaMKII T286 phosphorylation has distinct essential functions in three forms of long-term plasticity

Targeting sphingosine-1-phosphate lyase as an anabolic therapy for bone loss

CaMKII "autonomy" is required for initiating but not for maintaining neuronal long-term information storage

DAPK1 Mediates LTD by Making CaMKII/GluN2B Binding LTP Specific

LTP induction by structural rather than enzymatic functions of CaMKII

Stimulating β-adrenergic receptors promotes synaptic potentiation by switching CaMKII movement from LTD to LTP mode

Simultaneous Live Imaging of Multiple Endogenous Proteins Reveals a Mechanism for Alzheimer’s-Related Plasticity Impairment

Young DAPK1 knockout mice have altered presynaptic function

Transition from Reversible to Persistent Binding of CaMKII to Postsynaptic Sites and NR2B

Distinct synaptic pools of DAPK1 differentially regulate activity-dependent synaptic CaMKII accumulation

Calcium/Calmodulin-Dependent Kinase (CaMKII) Inhibition Protects Against Purkinje Cell Damage Following CA/CPR in Mice

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