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Urinary heparanase activity in patients with Type 1 and Type 2 diabetes

Nephrology, dialysis, transplantation, 2012-07, Vol.27 (7), p.2853-2861

2012

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Modulation of heparan sulfate in the glomerular endothelial glycocalyx decreases leukocyte influx during experimental glomerulonephritis

Kidney international, 2014-11, Vol.86 (5), p.932-942

2014

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Differential Expression of Specific Dermatan Sulfate Domains in Renal Pathology

PloS one, 2015-08, Vol.10 (9), p.e0134946-e0134946

2015

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The role of heparanase and the endothelial glycocalyx in the development of proteinuria

Nephrology, dialysis, transplantation, 2014-01, Vol.29 (1), p.49-55

2014

Volltextzugriff (PDF)

Cathepsin L is crucial for the development of early experimental diabetic nephropathy

Kidney international, 2016-11, Vol.90 (5), p.1012-1022

2016

Volltextzugriff (PDF)

Interleukin-6 is essential for glomerular immunoglobulin A deposition and the development of renal pathology in Cd37-deficient mice

Kidney international, 2018-06, Vol.93 (6), p.1356-1366

2018

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Glomerular endothelial glycocalyx-derived heparan sulfate inhibits glomerular leukocyte influx and attenuates experimental glomerulonephritis

Frontiers in molecular biosciences, 2023-06, Vol.10, p.1177560-1177560

2023

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Chemokine cooperativity is caused by competitive glycosaminoglycan binding

The Journal of immunology (1950), 2014-04, Vol.192 (8), p.3908-3914

2014

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In vivo degradation of heparan sulfates in the glomerular basement membrane does not result in proteinuria

Journal of the American Society of Nephrology, 2007-03, Vol.18 (3), p.823-832

2007

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Heparan sulfate proteoglycans in glomerular inflammation

Kidney international, 2004-03, Vol.65 (3), p.768-785

2004

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Removal of Heparan Sulfate from the Glomerular Basement Membrane Blocks Protein Passage

Journal of the American Society of Nephrology, 2007-12, Vol.18 (12), p.3119-3127

2007

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New approaches to the treatment of dense deposit disease

Journal of the American Society of Nephrology, 2007-09, Vol.18 (9), p.2447-2456

2007

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1,25-Vitamin D3 Deficiency Induces Albuminuria

The American journal of pathology, 2016-04, Vol.186 (4), p.794-804

2016

Volltextzugriff (PDF)

The role of heparan sulfate as determining pathogenic factor in complement factor H-associated diseases

Molecular immunology, 2015-02, Vol.63 (2), p.203-208

2015

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Adult and paediatric patients with minimal change nephrotic syndrome show no major alterations in glomerular expression of sulphated heparan sulphate domains

Nephrology, dialysis, transplantation, 2007-10, Vol.22 (10), p.2886-2893

2007

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Regulation of glomerular heparanase expression by aldosterone, angiotensin II and reactive oxygen species

Nephrology, dialysis, transplantation, 2009-09, Vol.24 (9), p.2637-2645

2009

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Are primed polymorphonuclear leukocytes contributors to the high heparanase levels in hemodialysis patients?

American journal of physiology. Heart and circulatory physiology, 2008-02, Vol.294 (2), p.H651-H658

2008

Volltextzugriff (PDF)

Localization and functional characterization of glycosaminoglycan domains in the normal human kidney as revealed by phage display-derived single chain antibodies

Journal of the American Society of Nephrology, 2005-05, Vol.16 (5), p.1279-1288

2005

Volltextzugriff (PDF)

Aberrant Heparan Sulfate Profile in the Human Diabetic Kidney Offers New Clues for Therapeutic Glycomimetics

American journal of kidney diseases, 2006-08, Vol.48 (2), p.250-261

2006

Volltextzugriff (PDF)

Heparanase Is Essential for the Development of Acute Experimental Glomerulonephritis

The American journal of pathology, 2016-04, Vol.186 (4), p.805-815

2016

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Expression of glomerular heparan sulphate domains in murine and human lupus nephritis

Nephrology, dialysis, transplantation, 2007-08, Vol.22 (8), p.2416-2416, Article 2416

2007

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Removal of heparan sulfate from glomerular endothelial cells does not abolish FH19-20 binding

Molecular immunology, 2009-09, Vol.46 (14), p.2840-2840

2009

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Are primed polymorphonuclear leukocytes contributors to the high heparanase levels in hemodialysis patients?

American Journal of Physiology: Cell Physiology, 2008-02, Vol.294 (2), p.H651-H658

2008

Volltextzugriff (PDF)

Are primed polymorphonuclear leukocytes contributors to the high heparanase levels in hemodialysis patients?

American journal of physiology. Heart and circulatory physiology, 2008, Vol.63 (2)

2008

Volltextzugriff (PDF)

Disruption of glomerular basement membrane charge through podocyte-specific mutation of agrin does not alter glomerular permselectivity. Commentary. Author's reply

The American journal of pathology, 2007, Vol.171 (1)

2007

Volltextzugriff (PDF)

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Urinary heparanase activity in patients with Type 1 and Type 2 diabetes

Modulation of heparan sulfate in the glomerular endothelial glycocalyx decreases leukocyte influx during experimental glomerulonephritis

Differential Expression of Specific Dermatan Sulfate Domains in Renal Pathology

The role of heparanase and the endothelial glycocalyx in the development of proteinuria

Cathepsin L is crucial for the development of early experimental diabetic nephropathy

Interleukin-6 is essential for glomerular immunoglobulin A deposition and the development of renal pathology in Cd37-deficient mice

Glomerular endothelial glycocalyx-derived heparan sulfate inhibits glomerular leukocyte influx and attenuates experimental glomerulonephritis

Chemokine cooperativity is caused by competitive glycosaminoglycan binding

In vivo degradation of heparan sulfates in the glomerular basement membrane does not result in proteinuria

Heparan sulfate proteoglycans in glomerular inflammation

Removal of Heparan Sulfate from the Glomerular Basement Membrane Blocks Protein Passage

New approaches to the treatment of dense deposit disease

1,25-Vitamin D3 Deficiency Induces Albuminuria

The role of heparan sulfate as determining pathogenic factor in complement factor H-associated diseases

Adult and paediatric patients with minimal change nephrotic syndrome show no major alterations in glomerular expression of sulphated heparan sulphate domains

Regulation of glomerular heparanase expression by aldosterone, angiotensin II and reactive oxygen species

Are primed polymorphonuclear leukocytes contributors to the high heparanase levels in hemodialysis patients?

Localization and functional characterization of glycosaminoglycan domains in the normal human kidney as revealed by phage display-derived single chain antibodies

Aberrant Heparan Sulfate Profile in the Human Diabetic Kidney Offers New Clues for Therapeutic Glycomimetics

Heparanase Is Essential for the Development of Acute Experimental Glomerulonephritis

Expression of glomerular heparan sulphate domains in murine and human lupus nephritis

Removal of heparan sulfate from glomerular endothelial cells does not abolish FH19-20 binding

Are primed polymorphonuclear leukocytes contributors to the high heparanase levels in hemodialysis patients?

Are primed polymorphonuclear leukocytes contributors to the high heparanase levels in hemodialysis patients?

Disruption of glomerular basement membrane charge through podocyte-specific mutation of agrin does not alter glomerular permselectivity. Commentary. Author's reply

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