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The actions of relaxin on the human cardiovascular system

British journal of pharmacology, 2017-05, Vol.174 (10), p.933-949

2017

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β‐Adrenoceptor activation affects galectin‐3 as a biomarker and therapeutic target in heart disease

British journal of pharmacology, 2019-07, Vol.176 (14), p.2449-2464

2019

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Transitional changes in the CRP structure lead to the exposure of proinflammatory binding sites

Nature communications, 2017-01, Vol.8 (1), p.14188-14188, Article 14188

2017

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Improving the quality of preclinical research echocardiography: observations, training, and guidelines for measurement

American journal of physiology. Heart and circulatory physiology, 2018-07, Vol.315 (1), p.H58-H70

2018

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HDAC inhibition attenuates cardiac hypertrophy by acetylation and deacetylation of target genes

Epigenetics, 2015-05, Vol.10 (5), p.418-430

2015

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Relaxin mitigates microvascular damage and inflammation following cardiac ischemia–reperfusion

Basic research in cardiology, 2019-07, Vol.114 (4), p.30-15, Article 30

2019

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Transcriptomic Analysis of Dysregulated Genes of the nDNA-mtDNA Axis in a Mouse Model of Dilated Cardiomyopathy

Frontiers in genetics, 2022-06, Vol.13, p.921610-921610

2022

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Cardiac rupture complicating acute myocardial infarction: the clinical features from an observational study and animal experiment

BMC cardiovascular disorders, 2020-09, Vol.20 (1), p.409-409, Article 409

2020

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Manipulation of the gut microbiota by the use of prebiotic fibre does not override a genetic predisposition to heart failure

Scientific reports, 2020-10, Vol.10 (1), p.17919-17919, Article 17919

2020

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Therapeutic Effects of Serelaxin in Acute Heart Failure: Necessity for Bilateral Research Translation

Circulation Journal, 2014, Vol.78(3), pp.542-552

2014

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Cardiovascular effects of relaxin: from basic science to clinical therapy

Nature reviews cardiology, 2010-01, Vol.7 (1), p.48-58

2010

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Endogenous Annexin-A1 Regulates Haematopoietic Stem Cell Mobilisation and Inflammatory Response Post Myocardial Infarction in Mice In Vivo

Scientific reports, 2017-11, Vol.7 (1), p.16615-14, Article 16615

2017

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K Ca 3.1 Channels Promote Cardiac Fibrosis Through Mediating Inflammation and Differentiation of Monocytes Into Myofibroblasts in Angiotensin II -Treated Rats

Journal of the American Heart Association, 2019-01, Vol.8 (1), p.e010418

2019

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KCa3.1 channel mediates inflammatory signaling of pancreatic β cells and progression of type 2 diabetes mellitus

The FASEB journal, 2019-12, Vol.33 (12), p.14760-14771

2019

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Role of MIF in myocardial ischaemia and infarction: insight from recent clinical and experimental findings

Clinical science (1979), 2014-08, Vol.127 (3), p.149-161

2014

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Cardioprotective Actions of the Annexin-A1 N-Terminal Peptide, Ac2-26, Against Myocardial Infarction

Frontiers in pharmacology, 2019-04, Vol.10, p.269-269

2019

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N‐acetylcysteine attenuates the development of cardiac fibrosis and remodeling in a mouse model of heart failure

Physiological reports, 2016-04, Vol.4 (7), p.e12757-n/a

2016

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Up-Regulating Relaxin Expression by G-Quadruplex Interactive Ligand to Achieve Antifibrotic Action

Endocrinology (Philadelphia), 2012-08, Vol.153 (8), p.3692-3700

2012

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Relaxin Therapy Reverses Large Artery Remodeling and Improves Arterial Compliance in Senescent Spontaneously Hypertensive Rats

Hypertension (Dallas, Tex. 1979), 2010-05, Vol.55 (5), p.1260-1266

2010

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Correction to: Cardiac rupture complicating acute myocardial infarction: the clinical features from an observational study and animal experiment

BMC cardiovascular disorders, 2020-10, Vol.20 (1), p.1-441, Article 441

2020

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Relaxin Reverses Cardiac and Renal Fibrosis in Spontaneously Hypertensive Rats

Hypertension (Dallas, Tex. 1979), 2005-08, Vol.46 (2), p.412-418

2005

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AGEs-RAGE-KCa3.1 pathway mediates palmitic acid-induced migration of PBMCs from patients with type 2 diabetes

Heliyon, 2023-04, Vol.9 (4), p.e14823-e14823, Article e14823

2023

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Age-Related Differential Structural and Transcriptomic Responses in the Hypertensive Heart

Frontiers in physiology, 2018-07, Vol.9, p.817-817

2018

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Sex dimorphism in cardiac pathophysiology: experimental findings, hormonal mechanisms, and molecular mechanisms

Pharmacology & therapeutics (Oxford), 2006-08, Vol.111 (2), p.434-475

2006

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Towards better definition, quantification and treatment of fibrosis in heart failure. A scientific roadmap by the Committee of Translational Research of the Heart Failure Association (HFA) of the European Society of Cardiology

The actions of relaxin on the human cardiovascular system

β‐Adrenoceptor activation affects galectin‐3 as a biomarker and therapeutic target in heart disease

Transitional changes in the CRP structure lead to the exposure of proinflammatory binding sites

Improving the quality of preclinical research echocardiography: observations, training, and guidelines for measurement

HDAC inhibition attenuates cardiac hypertrophy by acetylation and deacetylation of target genes

Relaxin mitigates microvascular damage and inflammation following cardiac ischemia–reperfusion

Transcriptomic Analysis of Dysregulated Genes of the nDNA-mtDNA Axis in a Mouse Model of Dilated Cardiomyopathy

Cardiac rupture complicating acute myocardial infarction: the clinical features from an observational study and animal experiment

Manipulation of the gut microbiota by the use of prebiotic fibre does not override a genetic predisposition to heart failure

Therapeutic Effects of Serelaxin in Acute Heart Failure: Necessity for Bilateral Research Translation

Cardiovascular effects of relaxin: from basic science to clinical therapy

Endogenous Annexin-A1 Regulates Haematopoietic Stem Cell Mobilisation and Inflammatory Response Post Myocardial Infarction in Mice In Vivo

K Ca 3.1 Channels Promote Cardiac Fibrosis Through Mediating Inflammation and Differentiation of Monocytes Into Myofibroblasts in Angiotensin II -Treated Rats

KCa3.1 channel mediates inflammatory signaling of pancreatic β cells and progression of type 2 diabetes mellitus

Role of MIF in myocardial ischaemia and infarction: insight from recent clinical and experimental findings

Cardioprotective Actions of the Annexin-A1 N-Terminal Peptide, Ac2-26, Against Myocardial Infarction

N‐acetylcysteine attenuates the development of cardiac fibrosis and remodeling in a mouse model of heart failure

Up-Regulating Relaxin Expression by G-Quadruplex Interactive Ligand to Achieve Antifibrotic Action

Relaxin Therapy Reverses Large Artery Remodeling and Improves Arterial Compliance in Senescent Spontaneously Hypertensive Rats

Correction to: Cardiac rupture complicating acute myocardial infarction: the clinical features from an observational study and animal experiment

Relaxin Reverses Cardiac and Renal Fibrosis in Spontaneously Hypertensive Rats

AGEs-RAGE-KCa3.1 pathway mediates palmitic acid-induced migration of PBMCs from patients with type 2 diabetes

Age-Related Differential Structural and Transcriptomic Responses in the Hypertensive Heart

Sex dimorphism in cardiac pathophysiology: experimental findings, hormonal mechanisms, and molecular mechanisms

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