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Phosphorylated Neurofilament Heavy Chain in the Cerebrospinal Fluid Is a Suitable Biomarker of Acute and Chronic Blast-Induced Traumatic Brain Injury

Journal of neurotrauma, 2021-10, Vol.38 (20), p.2801

2021

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Blast-induced hearing impairment in rats is associated with structural and molecular changes of the inner ear

Scientific reports, 2020-06, Vol.10 (1), p.10652-10652, Article 10652

2020

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Repeated Low-Level Blast Acutely Alters Brain Cytokines, Neurovascular Proteins, Mechanotransduction, and Neurodegenerative Markers in a Rat Model

Frontiers in cellular neuroscience, 2021-02, Vol.15, p.636707-636707

2021

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Brief isoflurane administration as an adjunct treatment to control organophosphate-induced convulsions and neuropathology

Frontiers in pharmacology, 2023-12, Vol.14, p.1293280-1293280

2023

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Blast Exposure Induces Acute Alterations in Circadian Clock Genes in the Hypothalamus and Pineal Gland in Rats: An Exploratory Study

Neurotrauma reports, 2023-08, Vol.4 (1), p.586-597

2023

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N-Acetylaspartate reductions in brain injury: impact on post-injury neuroenergetics, lipid synthesis, and protein acetylation

Frontiers in neuroenergetics, 2013-12, Vol.5, p.11-11

2013

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Blast Exposure Leads to Accelerated Cellular Senescence in the Rat Brain

Frontiers in neurology, 2020-05, Vol.11, p.438-438

2020

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Blast Exposure Dysregulates Nighttime Melatonin Synthesis and Signaling in the Pineal Gland: A Potential Mechanism of Blast-Induced Sleep Disruptions

Brain sciences, 2022-10, Vol.12 (10), p.1340

2022

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Blast Exposure Causes Long-Term Degeneration of Neuronal Cytoskeletal Elements in the Cochlear Nucleus: A Potential Mechanism for Chronic Auditory Dysfunctions

Frontiers in neurology, 2021-03, Vol.12, p.652190-652190

2021

Volltextzugriff (PDF)

The Neurobehavioral Effects of Buprenorphine and Meloxicam on a Blast-Induced Traumatic Brain Injury Model in the Rat

Frontiers in neurology, 2021-10, Vol.12, p.746370-746370

2021

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Antibodies Against Lysophosphatidic Acid Protect Against Blast-Induced Ocular Injuries

Frontiers in neurology, 2020-12, Vol.11, p.611816-611816

2020

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Nuclear-cytoplasmic localization of acetyl coenzyme a synthetase-1 in the rat brain

Journal of comparative neurology (1911), 2010-08, Vol.518 (15), p.2952-2977

2010

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Increasing N-acetylaspartate in the Brain during Postnatal Myelination Does Not Cause the CNS Pathologies of Canavan Disease

Frontiers in molecular neuroscience, 2017-06, Vol.10, p.161-161

2017

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Metabolic acetate therapy for the treatment of traumatic brain injury

Journal of neurotrauma, 2010-01, Vol.27 (1), p.293

2010

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Metabolic acetate therapy improves phenotype in the tremor rat model of Canavan disease

Journal of inherited metabolic disease, 2010-06, Vol.33 (3), p.195-210

2010

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Evidence for mitochondrial and cytoplasmic N-acetylaspartate synthesis in SH-SY5Y neuroblastoma cells

Neurochemistry international, 2009-09, Vol.55 (4), p.219-225

2009

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Extracellular cyclophilin A protects against blast-induced neuronal injury

Neuroscience research, 2013-05, Vol.76 (1-2), p.98-100

2013

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Regulation of N‐acetylaspartate and N‐acetylaspartylglutamate biosynthesis by protein kinase activators

Journal of neurochemistry, 2006-09, Vol.98 (6), p.2034-2042

2006

Volltextzugriff (PDF)

Antipsychotic drugs increase N‐acetylaspartate and N‐acetylaspartylglutamate in SH‐SY5Y human neuroblastoma cells

Journal of neurochemistry, 2008-08, Vol.106 (4), p.1669-1680

2008

Volltextzugriff (PDF)

N-Acetylaspartate in the CNS: from neurodiagnostics to neurobiology

Progress in neurobiology, 2007-02, Vol.81 (2), p.89-131

2007

Volltextzugriff (PDF)

Repeated Mild Concussive Events Heighten the Vulnerability of Brain to Blast Exposure

Journal of neurotrauma, 2024-04, Vol.41 (7-8), p.1000

2024

Volltextzugriff (PDF)

Upregulation of multiple toll-like receptors in ferret brain after blast exposure: Potential targets for treatment

Neuroscience letters, 2023-07, Vol.810, p.137364-137364, Article 137364

2023

Volltextzugriff (PDF)

Defective methionine metabolism in the brain after repeated blast exposures might contribute to increased oxidative stress

Neurochemistry international, 2018-01, Vol.112, p.234-238

2018

Volltextzugriff (PDF)

Cerebrospinal Fluid Chemokine (C-C Motif) Ligand 2 Is an Early-Response Biomarker for Blast-Overpressure-Wave-Induced Neurotrauma in Rats

Journal of neurotrauma, 2017-02, Vol.34 (4), p.952

2017

Volltextzugriff (PDF)

Acute decrease in alkaline phosphatase after brain injury: A potential mechanism for tauopathy

Neuroscience letters, 2015-11, Vol.609, p.152-158

2015

Volltextzugriff (PDF)

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Phosphorylated Neurofilament Heavy Chain in the Cerebrospinal Fluid Is a Suitable Biomarker of Acute and Chronic Blast-Induced Traumatic Brain Injury

Blast-induced hearing impairment in rats is associated with structural and molecular changes of the inner ear

Repeated Low-Level Blast Acutely Alters Brain Cytokines, Neurovascular Proteins, Mechanotransduction, and Neurodegenerative Markers in a Rat Model

Brief isoflurane administration as an adjunct treatment to control organophosphate-induced convulsions and neuropathology

Blast Exposure Induces Acute Alterations in Circadian Clock Genes in the Hypothalamus and Pineal Gland in Rats: An Exploratory Study

N-Acetylaspartate reductions in brain injury: impact on post-injury neuroenergetics, lipid synthesis, and protein acetylation

Blast Exposure Leads to Accelerated Cellular Senescence in the Rat Brain

Blast Exposure Dysregulates Nighttime Melatonin Synthesis and Signaling in the Pineal Gland: A Potential Mechanism of Blast-Induced Sleep Disruptions

Blast Exposure Causes Long-Term Degeneration of Neuronal Cytoskeletal Elements in the Cochlear Nucleus: A Potential Mechanism for Chronic Auditory Dysfunctions

The Neurobehavioral Effects of Buprenorphine and Meloxicam on a Blast-Induced Traumatic Brain Injury Model in the Rat

Antibodies Against Lysophosphatidic Acid Protect Against Blast-Induced Ocular Injuries

Nuclear-cytoplasmic localization of acetyl coenzyme a synthetase-1 in the rat brain

Increasing N-acetylaspartate in the Brain during Postnatal Myelination Does Not Cause the CNS Pathologies of Canavan Disease

Metabolic acetate therapy for the treatment of traumatic brain injury

Metabolic acetate therapy improves phenotype in the tremor rat model of Canavan disease

Evidence for mitochondrial and cytoplasmic N-acetylaspartate synthesis in SH-SY5Y neuroblastoma cells

Extracellular cyclophilin A protects against blast-induced neuronal injury

Regulation of N‐acetylaspartate and N‐acetylaspartylglutamate biosynthesis by protein kinase activators

Antipsychotic drugs increase N‐acetylaspartate and N‐acetylaspartylglutamate in SH‐SY5Y human neuroblastoma cells

N-Acetylaspartate in the CNS: from neurodiagnostics to neurobiology

Repeated Mild Concussive Events Heighten the Vulnerability of Brain to Blast Exposure

Upregulation of multiple toll-like receptors in ferret brain after blast exposure: Potential targets for treatment

Defective methionine metabolism in the brain after repeated blast exposures might contribute to increased oxidative stress

Cerebrospinal Fluid Chemokine (C-C Motif) Ligand 2 Is an Early-Response Biomarker for Blast-Overpressure-Wave-Induced Neurotrauma in Rats

Acute decrease in alkaline phosphatase after brain injury: A potential mechanism for tauopathy

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