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Direct Measurement of Free Radical Generation Following Reperfusion of Ischemic Myocardium

Proceedings of the National Academy of Sciences - PNAS, 1987-03, Vol.84 (5), p.1404-1407

1987

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Dissociation between left ventricular untwisting and filling : accentuation by catecholamines

Circulation (New York, N.Y.), 1992-04, Vol.85 (4), p.1572-1581

1992

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Potential cost-effectiveness of public access defibrillation in the United States

Circulation (New York, N.Y.), 1998-04, Vol.97 (13), p.1315-1320

1998

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Soluble Human Complement Receptor Type 1: In Vivo Inhibitor of Complement Suppressing Post-Ischemic Myocardial Inflammation and Necrosis

Science (American Association for the Advancement of Science), 1990-07, Vol.249 (4965), p.146-151

1990

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American Heart Association Report on the Second Public access Defibrillation Conference, april 17-19,1997

Circulation, 1998-04, Vol.97 (13), p.1309-1314

1998

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Cellular mechanisms of myocardial infarct expansion

Circulation (New York, N.Y.), 1988-07, Vol.78 (1), p.186-201

1988

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Evaluation of the role of polymorphonuclear leukocytes on contractile function in myocardial reperfusion injury : evidence for plasma-mediated leukocyte activation

Circulation (New York, N.Y.), 1993-02, Vol.87 (2), p.536-546

1993

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Recombinant superoxide dismutase reduces oxygen free radical concentrations in reperfused myocardium

The Journal of clinical investigation, 1987-12, Vol.80 (6), p.1728-1734

1987

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Evidence for a reversible oxygen radical―mediated component of reperfusion injury: reduction by recombinant human superoxide dismutase administered at the time of reflow

Circulation (New York, N.Y.), 1987, Vol.75 (1), p.282-291

1987

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Soluble complement receptor type 1 inhibits the complement pathway and prevents contractile failure in the postischemic heart : evidence that complement activation is required for neutrophil-mediated reperfusion injury

Circulation (New York, N.Y.), 1993-12, Vol.88 (6), p.2812-2826

1993

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The use of left ventricular end-ejection pressure and peak pressure in the estimation of the end-systolic pressure-volume relationship

Circulation (New York, N.Y.), 1984-12, Vol.70 (6), p.1057-1065

1984

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Silent ischemia predicts infarction and death during 2 year follow-up of unstable angina

Journal of the American College of Cardiology, 1987-10, Vol.10 (4), p.756-760

1987

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Quantification and validation of left ventricular wall thickening by a three-dimensional volume element magnetic resonance imaging approach

Circulation (New York, N.Y.), 1990, Vol.81 (1), p.297-307

1990

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Impaired thickening of nonischemic myocardium during acute regional ischemia in the dog

Circulation (New York, N.Y.), 1985-05, Vol.71 (5), p.1048-1059

1985

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Localization of regional myocardial ischemia by recording of monophasic action potentials

Circulation (New York, N.Y.), 1984-03, Vol.69 (3), p.593-604

1984

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Mechanisms precipitating acute cardiac events: Review and recommendations of an NHLBI workshop

Circulation (New York, N.Y.), 1997-11, Vol.96 (9), p.3233-3239

1997

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Intermittent Coronary Sinus Occlusion After Coronary Arterial Ligation Results in Venous Retroperfusion

Circulation research, 1989-09, Vol.65 (3), p.695-707

1989

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A randomized trial of intravenous tissue plasminogen activator for acute myocardial infarction with subsequent randomization to elective coronary angioplasty

The New England journal of medicine, 1987-12, Vol.317 (26), p.1613-1618

1987

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Disproportionate epicardial dilation after transmural infarction of the canine left ventricle: Acute and chronic differences

Journal of the American College of Cardiology, 1988-01, Vol.11 (1), p.177-185

1988

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New approaches to CPR. Four hands, a plunger, or a vest

JAMA : the journal of the American Medical Association, 1992-06, Vol.267 (21), p.2940

1992

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Mechanism of early contractile failure during hypoxia in intact ferret heart: evidence for modulation of maximal Ca2+-activated force by inorganic phosphate

Circulation research, 1986-09, Vol.59 (3), p.270-282

1986

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Intermittent coronary sinus occlusion in dogs: Reduction of infarct size 10 days after reperfusion

Journal of the American College of Cardiology, 1987-05, Vol.9 (5), p.1075-1081

1987

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Maximal Ca2+-Activated Force Elicited by Tetanization Ferret Papillary Muscle and Whole Heart: Mechanism and Characteristics of Steady Contractile Activation in Intact Myocardium

Circulation research, 1986-09, Vol.59 (3), p.262-269

1986

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Toward an understanding of the molecular basis of cardiomyopathies

Journal of the American College of Cardiology, 1987-11, Vol.10 (5), p.1135-1138

1987

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Exploring myocardial ischemia: silent and symptomatic. Symposium, Washington DC November 9, 1985

The American journal of medicine, 1986-04, Vol.80 (4C), p.1-55

WEISFELDT, M. L. Ed

1986

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Direct Measurement of Free Radical Generation Following Reperfusion of Ischemic Myocardium

Dissociation between left ventricular untwisting and filling : accentuation by catecholamines

Potential cost-effectiveness of public access defibrillation in the United States

Soluble Human Complement Receptor Type 1: In Vivo Inhibitor of Complement Suppressing Post-Ischemic Myocardial Inflammation and Necrosis

American Heart Association Report on the Second Public access Defibrillation Conference, april 17-19,1997

Cellular mechanisms of myocardial infarct expansion

Evaluation of the role of polymorphonuclear leukocytes on contractile function in myocardial reperfusion injury : evidence for plasma-mediated leukocyte activation

Recombinant superoxide dismutase reduces oxygen free radical concentrations in reperfused myocardium

Evidence for a reversible oxygen radical―mediated component of reperfusion injury: reduction by recombinant human superoxide dismutase administered at the time of reflow

Soluble complement receptor type 1 inhibits the complement pathway and prevents contractile failure in the postischemic heart : evidence that complement activation is required for neutrophil-mediated reperfusion injury

The use of left ventricular end-ejection pressure and peak pressure in the estimation of the end-systolic pressure-volume relationship

Silent ischemia predicts infarction and death during 2 year follow-up of unstable angina

Quantification and validation of left ventricular wall thickening by a three-dimensional volume element magnetic resonance imaging approach

Impaired thickening of nonischemic myocardium during acute regional ischemia in the dog

Localization of regional myocardial ischemia by recording of monophasic action potentials

Mechanisms precipitating acute cardiac events: Review and recommendations of an NHLBI workshop

Intermittent Coronary Sinus Occlusion After Coronary Arterial Ligation Results in Venous Retroperfusion

A randomized trial of intravenous tissue plasminogen activator for acute myocardial infarction with subsequent randomization to elective coronary angioplasty

Disproportionate epicardial dilation after transmural infarction of the canine left ventricle: Acute and chronic differences

New approaches to CPR. Four hands, a plunger, or a vest

Mechanism of early contractile failure during hypoxia in intact ferret heart: evidence for modulation of maximal Ca2+-activated force by inorganic phosphate

Intermittent coronary sinus occlusion in dogs: Reduction of infarct size 10 days after reperfusion

Maximal Ca2+-Activated Force Elicited by Tetanization Ferret Papillary Muscle and Whole Heart: Mechanism and Characteristics of Steady Contractile Activation in Intact Myocardium

Toward an understanding of the molecular basis of cardiomyopathies

Exploring myocardial ischemia: silent and symptomatic. Symposium, Washington DC November 9, 1985

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