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Exploring the links between cancer and placenta development

Open biology, 2018-06, Vol.8 (6)

2018

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SHP2 is required for growth of KRAS-mutant non-small-cell lung cancer in vivo

Nature medicine, 2018-07, Vol.24 (7), p.961-967

2018

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The road to resistance: EGFR mutation and cetuximab

Nature medicine, 2012-02, Vol.18 (2), p.199-200

2012

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Higher metastatic efficiency of KRas G12V than KRas G13D in a colorectal cancer model

The FASEB journal, 2015-02, Vol.29 (2), p.464-476

2015

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Preclinical models for precision oncology

Biochimica et biophysica acta. Reviews on cancer, 2018-12, Vol.1870 (2), p.239-246

2018

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Oncogenes and angiogenesis: a way to personalize anti-angiogenic therapy?

Cellular and molecular life sciences : CMLS, 2013-11, Vol.70 (21), p.4131-4140

2013

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A multifunctional docking site mediates signaling and transformation by the hepatocyte growth factor/scatter factor receptor family

Cell, 1994-04, Vol.77 (2), p.261-271

1994

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The EGFR-specific antibody cetuximab combined with chemotherapy triggers immunogenic cell death

Nature medicine, 2016-06, Vol.22 (6), p.624-631

2016

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Toward understanding and exploiting tumor heterogeneity

Nature medicine, 2015-08, Vol.21 (8), p.846-853

2015

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Mutational signatures of colorectal cancers according to distinct computational workflows

Briefings in bioinformatics, 2024-05, Vol.25 (4)

2024

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Biological Activation of pro-HGF (Hepatocyte Growth Factor) by Urokinase Is Controlled by a Stoichiometric Reaction (∗)

The Journal of biological chemistry, 1995-01, Vol.270 (2), p.603-611

1995

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Restoring PUMA induction overcomes KRAS-mediated resistance to anti-EGFR antibodies in colorectal cancer

Oncogene, 2018-08, Vol.37 (33), p.4599-4610

2018

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A Peptide Representing the Carboxyl-terminal Tail of the Met Receptor Inhibits Kinase Activity and Invasive Growth

The Journal of biological chemistry, 1999-10, Vol.274 (41), p.29274-29281

1999

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Loss of the exon encoding the juxtamembrane domain is essential for the oncogenic activation of TPR-MET

Oncogene, 1999-07, Vol.18 (29), p.4275-4281

1999

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Selective cytotoxicity of a bicyclic Ras inhibitor in cancer cells expressing K-RasG13D

Biochemical and biophysical research communications, 2009-09, Vol.386 (4), p.593-597

2009

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HGF receptor associates with the anti‐apoptotic protein BAG‐1 and prevents cell death

The EMBO journal, 1996-11, Vol.15 (22), p.6205-6212

1996

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Identification of cancer genes by mutational profiling of tumor genomes

FEBS letters, 2005-03, Vol.579 (8), p.1884-1890

2005

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Scatter factor and hepatocyte growth factor are indistinguishable ligands for the MET receptor

The EMBO journal, 1991-10, Vol.10 (10), p.2867-2878

1991

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Mutant Met-mediated transformation is ligand-dependent and can be inhibited by HGF antagonists

Oncogene, 1999-09, Vol.18 (37), p.5221-5231

1999

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Gab1 coupling to the HGF/Met receptor multifunctional docking site requires binding of Grb2 and correlates with the transforming potential

Oncogene, 1997-12, Vol.15 (25), p.3103-3111

1997

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Exploring the links between cancer and placenta development

SHP2 is required for growth of KRAS-mutant non-small-cell lung cancer in vivo

Tumor cells can follow distinct evolutionary paths to become resistant to epidermal growth factor receptor inhibition

Clonal evolution and resistance to EGFR blockade in the blood of colorectal cancer patients

The road to resistance: EGFR mutation and cetuximab

Higher metastatic efficiency of KRas G12V than KRas G13D in a colorectal cancer model

Preclinical models for precision oncology

Oncogenes and angiogenesis: a way to personalize anti-angiogenic therapy?

A multifunctional docking site mediates signaling and transformation by the hepatocyte growth factor/scatter factor receptor family

Circulating tumor DNA to guide rechallenge with panitumumab in metastatic colorectal cancer: the phase 2 CHRONOS trial

The EGFR-specific antibody cetuximab combined with chemotherapy triggers immunogenic cell death

Toward understanding and exploiting tumor heterogeneity

Mutational signatures of colorectal cancers according to distinct computational workflows

Biological Activation of pro-HGF (Hepatocyte Growth Factor) by Urokinase Is Controlled by a Stoichiometric Reaction (∗)

Restoring PUMA induction overcomes KRAS-mediated resistance to anti-EGFR antibodies in colorectal cancer

A Peptide Representing the Carboxyl-terminal Tail of the Met Receptor Inhibits Kinase Activity and Invasive Growth

A Vulnerability of a Subset of Colon Cancers with Potential Clinical Utility

Loss of the exon encoding the juxtamembrane domain is essential for the oncogenic activation of TPR-MET

Selective cytotoxicity of a bicyclic Ras inhibitor in cancer cells expressing K-RasG13D

HGF receptor associates with the anti‐apoptotic protein BAG‐1 and prevents cell death

Clonal evolution and resistance to EGFR blockade in the blood of colorectal cancer patients

Identification of cancer genes by mutational profiling of tumor genomes

Scatter factor and hepatocyte growth factor are indistinguishable ligands for the MET receptor

Mutant Met-mediated transformation is ligand-dependent and can be inhibited by HGF antagonists

Gab1 coupling to the HGF/Met receptor multifunctional docking site requires binding of Grb2 and correlates with the transforming potential

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