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Altered EBV specific immune control in multiple sclerosis

Journal of neuroimmunology, 2024-05, Vol.390, p.578343-578343, Article 578343

Münz, Christian

2024

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The Autophagic Machinery in Viral Exocytosis

Frontiers in microbiology, 2017-02, Vol.8, p.269-269

Münz, Christian

2017

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Epstein-Barr virus as a leading cause of multiple sclerosis: mechanisms and implications

Nature reviews. Neurology, 2023-03, Vol.19 (3), p.160

2023

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Compartmentalization of Total and Virus-Specific Tissue-Resident Memory CD8+ T Cells in Human Lymphoid Organs

PLoS pathogens, 2016-08, Vol.12 (8), p.e1005799-e1005799

2016

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Cytotoxic CD4+ T-cells specific for EBV capsid antigen BORF1 are maintained in long-term latently infected healthy donors

PLoS pathogens, 2021-12, Vol.17 (12), p.e1010137-e1010137

2021

Volltextzugriff (PDF)

Role of human natural killer cells during Epstein-Barr virus infection

Critical reviews in immunology, 2014, Vol.34 (6), p.501

Münz, Christian

2014

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A distinct subpopulation of human NK cells restricts B cell transformation by EBV

The Journal of immunology (1950), 2013-11, Vol.191 (10), p.4989-4995

2013

Volltextzugriff (PDF)

Autophagy Proteins in Phagocyte Endocytosis and Exocytosis

Frontiers in immunology, 2017-09, Vol.8, p.1183-1183

Münz, Christian

2017

Volltextzugriff (PDF)

Immunodeficiencies that predispose to pathologies by human oncogenic y-herpesviruses

FEMS microbiology reviews, 2019-03, Vol.43 (2), p.181

2019

Volltextzugriff (PDF)

Human γ-Herpesvirus Infection, Tumorigenesis, and Immune Control in Mice with Reconstituted Human Immune System Components

Frontiers in immunology, 2018-02, Vol.9, p.238-238

Münz, Christian

2018

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Attenuated immune control of Epstein–Barr virus in humanized mice is associated with the multiple sclerosis risk factor HLA‐DR15

European journal of immunology, 2021-01, Vol.51 (1), p.64-75

2021

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Persistent KSHV Infection Increases EBV-Associated Tumor Formation In Vivo via Enhanced EBV Lytic Gene Expression

Cell host & microbe, 2017-07, Vol.22 (1), p.61-73.e7

2017

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Patients with Epstein Barr virus‐positive lymphomas have decreased CD4+ T‐cell responses to the viral nuclear antigen 1

International journal of cancer, 2008-12, Vol.123 (12), p.2824-2831

2008

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The autophagy machinery interacts with EBV capsids during viral envelope release

Proceedings of the National Academy of Sciences - PNAS, 2023-08, Vol.120 (34), p.e2211281120-e2211281120

2023

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Elevated Epstein-Barr virus-encoded nuclear antigen-1 immune responses predict conversion to multiple sclerosis

Annals of neurology, 2010-02, Vol.67 (2), p.159-169

2010

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Editorial: EBV-induced T cell immunity in cancers

Frontiers in immunology, 2024-07, Vol.15, p.1454792

2024

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MHC class II‐deficient mice allow functional human CD4+ T‐cell development

European journal of immunology, 2023-07, Vol.53 (7), p.e2250313-n/a

2023

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Cognate HLA absence in trans diminishes human NK cell education

The Journal of clinical investigation, 2016-10, Vol.126 (10), p.3772-3782

2016

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Infectious diseases in humanized mice

European journal of immunology, 2013-09, Vol.43 (9), p.2246-2254

2013

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Immunosuppressive FK506 treatment leads to more frequent EBV-associated lymphoproliferative disease in humanized mice

PLoS pathogens, 2020-04, Vol.16 (4), p.e1008477-e1008477

2020

Volltextzugriff (PDF)

Altered Immune Response to the Epstein–Barr Virus as a Prerequisite for Multiple Sclerosis

Cells (Basel, Switzerland), 2022-09, Vol.11 (17), p.2757

2022

Volltextzugriff (PDF)

Spontaneous Lytic Replication and Epitheliotropism Define an Epstein-Barr Virus Strain Found in Carcinomas

Cell reports (Cambridge), 2013-10, Vol.5 (2), p.458-470

2013

Volltextzugriff (PDF)

Innate immune responses against Epstein Barr virus infection

Journal of leukocyte biology, 2013-12, Vol.94 (6), p.1185-1190

2013

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Human Natural Killer Cells Prevent Infectious Mononucleosis Features by Targeting Lytic Epstein-Barr Virus Infection

Cell reports (Cambridge), 2013-12, Vol.5 (6), p.1489-1498

2013

Volltextzugriff (PDF)

Two alternate strategies for innate immunity to Epstein-Barr virus: One using NK cells and the other NK cells and gamma delta T cells

The Journal of experimental medicine, 2017-06, Vol.214 (6), p.1827-1841

2017

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Altered EBV specific immune control in multiple sclerosis

The Autophagic Machinery in Viral Exocytosis

Epstein-Barr virus as a leading cause of multiple sclerosis: mechanisms and implications

Compartmentalization of Total and Virus-Specific Tissue-Resident Memory CD8+ T Cells in Human Lymphoid Organs

Cytotoxic CD4+ T-cells specific for EBV capsid antigen BORF1 are maintained in long-term latently infected healthy donors

Role of human natural killer cells during Epstein-Barr virus infection

A distinct subpopulation of human NK cells restricts B cell transformation by EBV

Autophagy Proteins in Phagocyte Endocytosis and Exocytosis

Immunodeficiencies that predispose to pathologies by human oncogenic y-herpesviruses

Human γ-Herpesvirus Infection, Tumorigenesis, and Immune Control in Mice with Reconstituted Human Immune System Components

Attenuated immune control of Epstein–Barr virus in humanized mice is associated with the multiple sclerosis risk factor HLA‐DR15

Persistent KSHV Infection Increases EBV-Associated Tumor Formation In Vivo via Enhanced EBV Lytic Gene Expression

Patients with Epstein Barr virus‐positive lymphomas have decreased CD4+ T‐cell responses to the viral nuclear antigen 1

The autophagy machinery interacts with EBV capsids during viral envelope release

Elevated Epstein-Barr virus-encoded nuclear antigen-1 immune responses predict conversion to multiple sclerosis

Editorial: EBV-induced T cell immunity in cancers

MHC class II‐deficient mice allow functional human CD4+ T‐cell development

Cognate HLA absence in trans diminishes human NK cell education

Infectious diseases in humanized mice

Immunosuppressive FK506 treatment leads to more frequent EBV-associated lymphoproliferative disease in humanized mice

Altered Immune Response to the Epstein–Barr Virus as a Prerequisite for Multiple Sclerosis

Spontaneous Lytic Replication and Epitheliotropism Define an Epstein-Barr Virus Strain Found in Carcinomas

Innate immune responses against Epstein Barr virus infection

Human Natural Killer Cells Prevent Infectious Mononucleosis Features by Targeting Lytic Epstein-Barr Virus Infection

Two alternate strategies for innate immunity to Epstein-Barr virus: One using NK cells and the other NK cells and gamma delta T cells

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