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Circulating myocardial microRNAs from infarcted hearts are carried in exosomes and mobilise bone marrow progenitor cells

Nature communications, 2019-02, Vol.10 (1), p.959-959, Article 959

2019

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Mononuclear Phagocytes Are Dispensable for Cardiac Remodeling in Established Pressure-Overload Heart Failure

PloS one, 2017-01, Vol.12 (1), p.e0170781-e0170781

2017

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Inhibiting Na+/K+ ATPase can impair mitochondrial energetics and induce abnormal Ca2+ cycling and automaticity in guinea pig cardiomyocytes

PloS one, 2014-04, Vol.9 (4), p.e93928

2014

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O-linked β-N-acetylglucosamine transferase is indispensable in the failing heart

Proceedings of the National Academy of Sciences - PNAS, 2010-10, Vol.107 (41), p.17797-17802

2010

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Endothelial Cell Culture Model for Replication of Physiological Profiles of Pressure, Flow, Stretch, and Shear Stress in Vitro

Analytical chemistry (Washington), 2011-04, Vol.83 (8), p.3170-3177

2011

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Cardiac Stem Cells Delivered Intravascularly Traverse the Vessel Barrier, Regenerate Infarcted Myocardium, and Improve Cardiac Function

Proceedings of the National Academy of Sciences - PNAS, 2005-03, Vol.102 (10), p.3766-3771

2005

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Cardiomyocyte Ogt limits ventricular dysfunction in mice following pressure overload without affecting hypertrophy

Basic research in cardiology, 2017-05, Vol.112 (3), p.23-23, Article 23

2017

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Remodeling of the mononuclear phagocyte network underlies chronic inflammation and disease progression in heart failure: critical importance of the cardiosplenic axis

Circulation research, 2014-01, Vol.114 (2), p.266-282

2014

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O-GlcNAcylation Negatively Regulates Cardiomyogenic Fate in Adult Mouse Cardiac Mesenchymal Stromal Cells

PloS one, 2015-11, Vol.10 (11), p.e0142939-e0142939

2015

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Complete heart block without ventricular escape secondary to hyperkalemia induced by herbal tea

HeartRhythm case reports, 2022-01, Vol.8 (1), p.45-49

2022

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THE CARDIOSPLENIC AXIS IS ESSENTIAL FOR THE PATHOGENESIS OF ISCHEMIC HEART FAILURE

Transactions of the American Clinical and Climatological Association, 2018, Vol.129, p.202-214

Prabhu, Sumanth D

2018

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Resolution Agonist 15-epi-Lipoxin A4 Programs Early Activation of Resolving Phase in Post-Myocardial Infarction Healing

Scientific reports, 2017-08, Vol.7 (1), p.1-14, Article 9999

2017

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Spironolactone Use and Higher Hospital Readmission for Medicare Beneficiaries With Heart Failure, Left Ventricular Ejection Fraction <45%, and Estimated Glomerular Filtration Rate <45 ml/min/1.73 m2

The American journal of cardiology, 2014-07, Vol.114 (1), p.79-82

2014

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EMMPRIN activates multiple transcription factors in cardiomyocytes, and induces interleukin-18 expression via Rac1-dependent PI3K/Akt/IKK/NF-κB andMKK7/JNK/AP-1 signaling

Journal of molecular and cellular cardiology, 2010-10, Vol.49 (4), p.655-663

2010

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Publisher Correction: Reappraising the role of inflammation in heart failure

Nature reviews cardiology, 2021-10, Vol.18 (10), p.735-735

2021

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Heme oxygenase-1 regulates mitochondrial quality control in the heart

JCI insight, 2016-02, Vol.1 (2)

2016

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Microfluidic Cardiac Cell Culture Model (μCCCM)

Analytical chemistry (Washington), 2010-09, Vol.82 (18), p.7581-7587

2010

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Genetic deletion of 12/15 lipoxygenase delays vascular remodeling and limits cardiorenal dysfunction after pressure overload

Journal of Molecular and Cellular Cardiology Plus (Online), 2023-09, Vol.5, p.100046, Article 100046

2023

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Continuous-Flow Left Ventricular Assist Device Support Improves Myocardial Supply:Demand in Chronic Heart Failure

Annals of biomedical engineering, 2017-06, Vol.45 (6), p.1475-1486

2017

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TCT-214 Pharmacodynamic Effects of Ticagrelor and Eptifibatide Bolus-Only vs. Ticagrelor and Eptifibatide Bolus plus 2 h Infusion in Patients with Acute Coronary Syndrome

Journal of the American College of Cardiology, 2015-10, Vol.66 (15), p.B82-B82

2015

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Hemodynamic Responses to Continuous versus Pulsatile Mechanical Unloading of the Failing Left Ventricle

ASAIO journal (1992), 2010-09, Vol.56 (5), p.410-416

2010

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β-Adrenergic stimulation induces interleukin-18 expression via β2-AR, PI3K, Akt, IKK, and NF-κB

Biochemical and biophysical research communications, 2004-06, Vol.319 (2), p.304-311

2004

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Reply to the Editor

The Journal of thoracic and cardiovascular surgery, 2012-12, Vol.144 (6), p.1531-1533

2012

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Effect of Digoxin in Patients with Systolic Heart Failure and Hypokalemia

Journal of cardiac failure, 2013-08, Vol.19 (8), p.S74-S74

2013

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Myocardial Recovery with Optimization of Medical Management in Patients with Advanced Systolic Heart Failure

Journal of cardiac failure, 2013-08, Vol.19 (8), p.S47-S47

2013

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Circulating myocardial microRNAs from infarcted hearts are carried in exosomes and mobilise bone marrow progenitor cells

Mononuclear Phagocytes Are Dispensable for Cardiac Remodeling in Established Pressure-Overload Heart Failure

Inhibiting Na+/K+ ATPase can impair mitochondrial energetics and induce abnormal Ca2+ cycling and automaticity in guinea pig cardiomyocytes

O-linked β-N-acetylglucosamine transferase is indispensable in the failing heart

Endothelial Cell Culture Model for Replication of Physiological Profiles of Pressure, Flow, Stretch, and Shear Stress in Vitro

Cardiac Stem Cells Delivered Intravascularly Traverse the Vessel Barrier, Regenerate Infarcted Myocardium, and Improve Cardiac Function

Cardiomyocyte Ogt limits ventricular dysfunction in mice following pressure overload without affecting hypertrophy

Remodeling of the mononuclear phagocyte network underlies chronic inflammation and disease progression in heart failure: critical importance of the cardiosplenic axis

O-GlcNAcylation Negatively Regulates Cardiomyogenic Fate in Adult Mouse Cardiac Mesenchymal Stromal Cells

Complete heart block without ventricular escape secondary to hyperkalemia induced by herbal tea

THE CARDIOSPLENIC AXIS IS ESSENTIAL FOR THE PATHOGENESIS OF ISCHEMIC HEART FAILURE

Resolution Agonist 15-epi-Lipoxin A4 Programs Early Activation of Resolving Phase in Post-Myocardial Infarction Healing

Spironolactone Use and Higher Hospital Readmission for Medicare Beneficiaries With Heart Failure, Left Ventricular Ejection Fraction <45%, and Estimated Glomerular Filtration Rate <45 ml/min/1.73 m2

EMMPRIN activates multiple transcription factors in cardiomyocytes, and induces interleukin-18 expression via Rac1-dependent PI3K/Akt/IKK/NF-κB andMKK7/JNK/AP-1 signaling

Publisher Correction: Reappraising the role of inflammation in heart failure

Heme oxygenase-1 regulates mitochondrial quality control in the heart

Microfluidic Cardiac Cell Culture Model (μCCCM)

Genetic deletion of 12/15 lipoxygenase delays vascular remodeling and limits cardiorenal dysfunction after pressure overload

Continuous-Flow Left Ventricular Assist Device Support Improves Myocardial Supply:Demand in Chronic Heart Failure

TCT-214 Pharmacodynamic Effects of Ticagrelor and Eptifibatide Bolus-Only vs. Ticagrelor and Eptifibatide Bolus plus 2 h Infusion in Patients with Acute Coronary Syndrome

Hemodynamic Responses to Continuous versus Pulsatile Mechanical Unloading of the Failing Left Ventricle

β-Adrenergic stimulation induces interleukin-18 expression via β2-AR, PI3K, Akt, IKK, and NF-κB

Reply to the Editor

Effect of Digoxin in Patients with Systolic Heart Failure and Hypokalemia

Myocardial Recovery with Optimization of Medical Management in Patients with Advanced Systolic Heart Failure

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