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The Transition-State Structure for Human MAT2A from Isotope Effects

Journal of the American Chemical Society, 2017-10, Vol.139 (39), p.13754-13760

2017

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Transition state analogue of MTAP extends lifespan of APCMin/+ mice

Scientific reports, 2021-04, Vol.11 (1), p.1-11, Article 8844

2021

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Continuous Fluorescence Assays for Reactions Involving Adenine

Analytical chemistry (Washington), 2016-12, Vol.88 (23), p.11860-11867

2016

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Microwave spectra, structure, and dynamics of the weakly bound complex, N2 CO2

The Journal of chemical physics, 2010-12, Vol.133 (24), p.244303-244303

2010

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P962: COMMERCIAL TECLISTAMAB IN ANTI‐BCMA THERAPY EXPOSED RELAPSED REFRACTORY MULTIPLE MYELOMA PATIENTS: THE MSKCC EXPERIENCE

HemaSphere, 2023-08, Vol.7 (S3), p.e81198bb-n/a

2023

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Abstract 18: Doubled lifespan in APCMin/+ mice by targeting MTAP

Cancer research (Chicago, Ill.), 2019-07, Vol.79 (13_Supplement), p.18-18

2019

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Evaluating the efficacy of commercial teclistamab in relapsed refractory multiple myeloma patients with prior exposure to anti-BCMA therapies

Journal of clinical oncology, 2023-06, Vol.41 (16_suppl), p.8049-8049

2023

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P-486 Clinical significance of baseline body mass index and its trajectory during triplet induction therapy in newly diagnosed multiple myeloma

Clinical lymphoma, myeloma and leukemia, 2023-09, Vol.23, p.S307-S308

2023

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OA-03 Bispecific T-cell engager response is driven by pre-treatment CD8+ effector memory cells and inhibited by TIGIT+ Tregs in relapsed/refractory multiple myeloma

Clinical lymphoma, myeloma and leukemia, 2023-09, Vol.23, p.S2-S2

2023

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P-013 Patterns of cytokine release syndrome with teclistamab in relapsed/refractory multiple myeloma with or without prior T-cell redirection therapy

Clinical lymphoma, myeloma and leukemia, 2023-09, Vol.23, p.S40-S40

2023

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Transition state analogue of MTAP extends lifespan of APC Min/+ mice

Scientific reports, 2021-04, Vol.11 (1), p.8844

2021

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Enzymes and Transition State Structures Relating to SAM Metabolism

Firestone, Ross

2017

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Microwave spectra, structure, and dynamics of the weakly bound complex, N 2 CO 2

The Journal of chemical physics, 2010-12, Vol.133 (24), p.244303-244303-6

2010

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Application of Thermoluminescence and Reflectance Methods to Study of Lattice Defects in Alumina Ceramics

Journal of the American Ceramic Society, 1960-09, Vol.43 (9), p.476-483

1960

Volltextzugriff (PDF)

BASAL AND NON-BASAL SLIP IN SAPPHIRE

FIRESTONE, ROSS FRANCIS

1974

Volltextzugriff (PDF)

Diahann Carroll Tells All: Autobiography Details Stormy, Nine-Year Love Affair with Sidney Poitier

Ebony, 1986-06, Vol.41 (8), p.126

1986

Volltextzugriff (PDF)

Life with Bing Crosby, my father

Good housekeeping (U.S. ed.), 1983-03, Vol.196 (3), p.134

1983

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The last word

New scientist (1971), 2003-05, Vol.178 (2395), p.57

2003

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Actress: Postcards from the Road

Theatre Journal, 1979, Vol.31 (4), p.560-561

Robbins, Kathleen M.

1979

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Glucocorticoid-stimulated CCAAT/enhancer-binding protein alpha expression is required for steroid-induced G1 cell cycle arrest of minimal-deviation rat hepatoma cells

Molecular and Cellular Biology, 1996-10, Vol.16 (10), p.5288-5301

1996

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Role of the CCAAT/Enhancer Binding Protein-Î± Transcription Factor in the Glucocorticoid Stimulation of p21 waf1/cip1 Gene Promoter Activity in Growth-arrested Rat Hepatoma Cells

The Journal of biological chemistry, 1998-01, Vol.273 (4), p.2008

1998

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Role of the CCAAT/Enhancer Binding Protein-α Transcription Factor in the Glucocorticoid Stimulation of p21 waf1/cip1 Gene Promoter Activity in Growth-arrested Rat Hepatoma Cells

The Journal of biological chemistry, 1998-01, Vol.273 (4), p.2008-2014

1998

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Glucocorticoid-Stimulated CCAAT/Enhancer-Binding Protein α Expression Is Required for Steroid-Induced G 1 Cell Cycle Arrest of Minimal-Deviation Rat Hepatoma Cells

Molecular and cellular biology, 1996-10, Vol.16 (10), p.5288-5301

1996

Volltextzugriff (PDF)

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The Transition-State Structure for Human MAT2A from Isotope Effects

Transition state analogue of MTAP extends lifespan of APCMin/+ mice

Continuous Fluorescence Assays for Reactions Involving Adenine

Microwave spectra, structure, and dynamics of the weakly bound complex, N2 CO2

P962: COMMERCIAL TECLISTAMAB IN ANTI‐BCMA THERAPY EXPOSED RELAPSED REFRACTORY MULTIPLE MYELOMA PATIENTS: THE MSKCC EXPERIENCE

Abstract 18: Doubled lifespan in APCMin/+ mice by targeting MTAP

Evaluating the efficacy of commercial teclistamab in relapsed refractory multiple myeloma patients with prior exposure to anti-BCMA therapies

P-486 Clinical significance of baseline body mass index and its trajectory during triplet induction therapy in newly diagnosed multiple myeloma

OA-03 Bispecific T-cell engager response is driven by pre-treatment CD8+ effector memory cells and inhibited by TIGIT+ Tregs in relapsed/refractory multiple myeloma

P-013 Patterns of cytokine release syndrome with teclistamab in relapsed/refractory multiple myeloma with or without prior T-cell redirection therapy

Transition state analogue of MTAP extends lifespan of APC Min/+ mice

Enzymes and Transition State Structures Relating to SAM Metabolism

Unscheduled healthcare interactions in multiple myeloma patients receiving T cell redirection therapies

Patterns of CRS with Teclistamab in Relapsed/Refractory Multiple Myeloma patients with Prior T-Cell Redirection Therapy

Microwave spectra, structure, and dynamics of the weakly bound complex, N 2 CO 2

Application of Thermoluminescence and Reflectance Methods to Study of Lattice Defects in Alumina Ceramics

BASAL AND NON-BASAL SLIP IN SAPPHIRE

Diahann Carroll Tells All: Autobiography Details Stormy, Nine-Year Love Affair with Sidney Poitier

Life with Bing Crosby, my father

The last word

Actress: Postcards from the Road

Glucocorticoid-stimulated CCAAT/enhancer-binding protein alpha expression is required for steroid-induced G1 cell cycle arrest of minimal-deviation rat hepatoma cells

Role of the CCAAT/Enhancer Binding Protein-Î± Transcription Factor in the Glucocorticoid Stimulation of p21 waf1/cip1 Gene Promoter Activity in Growth-arrested Rat Hepatoma Cells

Role of the CCAAT/Enhancer Binding Protein-α Transcription Factor in the Glucocorticoid Stimulation of p21 waf1/cip1 Gene Promoter Activity in Growth-arrested Rat Hepatoma Cells

Glucocorticoid-Stimulated CCAAT/Enhancer-Binding Protein α Expression Is Required for Steroid-Induced G 1 Cell Cycle Arrest of Minimal-Deviation Rat Hepatoma Cells

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