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Mechanism of adrenergic CaV1.2 stimulation revealed by proximity proteomics

Nature (London), 2020-01, Vol.577 (7792), p.695-700

2020

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Inhibition of NAPDH Oxidase 2 (NOX2) Prevents Oxidative Stress and Mitochondrial Abnormalities Caused by Saturated Fat in Cardiomyocytes

PloS one, 2016-01, Vol.11 (1), p.e0145750-e0145750

2016

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Structure and selectivity in bestrophin ion channels

Science (American Association for the Advancement of Science), 2014-10, Vol.346 (6207), p.355-359

2014

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Distinct RGK GTPases differentially use α1- and auxiliary β-binding-dependent mechanisms to inhibit CaV1.2/CaV2.2 channels

PloS one, 2012-05, Vol.7 (5), p.e37079-e37079

2012

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A potent voltage-gated calcium channel inhibitor engineered from a nanobody targeted to auxiliary CaVβ subunits

eLife, 2019-08, Vol.8

2019

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Preassociated apocalmodulin mediates Ca²⁺-dependent sensitization of activation and inactivation of TMEM16A/16B Ca²⁺-gated Cl⁻ channels

Proceedings of the National Academy of Sciences - PNAS, 2014-12, Vol.111 (51), p.18213-18218

2014

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Selective posttranslational inhibition of CaVβ1-associated voltage-dependent calcium channels with a functionalized nanobody

Nature communications, 2022-12, Vol.13 (1), p.7556-7556, Article 7556

2022

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Regulation of Blood Pressure by Targeting CaV1.2-Galectin-1 Protein Interaction

Circulation (New York, N.Y.), 2018-10, Vol.138 (14), p.1431-1445

2018

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Manipulating L-type calcium channels in cardiomyocytes using split-intein protein transsplicing

Proceedings of the National Academy of Sciences - PNAS, 2013-09, Vol.110 (38), p.15461-15466

2013

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David T. Yue: In Memoriam

Neuron (Cambridge, Mass.), 2015-03, Vol.85 (6), p.1158-1161

2015

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Molecular Mechanisms, and Selective Pharmacological Rescue, of Rem-Inhibited CaV1.2 Channels in Heart

Circulation research, 2010-09, Vol.107 (5), p.620-630

2010

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Correction: Inhibition of NAPDH Oxidase 2 (NOX2) Prevents Oxidative Stress and Mitochondrial Abnormalities Caused by Saturated Fat in Cardiomyocytes

PloS one, 2017-03, Vol.12 (3), p.e0174525-e0174525

2017

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Mechanism of auxiliary β‐subunit‐mediated membrane targeting of L‐type (CaV1.2) channels

The Journal of physiology, 2011-09, Vol.589 (18), p.4437-4455

2011

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Targeted ubiquitination of sensory neuron calcium channels reduces the development of neuropathic pain

Proceedings of the National Academy of Sciences - PNAS, 2022-05, Vol.119 (20), p.e2118129119

2022

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Engineered Calmodulins Reveal the Unexpected Eminence of Ca2+Channel Inactivation in Controlling Heart Excitation

Proceedings of the National Academy of Sciences - PNAS, 2002-12, Vol.99 (26), p.17185-17190

2002

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Rem, a member of the RGK GTPases, inhibits recombinant CaV1.2 channels using multiple mechanisms that require distinct conformations of the GTPase

The Journal of physiology, 2010-05, Vol.588 (10), p.1665-1681

2010

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Proteolytic cleavage and PKA phosphorylation of α1C subunit are not required for adrenergic regulation of CaV1.2 in the heart

Proceedings of the National Academy of Sciences - PNAS, 2017-08, Vol.114 (34), p.9194-9199

2017

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Engineering selectivity into RGK GTPase inhibition of voltage-dependent calcium channels

Proceedings of the National Academy of Sciences - PNAS, 2018-11, Vol.115 (47), p.12051-12056

2018

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Bio-inspired voltage-dependent calcium channel blockers

Nature communications, 2013-10, Vol.4 (1), p.2540-2540, Article 2540

2013

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Calmodulin regulation of TMEM16A and 16B Ca(2+)-activated chloride channels

Channels (Austin, Tex.), 2016, Vol.10 (1), p.38-44

2016

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Design and Applications of Genetically-Encoded Voltage-Dependent Calcium Channel Inhibitors

Handbook of experimental pharmacology, 2023, Vol.279, p.139-155

2023

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Aberrant Splicing Promotes Proteasomal Degradation of L-type CaV1.2 Calcium Channels by Competitive Binding for CaVβ Subunits in Cardiac Hypertrophy

Scientific reports, 2016-10, Vol.6 (1), p.35247-35247, Article 35247

2016

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Role of CaVβ Subunits, and Lack of Functional Reserve, in Protein Kinase A Modulation of Cardiac CaV1.2 Channels

Circulation research, 2008-04, Vol.102 (7), p.e54-e64

2008

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Adrenergic CaV1.2 Activation via Rad Phosphorylation Converges at α1C I-II Loop

Circulation research, 2021-01, Vol.128 (1), p.76-88

2021

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Adrenergic Ca V 1.2 Activation via Rad Phosphorylation Converges at α 1C I-II Loop

Circulation research, 2021-01, Vol.128 (1), p.76-88

2021

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Mechanism of adrenergic CaV1.2 stimulation revealed by proximity proteomics

Inhibition of NAPDH Oxidase 2 (NOX2) Prevents Oxidative Stress and Mitochondrial Abnormalities Caused by Saturated Fat in Cardiomyocytes

Structure and selectivity in bestrophin ion channels

Distinct RGK GTPases differentially use α1- and auxiliary β-binding-dependent mechanisms to inhibit CaV1.2/CaV2.2 channels

A potent voltage-gated calcium channel inhibitor engineered from a nanobody targeted to auxiliary CaVβ subunits

Preassociated apocalmodulin mediates Ca²⁺-dependent sensitization of activation and inactivation of TMEM16A/16B Ca²⁺-gated Cl⁻ channels

Selective posttranslational inhibition of CaVβ1-associated voltage-dependent calcium channels with a functionalized nanobody

Regulation of Blood Pressure by Targeting CaV1.2-Galectin-1 Protein Interaction

Manipulating L-type calcium channels in cardiomyocytes using split-intein protein transsplicing

David T. Yue: In Memoriam

Molecular Mechanisms, and Selective Pharmacological Rescue, of Rem-Inhibited CaV1.2 Channels in Heart

Correction: Inhibition of NAPDH Oxidase 2 (NOX2) Prevents Oxidative Stress and Mitochondrial Abnormalities Caused by Saturated Fat in Cardiomyocytes

Mechanism of auxiliary β‐subunit‐mediated membrane targeting of L‐type (CaV1.2) channels

Targeted ubiquitination of sensory neuron calcium channels reduces the development of neuropathic pain

Engineered Calmodulins Reveal the Unexpected Eminence of Ca2+Channel Inactivation in Controlling Heart Excitation

Rem, a member of the RGK GTPases, inhibits recombinant CaV1.2 channels using multiple mechanisms that require distinct conformations of the GTPase

Proteolytic cleavage and PKA phosphorylation of α1C subunit are not required for adrenergic regulation of CaV1.2 in the heart

Engineering selectivity into RGK GTPase inhibition of voltage-dependent calcium channels

Bio-inspired voltage-dependent calcium channel blockers

Calmodulin regulation of TMEM16A and 16B Ca(2+)-activated chloride channels

Design and Applications of Genetically-Encoded Voltage-Dependent Calcium Channel Inhibitors

Aberrant Splicing Promotes Proteasomal Degradation of L-type CaV1.2 Calcium Channels by Competitive Binding for CaVβ Subunits in Cardiac Hypertrophy

Role of CaVβ Subunits, and Lack of Functional Reserve, in Protein Kinase A Modulation of Cardiac CaV1.2 Channels

Adrenergic CaV1.2 Activation via Rad Phosphorylation Converges at α1C I-II Loop

Adrenergic Ca V 1.2 Activation via Rad Phosphorylation Converges at α 1C I-II Loop

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