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A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28 (KAP1), a gene critical in early renal development: A Children's Oncology Group study

PloS one, 2018-12, Vol.13 (12), p.e0208936-e0208936

2018

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Reprogramming of Sertoli cells to fetal-like Leydig cells by Wt1 ablation

Proceedings of the National Academy of Sciences - PNAS, 2015-03, Vol.112 (13), p.4003-4008

2015

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The Wilms Tumor Gene, Wt1, Is Required for Sox9 Expression and Maintenance of Tubular Architecture in the Developing Testis

Proceedings of the National Academy of Sciences - PNAS, 2006-08, Vol.103 (32), p.11987-11992

2006

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Wilm’s tumor 1 promotes memory flexibility

Nature communications, 2019-08, Vol.10 (1), p.3756-18, Article 3756

2019

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Rare variant detection using family-based sequencing analysis

Proceedings of the National Academy of Sciences - PNAS, 2013-03, Vol.110 (10), p.3985-3990

2013

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A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28

PloS one, 2018-12, Vol.13 (12), p.e0208936

2018

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MLLT1 YEATS domain mutations in clinically distinctive Favourable Histology Wilms tumours

Nature communications, 2015-12, Vol.6 (1), p.10013, Article 10013

2015

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Identification of germline DICER1 mutations and loss of heterozygosity in familial Wilms tumor using whole genome sequencing

Journal of medical genetics, 2015-11, Vol.53 (6), p.385-388

2015

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Abstract 5047: Impact of kidney progenitor cell differentiation status in Wilms tumorigenesis

Cancer research (Chicago, Ill.), 2013-04, Vol.73 (8_Supplement), p.5047-5047

2013

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Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with β-Catenin Activation or Wt1 Ablation and Igf2 Upregulation1

Neoplasia (New York, N.Y.), 2016-02, Vol.18 (2), p.71-81

2016

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Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with [beta]-Catenin Activation or Wt1 Ablation and Igf2 Upregulation

Neoplasia (New York, N.Y.), 2016-02, Vol.18 (2), p.71-81

2016

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Knock-in of the Wt1 R394W mutation causes MDS and cooperates with Flt3/ITD to drive aggressive myeloid neoplasms in mice

Oncotarget, 2018-10, Vol.9 (82), p.35313-35326

2018

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A Wilms Tumor 1 (WT1) Mutation Causes Myelodysplastic Syndrome in a Knock-in Mouse Model, and a Mixed Myelodysplastic/Myeloproliferative Neoplam in Double Knock-in Mice with WT1 and FLT3/ITD Mutations

Blood, 2015-12, Vol.126 (23), p.312-312

2015

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The Wt1+/R394W Mouse Displays Glomerulosclerosis and Early-Onset Renal Failure Characteristic of Human Denys-Drash Syndrome

Molecular and Cellular Biology, 2004-11, Vol.24 (22), p.9899-9910

2004

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Reprogramming of Sertoli cells to fetal-like Leydig cells byWt1ablation

Proceedings of the National Academy of Sciences - PNAS, 2015-03, Vol.112 (13), p.4003-4008

2015

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Clinical outcome and biological predictors of relapse following nephrectomy only for very low risk Wilms tumor (VLR WT): A report from Children’s Oncology Group AREN0532

Journal of clinical oncology, 2015-05, Vol.33 (15_suppl), p.10023-10023

2015

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Abstract LB-180: The genetic landscape of Wilms tumor

Cancer research (Chicago, Ill.), 2016-07, Vol.76 (14_Supplement), p.LB-180-LB-180

2016

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Abstract 5353: Comprehensive Ggnomic analysis of diffuse hyperplastic perilobar nephroblastomatosis (DHPLN)

Cancer research (Chicago, Ill.), 2011-04, Vol.71 (8_Supplement), p.5353-5353

2011

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Reply to B. Royer-Pokora et al

Journal of clinical oncology, 2011-06, Vol.29 (16), p.e487-e488

2011

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Abstract 1439: Loss of expression of SWI/SNF subunits in high risk Wilms tumor is accompanied by global gene expression changes

Cancer research (Chicago, Ill.), 2012-04, Vol.72 (8_Supplement), p.1439-1439

2012

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The Wilms Tumor Gene, Wt1, Is Critical for Mouse Spermatogenesis via Regulation of Sertoli Cell Polarity and Is Associated with Non-Obstructive Azoospermia in Humans: e1003645

PLoS genetics, 2013-08, Vol.9 (8)

2013

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Clinically Relevant Subsets Identified by Gene Expression Patterns Support a Revised Ontogenic Model of Wilms Tumor: A Children's Oncology Group Study12

Neoplasia (New York, N.Y.), 2012-08, Vol.14 (8), p.742-756

2012

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Monitoring Therapy with MEK Inhibitor U0126 in a Novel Wilms Tumor Model in Wt1 Knockout Igf2 Transgenic Mice Using super(18)F-FDG PET with Dual-Contrast Enhanced CT and MRI: Early Metabolic Response Without Inhibition of Tumor Growth

Molecular imaging and biology, 2013-04, Vol.15 (2), p.175-185

2013

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Monitoring Therapy with MEK Inhibitor U0126 in a Novel Wilms Tumor Model in Wt1 Knockout Igf2 Transgenic Mice Using ^sup 18^F-FDG PET with Dual-Contrast Enhanced CT and MRI: Early Metabolic Response Without Inhibition of Tumor Growth

Molecular imaging and biology, 2013-04, Vol.15 (2), p.175

2013

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A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28 (KAP1), a gene critical in early renal development: A Children's Oncology Group study

Reprogramming of Sertoli cells to fetal-like Leydig cells by Wt1 ablation

The Wilms Tumor Gene, Wt1, Is Required for Sox9 Expression and Maintenance of Tubular Architecture in the Developing Testis

Wilm’s tumor 1 promotes memory flexibility

Rare variant detection using family-based sequencing analysis

A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28

Genetic and epigenetic features of bilateral Wilms tumor predisposition in patients from the Children’s Oncology Group AREN18B5-Q

MLLT1 YEATS domain mutations in clinically distinctive Favourable Histology Wilms tumours

Identification of germline DICER1 mutations and loss of heterozygosity in familial Wilms tumor using whole genome sequencing

Abstract 5047: Impact of kidney progenitor cell differentiation status in Wilms tumorigenesis

Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with β-Catenin Activation or Wt1 Ablation and Igf2 Upregulation1

Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with [beta]-Catenin Activation or Wt1 Ablation and Igf2 Upregulation

Knock-in of the Wt1 R394W mutation causes MDS and cooperates with Flt3/ITD to drive aggressive myeloid neoplasms in mice

A Wilms Tumor 1 (WT1) Mutation Causes Myelodysplastic Syndrome in a Knock-in Mouse Model, and a Mixed Myelodysplastic/Myeloproliferative Neoplam in Double Knock-in Mice with WT1 and FLT3/ITD Mutations

The Wt1+/R394W Mouse Displays Glomerulosclerosis and Early-Onset Renal Failure Characteristic of Human Denys-Drash Syndrome

Reprogramming of Sertoli cells to fetal-like Leydig cells byWt1ablation

Clinical outcome and biological predictors of relapse following nephrectomy only for very low risk Wilms tumor (VLR WT): A report from Children’s Oncology Group AREN0532

Abstract LB-180: The genetic landscape of Wilms tumor

Abstract 5353: Comprehensive Ggnomic analysis of diffuse hyperplastic perilobar nephroblastomatosis (DHPLN)

Reply to B. Royer-Pokora et al

Abstract 1439: Loss of expression of SWI/SNF subunits in high risk Wilms tumor is accompanied by global gene expression changes

The Wilms Tumor Gene, Wt1, Is Critical for Mouse Spermatogenesis via Regulation of Sertoli Cell Polarity and Is Associated with Non-Obstructive Azoospermia in Humans: e1003645

Clinically Relevant Subsets Identified by Gene Expression Patterns Support a Revised Ontogenic Model of Wilms Tumor: A Children's Oncology Group Study12

Monitoring Therapy with MEK Inhibitor U0126 in a Novel Wilms Tumor Model in Wt1 Knockout Igf2 Transgenic Mice Using super(18)F-FDG PET with Dual-Contrast Enhanced CT and MRI: Early Metabolic Response Without Inhibition of Tumor Growth

Monitoring Therapy with MEK Inhibitor U0126 in a Novel Wilms Tumor Model in Wt1 Knockout Igf2 Transgenic Mice Using ^sup 18^F-FDG PET with Dual-Contrast Enhanced CT and MRI: Early Metabolic Response Without Inhibition of Tumor Growth

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