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Modelling sudden cardiac death risks factors in patients with coronavirus disease of 2019: the hydroxychloroquine and azithromycin case

Europace (London, England), 2021-05, Vol.23 (7), p.1124-1136

2021

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Infanticide vs. inherited cardiac arrhythmias

Europace (London, England), 2021-03, Vol.23 (3), p.441-450

2021

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Arrhythmias precede cardiomyopathy and remodeling of Ca2+ handling proteins in a novel model of long QT syndrome

Journal of molecular and cellular cardiology, 2018-10, Vol.123, p.13-25

2018

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Transforming growth factor β receptor inhibition prevents ventricular fibrosis in a mouse model of progressive cardiac conduction disease

Cardiovascular research, 2017-04, Vol.113 (5), p.464-474

2017

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Dysfunction of the Voltage-Gated K+ Channel beta 2 Subunit in a Familial Case of Brugada Syndrome

Journal of the American Heart Association, 2016-06, Vol.5 (6)

2016

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Toward Personalized Medicine: Using Cardiomyocytes Differentiated From Urine-Derived Pluripotent Stem Cells to Recapitulate Electrophysiological Characteristics of Type 2 Long QT Syndrome

Journal of the American Heart Association, 2015-09, Vol.4 (9), p.e002159-e002159

2015

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Neural modulation of ion channels in cardiac arrhythmias: Clinical implications and future investigations

Heart rhythm, 2010-06, Vol.7 (6), p.847-849

2010

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Trafficking-deficient long QT syndrome mutation KCNQ1-T587M confers severe clinical phenotype by impairment of KCNH2 membrane localization: Evidence for clinically significant I Kr-I Ks α-subunit interaction

Heart rhythm, 2009-12, Vol.6 (12), p.1792-1801

2009

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Torsades de pointes complicating atrioventricular block: Evidence for a genetic predisposition

Heart rhythm, 2007-02, Vol.4 (2), p.170-174

2007

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Atrial fibrillation: Is NO an answer for refractoriness?

Cardiovascular research, 2006-10, Vol.72 (1), p.7-8

BARO, I

2006

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14-3-3 Is a Regulator of the Cardiac Voltage-Gated Sodium Channel Nav1.5

Circulation research, 2006-06, Vol.98 (12), p.1538-1546

2006

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New KCNQ1 mutations leading to haploinsufficiency in a general population: Defective trafficking of a KvLQT1 mutant

Cardiovascular research, 2004-07, Vol.63 (1), p.60-68

2004

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Na + channel mutation leading to loss of function and non-progressive cardiac conduction defects

Journal of molecular and cellular cardiology, 2003-05, Vol.35 (5), p.549-557

2003

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Microarray Analysis Reveals Complex Remodeling of Cardiac Ion Channel Expression With Altered Thyroid Status: Relation to Cellular and Integrated Electrophysiology

Circulation research, 2003-02, Vol.92 (2), p.234-242

2003

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Transgenic mice overexpressing human KvLQT1 dominant-negative isoform. Part I: Phenotypic characterisation

Cardiovascular research, 2001-05, Vol.50 (2), p.314-327

2001

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AKAP proteins anchor cAMP-dependent protein kinase to KvLQT1/IsK channel complex

American journal of physiology. Heart and circulatory physiology, 2001-05, Vol.280 (5), p.H2038-H2045

2001

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Non-invasive testing of acquired long QT syndrome: evidence for multiple arrhythmogenic substrates

Cardiovascular research, 2001-05, Vol.50 (2), p.386-398

2001

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Differential expression of KvLQT1 isoforms across the human ventricular wall

American journal of physiology. Heart and circulatory physiology, 2000-06, Vol.278 (6), p.H1908-H1915

2000

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Modelling sudden cardiac death risks factors in patients with coronavirus disease of 2019: the hydroxychloroquine and azithromycin case

Infanticide vs. inherited cardiac arrhythmias

RRAD mutation causes electrical and cytoskeletal defects in cardiomyocytes derived from a familial case of Brugada syndrome

Arrhythmias precede cardiomyopathy and remodeling of Ca2+ handling proteins in a novel model of long QT syndrome

Transforming growth factor β receptor inhibition prevents ventricular fibrosis in a mouse model of progressive cardiac conduction disease

Dysfunction of the Voltage-Gated K+ Channel beta 2 Subunit in a Familial Case of Brugada Syndrome

Toward Personalized Medicine: Using Cardiomyocytes Differentiated From Urine-Derived Pluripotent Stem Cells to Recapitulate Electrophysiological Characteristics of Type 2 Long QT Syndrome

Neural modulation of ion channels in cardiac arrhythmias: Clinical implications and future investigations

Trafficking-deficient long QT syndrome mutation KCNQ1-T587M confers severe clinical phenotype by impairment of KCNH2 membrane localization: Evidence for clinically significant I Kr-I Ks α-subunit interaction

Torsades de pointes complicating atrioventricular block: Evidence for a genetic predisposition

Atrial fibrillation: Is NO an answer for refractoriness?

14-3-3 Is a Regulator of the Cardiac Voltage-Gated Sodium Channel Nav1.5

New KCNQ1 mutations leading to haploinsufficiency in a general population: Defective trafficking of a KvLQT1 mutant

Na + channel mutation leading to loss of function and non-progressive cardiac conduction defects

Microarray Analysis Reveals Complex Remodeling of Cardiac Ion Channel Expression With Altered Thyroid Status: Relation to Cellular and Integrated Electrophysiology

Transgenic mice overexpressing human KvLQT1 dominant-negative isoform. Part I: Phenotypic characterisation

AKAP proteins anchor cAMP-dependent protein kinase to KvLQT1/IsK channel complex

Non-invasive testing of acquired long QT syndrome: evidence for multiple arrhythmogenic substrates

Differential expression of KvLQT1 isoforms across the human ventricular wall

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