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The role of GSK3 in glucose homeostasis and the development of insulin resistance

Diabetes research and clinical practice, 2007-09, Vol.77 (3), p.S49-S57

2007

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The emerging era of multidisciplinary metabolism research

Molecules and cells, 2024-02, Vol.47 (2), p.100032-100032, Article 100032

Lee, Jongsoon

2024

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The emerging era of multidisciplinary metabolism research

Molecules and cells, 2024, Vol.47 (2), p.100032.1-100032.2

Jongsoon Lee

2024

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Adipose tissue macrophage heterogeneity in the single-cell genomics era

Molecules and cells, 2024, Vol.47 (2), p.100031.1-100031.13

2024

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Two New Substrates in Insulin Signaling, IRS5/DOK4 and IRS6/DOK5

The Journal of biological chemistry, 2003-07, Vol.278 (28), p.25323-25330

2003

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Self-organized insulin-producing β-cells differentiated from human omentum-derived stem cells and their in vivo therapeutic potential

생체재료학회지, 2023, 27(00), , pp.1999-2016

2023

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The many faces of the creatine/phosphocreatine system

Nature metabolism, 2022-02, Vol.4 (2), p.155-156

2022

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Lean, but not obese, fat is enriched for a unique population of regulatory T cells that affect metabolic parameters

Nature medicine, 2009-08, Vol.15 (8), p.930-939

2009

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Kinase Activation through Dimerization by Human SH2-B

Molecular and Cellular Biology, 2005-04, Vol.25 (7), p.2607-2621

2005

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NK cell-activating receptor NKp46 does not participate in the development of obesity-induced inflammation and insulin resistance

Molecules and cells, 2024, Vol.47 (3), p.100007.1-100007.11

2024

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Externalized phosphatidylinositides on apoptotic cells are eat-me signals recognized by CD14

Cell death and differentiation, 2022-07, Vol.29 (7), p.1423-1432

2022

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Local and systemic insulin resistance resulting from hepatic activation of IKK-β and NF-κB

Nature medicine, 2005-02, Vol.11 (2), p.183-190

2005

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PTB Domains of IRS-1 and Shc Have Distinct but Overlapping Binding Specificities

The Journal of biological chemistry, 1995-11, Vol.270 (46), p.27407-27410

1995

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4331 UPREGULATION OF ADIPOSE TISSUE FATTY ACID-BINDING PROTEIN 4 IN PATIENTS WITH CHRONIC KIDNEY DISEASE: IMPLICATIONS FOR DYSFUNCTIONAL VASCULAR CELLS

Nephrology, dialysis, transplantation, 2023-06, Vol.38 (Supplement_1)

2023

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Unusual Suspects in the Development of Obesity-Induced Inflammation and Insulin Resistance: NK cells, iNKT cells, and ILCs

Diabetes & metabolism journal, 2017-08, Vol.41 (4), p.229

2017

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IKKβ/NF-κB Activation Causes Severe Muscle Wasting in Mice

Cell, 2004-10, Vol.119 (2), p.285-298

2004

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Dynamics of Signaling during Insulin-stimulated Endocytosis of Its Receptor in Adipocytes (∗)

The Journal of biological chemistry, 1995-01, Vol.270 (1), p.59-65

1995

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Retinal not systemic oxidative and inflammatory stress correlated with VEGF expression in rodent models of insulin resistance and diabetes

Investigative ophthalmology & visual science, 2012-12, Vol.53 (13), p.8424-8432

2012

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Self-organized insulin-producing [beta]-cells differentiated from human omentum-derived stem cells and their in vivo therapeutic potential

Biomaterials research, 2023-08, Vol.27 (1)

2023

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Structural Studies of the Detergent-solubilized and Vesicle-reconstituted Insulin Receptor

The Journal of biological chemistry, 1999-12, Vol.274 (49), p.34981-34992

1999

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PPAR-γ is a major driver of the accumulation and phenotype of adipose tissue Treg cells

Nature (London), 2012-06, Vol.486 (7404), p.549-553

2012

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PPAR-γ is a major driver of the accumulation and phenotype of adipose tissue [T.sub.reg] cells

Nature (London), 2012-06, Vol.486 (7404), p.549

2012

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Insulin receptor autophosphorylation occurs asymmetrically

The Journal of biological chemistry, 1993-02, Vol.268 (6), p.4092-4098

1993

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Fat Treg cells: a liaison between the immune and metabolic systems

Nature medicine, 2009-07, Vol.15 (8), p.930-939

2009

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Inflammation and insulin resistance

The Journal of clinical investigation, 2006-08, Vol.116 (8), p.2308-2308

Shoelson, S. E.

2006

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The role of GSK3 in glucose homeostasis and the development of insulin resistance

The emerging era of multidisciplinary metabolism research

The emerging era of multidisciplinary metabolism research

Adipose tissue macrophage heterogeneity in the single-cell genomics era

Two New Substrates in Insulin Signaling, IRS5/DOK4 and IRS6/DOK5

Self-organized insulin-producing β-cells differentiated from human omentum-derived stem cells and their in vivo therapeutic potential

The many faces of the creatine/phosphocreatine system

Lean, but not obese, fat is enriched for a unique population of regulatory T cells that affect metabolic parameters

Kinase Activation through Dimerization by Human SH2-B

NK cell-activating receptor NKp46 does not participate in the development of obesity-induced inflammation and insulin resistance

Externalized phosphatidylinositides on apoptotic cells are eat-me signals recognized by CD14

Local and systemic insulin resistance resulting from hepatic activation of IKK-β and NF-κB

PTB Domains of IRS-1 and Shc Have Distinct but Overlapping Binding Specificities

4331 UPREGULATION OF ADIPOSE TISSUE FATTY ACID-BINDING PROTEIN 4 IN PATIENTS WITH CHRONIC KIDNEY DISEASE: IMPLICATIONS FOR DYSFUNCTIONAL VASCULAR CELLS

Unusual Suspects in the Development of Obesity-Induced Inflammation and Insulin Resistance: NK cells, iNKT cells, and ILCs

IKKβ/NF-κB Activation Causes Severe Muscle Wasting in Mice

Dynamics of Signaling during Insulin-stimulated Endocytosis of Its Receptor in Adipocytes (∗)

Retinal not systemic oxidative and inflammatory stress correlated with VEGF expression in rodent models of insulin resistance and diabetes

Self-organized insulin-producing [beta]-cells differentiated from human omentum-derived stem cells and their in vivo therapeutic potential

Structural Studies of the Detergent-solubilized and Vesicle-reconstituted Insulin Receptor

PPAR-γ is a major driver of the accumulation and phenotype of adipose tissue Treg cells

PPAR-γ is a major driver of the accumulation and phenotype of adipose tissue [T.sub.reg] cells

Insulin receptor autophosphorylation occurs asymmetrically

Fat Treg cells: a liaison between the immune and metabolic systems

Inflammation and insulin resistance

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