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Tolerogenic Dendritic Cells from Poorly Compensated Type 1 Diabetes Patients Have Decreased Ability To Induce Stable Antigen-Specific T Cell Hyporesponsiveness and Generation of Suppressive Regulatory T Cells

The Journal of immunology (1950), 2017-01, Vol.198 (2), p.729-740

2017

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Gliadin Fragments Induce Phenotypic and Functional Maturation of Human Dendritic Cells

Journal of Immunology, 2005-11, Vol.175 (10), p.7038-7045

2005

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Tissue contexture determines the pattern and density of tumor-infiltrating immune cells in HPV-associated squamous cell carcinomas of oropharynx and uterine cervix

Translational oncology, 2024-03, Vol.41, p.101884-101884, Article 101884

2024

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Involvement of Innate Immunity in the Development of Inflammatory and Autoimmune Diseases

Annals of the New York Academy of Sciences, 2005-06, Vol.1051 (1), p.787-798

2005

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Myeloid - derived suppressor cells in Type 1 diabetes are an expanded population exhibiting diverse T-cell suppressor mechanisms

PloS one, 2020-11, Vol.15 (11), p.e0242092-e0242092

2020

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Cell Based Therapy for Type 1 Diabetes: Should We Take Hyperglycemia Into Account?

Frontiers in immunology, 2019-02, Vol.10, p.79-79

2019

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Pepsin digest of wheat gliadin fraction increases production of IL-1β via TLR4/MyD88/TRIF/MAPK/NF-κB signaling pathway and an NLRP3 inflammasome activation

PloS one, 2013-04, Vol.8 (4), p.e62426

2013

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Optimal Tolerogenic Dendritic Cells in Type 1 Diabetes (T1D) Therapy: What Can We Learn From Non-obese Diabetic (NOD) Mouse Models?

Frontiers in immunology, 2019-05, Vol.10, p.967-967

2019

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Antigen Loading (e.g., Glutamic Acid Decarboxylase 65) of Tolerogenic DCs (tolDCs) Reduces Their Capacity to Prevent Diabetes in the Non-Obese Diabetes (NOD)-Severe Combined Immunodeficiency Model of Adoptive Cotransfer of Diabetes As Well As in NOD Mice

Frontiers in immunology, 2018-02, Vol.9, p.290-290

2018

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Gliadin fragments promote migration of dendritic cells

Journal of cellular and molecular medicine, 2011-04, Vol.15 (4), p.938-948

2011

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Myeloid - derived suppressor cells in Type 1 diabetes are an expanded population exhibiting diverse T-cell suppressor mechanisms

PloS one, 2020-01, Vol.15 (11)

2020

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Heat-induced structural changes affect OVA-antigen processing and reduce allergic response in mouse model of food allergy

PloS one, 2012-05, Vol.7 (5), p.e37156-e37156

2012

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NF-κB, p38 MAPK, ERK1/2, mTOR, STAT3 and increased glycolysis regulate stability of paricalcitol/dexamethasone-generated tolerogenic dendritic cells in the inflammatory environment

Oncotarget, 2015-06, Vol.6 (16), p.14123-14138

2015

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Data on microbial DNA-induced IL-1β production in monocytes of type 1 diabetes patients

Data in brief, 2019-08, Vol.25, p.104321-104321, Article 104321

2019

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Gliadin peptides activate blood monocytes from patients with celiac disease

Journal of Clinical Immunology, 2007-03, Vol.27 (2), p.201-209

2007

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Monocytes contribute to DNA sensing through the TBK1 signaling pathway in type 1 diabetes patients

Journal of autoimmunity, 2019-12, Vol.105, p.102294-102294, Article 102294

2019

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Interim safety and efficacy results from AURELIO-03: A phase 1 dose escalation study of the IL-2/IL-15 receptor βγ superagonist SOT101 as a single agent and in combination with pembrolizumab in patients with advanced solid tumors

Journal of clinical oncology, 2022-06, Vol.40 (16_suppl), p.2502-2502

2022

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Immunological control of ovarian carcinoma by chemotherapy and targeted anticancer agents

Trends in cancer, 2022-05, Vol.8 (5), p.426-444

2022

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Methods to assess DC-dependent priming of T cell responses by dying cells

Methods in enzymology, 2020, Vol.632, p.55-65

2020

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Pepsin Digest of Wheat Gliadin Fraction Increases Production of IL-1[beta] via TLR4/MyD88/TRIF/MAPK/NF-[kappa]B Signaling Pathway and an NLRP3 Inflammasome Activation

PloS one, 2013-04, Vol.8 (4), p.e62426

2013

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Correction: Heat-Induced Structural Changes Affect OVA-Antigen Processing and Reduce Allergic Response in Mouse Model of Food Allergy

PloS one, 2012-08, Vol.7 (8)

2012

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Abstract CT040: SOT101, an IL-2/IL-15 Rβγ superagonist, in combination with pembrolizumab in patients with advanced solid tumors: Interim safety and efficacy results from the AURELIO-03 dose escalation trial

Cancer research (Chicago, Ill.), 2022-06, Vol.82 (12_Supplement), p.CT040-CT040

2022

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Pepsin Digest of Wheat Gliadin Fraction Increases Production of IL-1 beta via TLR4/MyD88/TRIF/MAPK/NF- Kappa B Signaling Pathway and an NLRP3 Inflammasome Activation. e62426

PloS one, 2013-04, Vol.8 (4)

2013

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Correction: Heat-Induced Structural Changes Affect OVA-Antigen Processing and Reduce Allergic Response in Mouse Model of Food Allergy

PloS one, 2012-01, Vol.7 (8)

2012

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Tolerogenic Dendritic Cells from Poorly Compensated Type 1 Diabetes Patients Have Decreased Ability To Induce Stable Antigen-Specific T Cell Hyporesponsiveness and Generation of Suppressive Regulatory T Cells

Gliadin Fragments Induce Phenotypic and Functional Maturation of Human Dendritic Cells

Tissue contexture determines the pattern and density of tumor-infiltrating immune cells in HPV-associated squamous cell carcinomas of oropharynx and uterine cervix

Involvement of Innate Immunity in the Development of Inflammatory and Autoimmune Diseases

Myeloid - derived suppressor cells in Type 1 diabetes are an expanded population exhibiting diverse T-cell suppressor mechanisms

Cell Based Therapy for Type 1 Diabetes: Should We Take Hyperglycemia Into Account?

Pepsin digest of wheat gliadin fraction increases production of IL-1β via TLR4/MyD88/TRIF/MAPK/NF-κB signaling pathway and an NLRP3 inflammasome activation

Optimal Tolerogenic Dendritic Cells in Type 1 Diabetes (T1D) Therapy: What Can We Learn From Non-obese Diabetic (NOD) Mouse Models?

Antigen Loading (e.g., Glutamic Acid Decarboxylase 65) of Tolerogenic DCs (tolDCs) Reduces Their Capacity to Prevent Diabetes in the Non-Obese Diabetes (NOD)-Severe Combined Immunodeficiency Model of Adoptive Cotransfer of Diabetes As Well As in NOD Mice

Gliadin fragments promote migration of dendritic cells

Myeloid - derived suppressor cells in Type 1 diabetes are an expanded population exhibiting diverse T-cell suppressor mechanisms

Heat-induced structural changes affect OVA-antigen processing and reduce allergic response in mouse model of food allergy

NF-κB, p38 MAPK, ERK1/2, mTOR, STAT3 and increased glycolysis regulate stability of paricalcitol/dexamethasone-generated tolerogenic dendritic cells in the inflammatory environment

Data on microbial DNA-induced IL-1β production in monocytes of type 1 diabetes patients

Gliadin peptides activate blood monocytes from patients with celiac disease

Monocytes contribute to DNA sensing through the TBK1 signaling pathway in type 1 diabetes patients

Interim safety and efficacy results from AURELIO-03: A phase 1 dose escalation study of the IL-2/IL-15 receptor βγ superagonist SOT101 as a single agent and in combination with pembrolizumab in patients with advanced solid tumors

Immunological control of ovarian carcinoma by chemotherapy and targeted anticancer agents

Methods to assess DC-dependent priming of T cell responses by dying cells

Pepsin Digest of Wheat Gliadin Fraction Increases Production of IL-1[beta] via TLR4/MyD88/TRIF/MAPK/NF-[kappa]B Signaling Pathway and an NLRP3 Inflammasome Activation

Correction: Heat-Induced Structural Changes Affect OVA-Antigen Processing and Reduce Allergic Response in Mouse Model of Food Allergy

Abstract CT040: SOT101, an IL-2/IL-15 Rβγ superagonist, in combination with pembrolizumab in patients with advanced solid tumors: Interim safety and efficacy results from the AURELIO-03 dose escalation trial

Pepsin Digest of Wheat Gliadin Fraction Increases Production of IL-1 beta via TLR4/MyD88/TRIF/MAPK/NF- Kappa B Signaling Pathway and an NLRP3 Inflammasome Activation. e62426

Correction: Heat-Induced Structural Changes Affect OVA-Antigen Processing and Reduce Allergic Response in Mouse Model of Food Allergy

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