UB Paderborn / Katalog / Suche / in CDI

1 – 25 von 37

Sortiert nach

Studies in a Murine Granuloma Model of Instilled Carbon Nanotubes: Relevance to Sarcoidosis

International journal of molecular sciences, 2021-04, Vol.22 (7), p.3705

2021

Volltextzugriff (PDF)

Myeloid ABCG1 Deficiency Enhances Apoptosis and Initiates Efferocytosis in Bronchoalveolar Lavage Cells of Murine Multi-Walled Carbon Nanotube-Induced Granuloma Model

International journal of molecular sciences, 2021-12, Vol.23 (1), p.47

2021

Volltextzugriff (PDF)

PPAR-gamma pathways attenuate pulmonary granuloma formation in a carbon nanotube induced murine model of sarcoidosis

Biochemical and biophysical research communications, 2018-09, Vol.503 (2), p.684-690

2018

Volltextzugriff (PDF)

The M2a Macrophage Phenotype Accompanies Pulmonary Granuloma Resolution in Mmp12 Knock-Out Mice Instilled with Multiwall Carbon Nanotubes

International journal of molecular sciences, 2021-10, Vol.22 (20), p.11019

2021

Volltextzugriff (PDF)

Peroxisome Proliferator-activated Receptor-γ Deficiency Exacerbates Fibrotic Response to Mycobacteria Peptide in Murine Sarcoidosis Model

American journal of respiratory cell and molecular biology, 2019-08, Vol.61 (2), p.198-208

2019

Volltextzugriff (PDF)

Alveolar Macrophage ABCG1 Deficiency Promotes Pulmonary Granulomatous Inflammation

American journal of respiratory cell and molecular biology, 2019-09, Vol.61 (3), p.332-340

2019

Volltextzugriff (PDF)

Targeted PPARγ deficiency in alveolar macrophages disrupts surfactant catabolism

Journal of lipid research, 2010-06, Vol.51 (6), p.1325-1331

2010

Volltextzugriff (PDF)

Matrix Metalloproteinase-12 Is Required for Granuloma Progression

Frontiers in immunology, 2020-09, Vol.11, p.553949

2020

Volltextzugriff (PDF)

The role of PPARγ in carbon nanotube-elicited granulomatous lung inflammation

Respiratory research, 2013-01, Vol.14 (1), p.7-7, Article 7

2013

Volltextzugriff (PDF)

Impaired mitochondrial function of alveolar macrophages in carbon nanotube-induced chronic pulmonary granulomatous disease

Toxicology (Amsterdam), 2020-12, Vol.445, p.152598-152598, Article 152598

2020

Volltextzugriff (PDF)

Hepatic Steatosis Accompanies Pulmonary Alveolar Proteinosis

American journal of respiratory cell and molecular biology, 2017-10, Vol.57 (4), p.448-458

2017

Volltextzugriff (PDF)

PPARγ regulates the expression of cholesterol metabolism genes in alveolar macrophages

Biochemical and biophysical research communications, 2010-03, Vol.393 (4), p.682-687

2010

Volltextzugriff (PDF)

Increased Th1 cells and enhanced pulmonary fibrosis in macrophage-specific PPARγ KO mice instilled with multiwall carbon nanotubes and ESAT-6 mycobacterial antigen

The Journal of immunology (1950), 2017-05, Vol.198 (1_Supplement), p.153-153.12

2017

Volltextzugriff (PDF)

PU.1 regulation of human alveolar macrophage differentiation requires granulocyte-macrophage colony-stimulating factor

American journal of physiology. Lung cellular and molecular physiology, 2003-11, Vol.29 (5), p.L1132-L1136

2003

Volltextzugriff (PDF)

Novel murine model of chronic granulomatous lung inflammation elicited by carbon nanotubes

American journal of respiratory cell and molecular biology, 2011-10, Vol.45 (4), p.858-866

2011

Volltextzugriff (PDF)

Surfactant Blocks Lipopolysaccharide Signaling by Inhibiting both Mitogen-Activated Protein and I{kappa}B Kinases in Human Alveolar Macrophages

American journal of respiratory cell and molecular biology, 2004-02, Vol.30 (2), p.228

2004

Volltextzugriff (PDF)

Growth Hormone Expression in Murine Bone Marrow Cells Is Independent of the Pituitary Transcription Factor Pit-1

Endocrinology (Philadelphia), 1997-09, Vol.138 (9), p.3949-3955

1997

Volltextzugriff (PDF)

Peroxisome Proliferator-Activated Receptor-{gamma} Is Deficient in Alveolar Macrophages from Patients with Alveolar Proteinosis

American journal of respiratory cell and molecular biology, 2003-12, Vol.29 (6), p.677

2003

Volltextzugriff (PDF)

Utilization of LC-MS to Determine Monoclonal Gammopathy-Associated Granulocyte Macrophage Colony Stimulating Factor Antibody and Novel Treatment of Pulmonary Alveolar Proteinosis

The journal of applied laboratory medicine, 2020-03, Vol.5 (2), p.394-400

2020

Volltextzugriff (PDF)

The role of PPAR[gamma] in carbon nanotube-elicited granulomatous lung inflammation

Respiratory research, 2013-01, Vol.14 (1), p.7-7

2013

Volltextzugriff (PDF)

PPARI[sup3 regulates the expression of cholesterol metabolism genes in alveolar macrophages

Biochemical and biophysical research communications, 2010-03, Vol.393 (4), p.682-687

2010

Volltextzugriff (PDF)

Restoration of PPARγ reverses lipid accumulation in alveolar macrophages of GM-CSF knockout mice

American journal of physiology. Lung cellular and molecular physiology, 2011, Vol.44 (1)

2011

Volltextzugriff (PDF)

Restoration of PPAR[gamma] reverses lipid accumulation in alveolar macrophages of GM-CSF knockout mice

American journal of physiology. Lung cellular and molecular physiology, 2011-01, Vol.300 (1), p.L73

2011

Volltextzugriff (PDF)

PPAR{gamma} regulates the expression of cholesterol metabolism genes in alveolar macrophages

Biochemical and biophysical research communications, 2010-03, Vol.393 (4)

2010

Volltextzugriff (PDF)

DEFICIENCIES OF CATHELICIDIN AND VITAMIN D ACCOMPANY DISEASE SEVERITY IN SARCOIDOSIS

Chest, 2009-10, Vol.136 (4), p.127S-127S

2009

Volltextzugriff (PDF)

1 – 25 von 37

Suche als RSS-Feed

Aktive Filter

ThemaHumans

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(37)
Mit Volltextzugriff
(37)

Publikationsform

Artikel
(37)

Sprache

Englisch
(37)
Japanisch
(4)

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

Studies in a Murine Granuloma Model of Instilled Carbon Nanotubes: Relevance to Sarcoidosis

Myeloid ABCG1 Deficiency Enhances Apoptosis and Initiates Efferocytosis in Bronchoalveolar Lavage Cells of Murine Multi-Walled Carbon Nanotube-Induced Granuloma Model

PPAR-gamma pathways attenuate pulmonary granuloma formation in a carbon nanotube induced murine model of sarcoidosis

The M2a Macrophage Phenotype Accompanies Pulmonary Granuloma Resolution in Mmp12 Knock-Out Mice Instilled with Multiwall Carbon Nanotubes

Peroxisome Proliferator-activated Receptor-γ Deficiency Exacerbates Fibrotic Response to Mycobacteria Peptide in Murine Sarcoidosis Model

Alveolar Macrophage ABCG1 Deficiency Promotes Pulmonary Granulomatous Inflammation

Targeted PPARγ deficiency in alveolar macrophages disrupts surfactant catabolism

Matrix Metalloproteinase-12 Is Required for Granuloma Progression

The role of PPARγ in carbon nanotube-elicited granulomatous lung inflammation

Impaired mitochondrial function of alveolar macrophages in carbon nanotube-induced chronic pulmonary granulomatous disease

Hepatic Steatosis Accompanies Pulmonary Alveolar Proteinosis

PPARγ regulates the expression of cholesterol metabolism genes in alveolar macrophages

Increased Th1 cells and enhanced pulmonary fibrosis in macrophage-specific PPARγ KO mice instilled with multiwall carbon nanotubes and ESAT-6 mycobacterial antigen

PU.1 regulation of human alveolar macrophage differentiation requires granulocyte-macrophage colony-stimulating factor

Novel murine model of chronic granulomatous lung inflammation elicited by carbon nanotubes

Surfactant Blocks Lipopolysaccharide Signaling by Inhibiting both Mitogen-Activated Protein and I{kappa}B Kinases in Human Alveolar Macrophages

Growth Hormone Expression in Murine Bone Marrow Cells Is Independent of the Pituitary Transcription Factor Pit-1

Peroxisome Proliferator-Activated Receptor-{gamma} Is Deficient in Alveolar Macrophages from Patients with Alveolar Proteinosis

Utilization of LC-MS to Determine Monoclonal Gammopathy-Associated Granulocyte Macrophage Colony Stimulating Factor Antibody and Novel Treatment of Pulmonary Alveolar Proteinosis

The role of PPAR[gamma] in carbon nanotube-elicited granulomatous lung inflammation

PPARI[sup3 regulates the expression of cholesterol metabolism genes in alveolar macrophages

Restoration of PPARγ reverses lipid accumulation in alveolar macrophages of GM-CSF knockout mice

Restoration of PPAR[gamma] reverses lipid accumulation in alveolar macrophages of GM-CSF knockout mice

PPAR{gamma} regulates the expression of cholesterol metabolism genes in alveolar macrophages

DEFICIENCIES OF CATHELICIDIN AND VITAMIN D ACCOMPANY DISEASE SEVERITY IN SARCOIDOSIS

Aktive Filter

Suchergebnisse filtern