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HDBR Expression: A Unique Resource for Global and Individual Gene Expression Studies during Early Human Brain Development

Frontiers in neuroanatomy, 2016-10, Vol.10, p.86-86

2016

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Cell non-autonomy amplifies disruption of neurulation by mosaic Vangl2 deletion in mice

Nature communications, 2021-02, Vol.12 (1), p.1159-17, Article 1159

2021

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Glycine decarboxylase deficiency causes neural tube defects and features of non-ketotic hyperglycinemia in mice

Nature communications, 2015-03, Vol.6 (1), p.6388-6388, Article 6388

2015

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Hindbrain neuropore tissue geometry determines asymmetric cell-mediated closure dynamics in mouse embryos

Proceedings of the National Academy of Sciences - PNAS, 2021-05, Vol.118 (19), p.1

2021

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Rho-kinase-dependent actin turnover and actomyosin disassembly are necessary for mouse spinal neural tube closure

Journal of cell science, 2015-07, Vol.128 (14), p.2468-2481

2015

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Partitioning of One-Carbon Units in Folate and Methionine Metabolism Is Essential for Neural Tube Closure

Cell reports (Cambridge), 2017-11, Vol.21 (7), p.1795-1808

2017

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Spinal neural tube closure depends on regulation of surface ectoderm identity and biomechanics by Grhl2

Nature communications, 2019-06, Vol.10 (1), p.2487-2487, Article 2487

2019

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Convergent extension, planar-cell-polarity signalling and initiation of mouse neural tube closure

Development (Cambridge), 2007-02, Vol.134 (4), p.789-799

2007

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Author Correction: Cell non-autonomy amplifies disruption of neurulation by mosaic Vangl2 deletion in mice

Nature communications, 2021-05, Vol.12 (1), p.3301-3301, Article 3301

2021

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Vangl2 disruption alters the biomechanics of late spinal neurulation leading to spina bifida in mouse embryos

Disease models & mechanisms, 2018-03, Vol.11 (3)

2018

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Knockout of the PKN Family of Rho Effector Kinases Reveals a Non-redundant Role for PKN2 in Developmental Mesoderm Expansion

Cell reports (Cambridge), 2016-01, Vol.14 (3), p.440-448

2016

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Dual mechanism underlying failure of neural tube closure in the Zic2 mutant mouse

Disease models & mechanisms, 2023-03, Vol.16 (3)

2023

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Vangl2-environment interaction causes severe neural tube defects, without abnormal neuroepithelial convergent extension

Disease models & mechanisms, 2022-01, Vol.15 (1)

2022

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Morphological phenotyping after mouse whole embryo culture

Frontiers in cell and developmental biology, 2023-08, Vol.11, p.1223849

2023

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Cellular mechanisms underlying Pax3- related neural tube defects and their prevention by folic acid

Disease models & mechanisms, 2019-11, Vol.12 (11)

2019

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Fetal spina bifida in a mouse model: loss of neural function in utero

Journal of neurosurgery, 2007-03, Vol.106 (3 Suppl), p.213-221

2007

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Regulation of cell protrusions by small GTPases during fusion of the neural folds

eLife, 2016-04, Vol.5, p.e13273-e13273

2016

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Genetic interactions between planar cell polarity genes cause diverse neural tube defects in mice

Disease models & mechanisms, 2014-10, Vol.7 (10), p.1153-1163

2014

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Mutation of Celsr1 Disrupts Planar Polarity of Inner Ear Hair Cells and Causes Severe Neural Tube Defects in the Mouse

Current biology, 2003-07, Vol.13 (13), p.1129-1133

2003

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Glycine Cleavage System H Protein Is Essential for Embryonic Viability, Implying Additional Function Beyond the Glycine Cleavage System

Frontiers in genetics, 2021-01, Vol.12, p.625120-625120

2021

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Dolutegravir in pregnant mice is associated with increased rates of fetal defects at therapeutic but not at supratherapeutic levels

EBioMedicine, 2021-01, Vol.63, p.103167-103167, Article 103167

2021

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Neural plate morphogenesis during mouse neurulation is regulated by antagonism of Bmp signalling

Development (Cambridge), 2007-09, Vol.134 (17), p.3203-3211

2007

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In vivo genetic ablation by Cre-mediated expression of diphtheria toxin fragment A

Genesis (New York, N.Y. : 2000), 2005-11, Vol.43 (3), p.129-135

2005

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Apoptosis is not required for mammalian neural tube closure

Proceedings of the National Academy of Sciences - PNAS, 2009-05, Vol.106 (20), p.8233-8238

2009

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Regulation of glycine metabolism by the glycine cleavage system and conjugation pathway in mouse models of non‐ketotic hyperglycinemia

Journal of inherited metabolic disease, 2020-11, Vol.43 (6), p.1186-1198

2020

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HDBR Expression: A Unique Resource for Global and Individual Gene Expression Studies during Early Human Brain Development

Cell non-autonomy amplifies disruption of neurulation by mosaic Vangl2 deletion in mice

Glycine decarboxylase deficiency causes neural tube defects and features of non-ketotic hyperglycinemia in mice

Hindbrain neuropore tissue geometry determines asymmetric cell-mediated closure dynamics in mouse embryos

Rho-kinase-dependent actin turnover and actomyosin disassembly are necessary for mouse spinal neural tube closure

Partitioning of One-Carbon Units in Folate and Methionine Metabolism Is Essential for Neural Tube Closure

Spinal neural tube closure depends on regulation of surface ectoderm identity and biomechanics by Grhl2

Convergent extension, planar-cell-polarity signalling and initiation of mouse neural tube closure

Author Correction: Cell non-autonomy amplifies disruption of neurulation by mosaic Vangl2 deletion in mice

Vangl2 disruption alters the biomechanics of late spinal neurulation leading to spina bifida in mouse embryos

Knockout of the PKN Family of Rho Effector Kinases Reveals a Non-redundant Role for PKN2 in Developmental Mesoderm Expansion

Dual mechanism underlying failure of neural tube closure in the Zic2 mutant mouse

Vangl2-environment interaction causes severe neural tube defects, without abnormal neuroepithelial convergent extension

Morphological phenotyping after mouse whole embryo culture

Cellular mechanisms underlying Pax3- related neural tube defects and their prevention by folic acid

Fetal spina bifida in a mouse model: loss of neural function in utero

Regulation of cell protrusions by small GTPases during fusion of the neural folds

Genetic interactions between planar cell polarity genes cause diverse neural tube defects in mice

Mutation of Celsr1 Disrupts Planar Polarity of Inner Ear Hair Cells and Causes Severe Neural Tube Defects in the Mouse

Glycine Cleavage System H Protein Is Essential for Embryonic Viability, Implying Additional Function Beyond the Glycine Cleavage System

Dolutegravir in pregnant mice is associated with increased rates of fetal defects at therapeutic but not at supratherapeutic levels

Neural plate morphogenesis during mouse neurulation is regulated by antagonism of Bmp signalling

In vivo genetic ablation by Cre-mediated expression of diphtheria toxin fragment A

Apoptosis is not required for mammalian neural tube closure

Regulation of glycine metabolism by the glycine cleavage system and conjugation pathway in mouse models of non‐ketotic hyperglycinemia

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