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Activation of caspase-3 is an initial step triggering accelerated muscle proteolysis in catabolic conditions

The Journal of clinical investigation, 2004-01, Vol.113 (1), p.115-123

2004

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Activation of caspase-3 is an initial step triggering accelerated muscle proteolysis in catabolic conditions

The Journal of clinical investigation, 2004-01, Vol.113 (1), p.115-123

2004

Volltextzugriff (PDF)

Pulmonary rehabilitation in Canada: A report from the Canadian Thoracic Society COPD Clinical Assembly

Canadian respiratory journal, 2015-05, Vol.22 (3), p.147-152

2015

Volltextzugriff (PDF)

An official American Thoracic Society/European Respiratory Society statement: update on limb muscle dysfunction in chronic obstructive pulmonary disease

American journal of respiratory and critical care medicine, 2014-05, Vol.189 (9), p.e15-e62

2014

Volltextzugriff (PDF)

Optimizing pulmonary rehabilitation in chronic obstructive pulmonary disease--practical issues: a Canadian Thoracic Society Clinical Practice Guideline

Canadian respiratory journal, 2010-07, Vol.17 (4), p.159-168

2010

Volltextzugriff (PDF)

The major limitation to exercise performance in COPD is lower limb muscle dysfunction

Journal of applied physiology (1985), 2008-08, Vol.105 (2), p.751-753

2008

Volltextzugriff (PDF)

Hypoxia upregulates Malat1 expression through a CaMKK/AMPK/HIF-1α axis

International journal of oncology, 2016-10, Vol.49 (4), p.1731-1736

2016

Volltextzugriff (PDF)

Exploring if and how evidence-based practice of occupational and physical therapists evolves over time: A longitudinal mixed methods national study

PloS one, 2023-03, Vol.18 (3), p.e0283860-e0283860

2023

Volltextzugriff (PDF)

Developing multisectoral strategies to promote evidence-based practice in rehabilitation: findings from an end-of-grant knowledge translation symposium

Disability and rehabilitation, 2024-06, Vol.ahead-of-print (ahead-of-print), p.1-12

2024

Volltextzugriff (PDF)

Satellite cells senescence in limb muscle of severe patients with COPD

PloS one, 2012-06, Vol.7 (6), p.e39124-e39124

2012

Volltextzugriff (PDF)

Alterations in skeletal muscle cell homeostasis in a mouse model of cigarette smoke exposure

PloS one, 2013-06, Vol.8 (6), p.e66433

2013

Volltextzugriff (PDF)

MAPK signaling in the quadriceps of patients with chronic obstructive pulmonary disease

Journal of applied physiology (1985), 2012-07, Vol.113 (1), p.159-166

2012

Volltextzugriff (PDF)

Comparative assessment of the quadriceps and the diaphragm in patients with COPD

Journal of applied physiology (1985), 2009-09, Vol.107 (3), p.952-961

2009

Volltextzugriff (PDF)

Should all patients with COPD be exercise trained?

Journal of applied physiology (1985), 2013-05, Vol.114 (9), p.1300-1308

2013

Volltextzugriff (PDF)

Regenerative defect in vastus lateralis muscle of patients with chronic obstructive pulmonary disease

Respiratory research, 2014-03, Vol.15 (1), p.35-35, Article 35

2014

Volltextzugriff (PDF)

Physiology of Walking in Patients with Moderate to Severe Chronic Obstructive Pulmonary Disease

Medicine and science in sports and exercise, 2009-08, Vol.41 (8), p.1540-1548

2009

Volltextzugriff (PDF)

CD34 regulates the skeletal muscle response to hypoxia

Journal of muscle research and cell motility, 2019-12, Vol.40 (3-4), p.309-318

2019

Volltextzugriff (PDF)

Atrophy and hypertrophy signalling of the quadriceps and diaphragm in COPD

Thorax, 2010-11, Vol.65 (11), p.963-970

2010

Volltextzugriff (PDF)

Preserved function and reduced angiogenesis potential of the quadriceps in patients with mild COPD

Respiratory research, 2014-01, Vol.15 (1), p.4-4, Article 4

2014

Volltextzugriff (PDF)

Last Word on Point:Counterpoint: The major limitation to exercise performance in COPD is 1) inadequate energy supply to the respiratory and locomotor muscles, 2) lower limb muscle dysfunction, 3) dynamic hyperinflation

Journal of applied physiology (1985), 2008-08, Vol.105 (2), p.764-764

2008

Volltextzugriff (PDF)

Quadriceps metabolism during constant workrate cycling exercise in chronic obstructive pulmonary disease

Journal of applied physiology (1985), 2011, Vol.110 (1), p.116-124

2011

Volltextzugriff (PDF)

Hypoxia upregulates Malat1 expression through a CaMKK/AMPK/HIF-1[alpha] axis.(metastasis-associated lung adenocarcinoma transcript 1; hypoxia inducible factor; adenosine monophosphate-activated protein kinase; and calcium/calmodulin-dependent protein kinase kinase)

International journal of oncology, 2016-10, Vol.49 (4), p.1731

2016

Volltextzugriff (PDF)

Hypoxia alters contractile protein homeostasis in L6 myotubes

FEBS letters, 2009-05, Vol.583 (9), p.1528-1534

2009

Volltextzugriff (PDF)

Proteolysis, the ubiquitin-proteasome system, and renal diseases

American journal of physiology. Renal physiology, 2003-07, Vol.285 (1), p.F1-F8

2003

Volltextzugriff (PDF)

Angiogenesis-related factors in skeletal muscles of COPD patients: roles of angiopoietin-2

Journal of applied physiology (1985), 2013-05, Vol.114 (9), p.1309-1318

2013

Volltextzugriff (PDF)

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Activation of caspase-3 is an initial step triggering accelerated muscle proteolysis in catabolic conditions

Activation of caspase-3 is an initial step triggering accelerated muscle proteolysis in catabolic conditions

Pulmonary rehabilitation in Canada: A report from the Canadian Thoracic Society COPD Clinical Assembly

An official American Thoracic Society/European Respiratory Society statement: update on limb muscle dysfunction in chronic obstructive pulmonary disease

Optimizing pulmonary rehabilitation in chronic obstructive pulmonary disease--practical issues: a Canadian Thoracic Society Clinical Practice Guideline

The major limitation to exercise performance in COPD is lower limb muscle dysfunction

Hypoxia upregulates Malat1 expression through a CaMKK/AMPK/HIF-1α axis

Exploring if and how evidence-based practice of occupational and physical therapists evolves over time: A longitudinal mixed methods national study

Developing multisectoral strategies to promote evidence-based practice in rehabilitation: findings from an end-of-grant knowledge translation symposium

Satellite cells senescence in limb muscle of severe patients with COPD

Alterations in skeletal muscle cell homeostasis in a mouse model of cigarette smoke exposure

MAPK signaling in the quadriceps of patients with chronic obstructive pulmonary disease

Comparative assessment of the quadriceps and the diaphragm in patients with COPD

Should all patients with COPD be exercise trained?

Regenerative defect in vastus lateralis muscle of patients with chronic obstructive pulmonary disease

Physiology of Walking in Patients with Moderate to Severe Chronic Obstructive Pulmonary Disease

CD34 regulates the skeletal muscle response to hypoxia

Atrophy and hypertrophy signalling of the quadriceps and diaphragm in COPD

Preserved function and reduced angiogenesis potential of the quadriceps in patients with mild COPD

Last Word on Point:Counterpoint: The major limitation to exercise performance in COPD is 1) inadequate energy supply to the respiratory and locomotor muscles, 2) lower limb muscle dysfunction, 3) dynamic hyperinflation

Quadriceps metabolism during constant workrate cycling exercise in chronic obstructive pulmonary disease

Hypoxia upregulates Malat1 expression through a CaMKK/AMPK/HIF-1[alpha] axis.(metastasis-associated lung adenocarcinoma transcript 1; hypoxia inducible factor; adenosine monophosphate-activated protein kinase; and calcium/calmodulin-dependent protein kinase kinase)

Hypoxia alters contractile protein homeostasis in L6 myotubes

Proteolysis, the ubiquitin-proteasome system, and renal diseases

Angiogenesis-related factors in skeletal muscles of COPD patients: roles of angiopoietin-2

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