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Why Alzheimer trials fail: removing soluble oligomeric beta amyloid is essential, inconsistent, and difficult

Neurobiology of aging, 2014-05, Vol.35 (5), p.969-974

Rosenblum, William I

2014

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Fibrinoid necrosis of small brain arteries and arterioles and miliary aneurysms as causes of hypertensive hemorrhage: a critical reappraisal

Acta neuropathologica, 2008-10, Vol.116 (4), p.361-369

Rosenblum, William I.

2008

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Lipohyalinosis and Fibrinoid: Consistent and Important Failure to Understand These Are Synonyms

Journal of neuropathology and experimental neurology, 2021-08, Vol.80 (8), p.802-803

Rosenblum, William I

2021

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Immediate, Irreversible, Posttraumatic Coma: A Review Indicating That Bilateral Brainstem Injury Rather Than Widespread Hemispheric Damage Is Essential for Its Production

Journal of neuropathology and experimental neurology, 2015-03, Vol.74 (3), p.198-202

Rosenblum, William I

2015

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ATP-sensitive potassium channels in the cerebral circulation

Stroke (1970), 2003-06, Vol.34 (6), p.1547-1552

ROSENBLUM, William I

2003

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Cytotoxic Edema: Monitoring Its Magnitude and Contribution to Brain Swelling

Journal of neuropathology and experimental neurology, 2007-09, Vol.66 (9), p.771-778

Rosenblum, William I

2007

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Structure and location of amyloid beta peptide chains and arrays in Alzheimer's disease: new findings require reevaluation of the amyloid hypothesis and of tests of the hypothesis

Neurobiology of aging, 2002-03, Vol.23 (2), p.225-230

Rosenblum, William I

2002

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Cerebral hemorrhage produced by ruptured dissecting aneurysm in miliary aneurysm

Annals of neurology, 2003-09, Vol.54 (3), p.376-378

Rosenblum, William I.

2003

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Immediate, Irreversible, Posttraumatic Coma

Journal of neuropathology and experimental neurology, 2015-03, Vol.74 (3), p.198-202

Rosenblum, William I.

2015

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The Presence, Origin, and Significance of Aβ Peptide in the Cell Bodies of Neurons

Journal of neuropathology and experimental neurology, 1999-06, Vol.58 (6), p.575-581

Rosenblum, William I

1999

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Vasodilation of brain surface arterioles by blockade of Na-H+ antiport and its inhibition by inhibitors of KATP channel openers

Brain research, 2004-04, Vol.1005 (1-2), p.77-83

2004

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Dimethylsulfoxide and ethanol, commonly used diluents, prevent dilation of pial arterioles by openers of KATP ion channels

European journal of pharmacology, 2001-10, Vol.430 (1), p.101-106

2001

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Platelet adhesion and aggregation without endothelial denudation or exposure of basal lamina and/or collagen

Journal of vascular research, 1997-11, Vol.34 (6), p.409-417

Rosenblum, W I

1997

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Evidence for a KATP ion channel link in the inhibition of hypercapnic dilation of pial arterioles by 7-nitroindazole and tetrodotoxin

European journal of pharmacology, 2001-04, Vol.417 (3), p.203-215

2001

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Failure of standards in reporting the composition of artificial cerebral spinal fluid in studies of the pial blood vessels

Stroke (1970), 2000-05, Vol.31 (5), p.1194-1198

Rosenblum, W I

2000

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Selective impairment of response to acetylcholine after ischemia/reperfusion in mice

Stroke (1970), 1997-02, Vol.28 (2), p.448-452

Rosenblum, W I

1997

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Role of platelet-endothelial cell adhesion molecule (PECAM) in platelet adhesion/aggregation over injured but not denuded endothelium in vivo and ex vivo

Stroke (1970), 1996-04, Vol.27 (4), p.709-711

1996

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Dilation of rat brain arterioles by hypercapnia in vivo can occur even after blockade of guanylate cyclase by ODQ

European journal of pharmacology, 2002-07, Vol.448 (2), p.201-206

2002

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Tetrahydrobiopterin, a cofactor for nitric oxide synthase, produces endothelium-dependent dilation of mouse pial arterioles

Stroke (1970), 1997-01, Vol.28 (1), p.186-189

Rosenblum, W I

1997

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Antisense Evidence for Two Functionally Active Forms of Nitric Oxide Synthase in Brain Microvascular Endothelium

Biochemical and biophysical research communications, 1996-07, Vol.224 (2), p.535-543

1996

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Re: Role of endothelial nitric oxide and smooth muscle potassium channels in cerebral arteriolar dilation in response to acidosis

Stroke (1970), 2002-07, Vol.33 (7), p.1742-1743

Rosenblum, William I

2002

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Re: Disconnection of the Ascending Arousal System in Traumatic Coma. J Neuropathol Exp Neurol 2013;72:505-523/Author's Reply

Journal of neuropathology and experimental neurology, 2014-03, Vol.73 (3), p.268

2014

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Selective depression of endothelium-dependent dilations during cerebral ischemia

Stroke (1970), 1995-10, Vol.26 (10), p.1877-1882

1995

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A review of vasomotor responses of arterioles on the surface of the mouse brain: the necessary prelude to studies using genetically manipulated mice

Microcirculation (New York, N.Y. 1994), 1998, Vol.5 (2-3), p.129-138

Rosenblum, W I

1998

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Is the EDRF in the Cerebral Circulation NO? Its Release by Shear and the Dangers in Interpreting the Effects of NOS Inhibitors

The Keio Journal of Medicine, 1998, Vol.47(3), pp.142-149

Rosenblum, William I.

1998

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Why Alzheimer trials fail: removing soluble oligomeric beta amyloid is essential, inconsistent, and difficult

Fibrinoid necrosis of small brain arteries and arterioles and miliary aneurysms as causes of hypertensive hemorrhage: a critical reappraisal

Lipohyalinosis and Fibrinoid: Consistent and Important Failure to Understand These Are Synonyms

Immediate, Irreversible, Posttraumatic Coma: A Review Indicating That Bilateral Brainstem Injury Rather Than Widespread Hemispheric Damage Is Essential for Its Production

ATP-sensitive potassium channels in the cerebral circulation

Cytotoxic Edema: Monitoring Its Magnitude and Contribution to Brain Swelling

Structure and location of amyloid beta peptide chains and arrays in Alzheimer's disease: new findings require reevaluation of the amyloid hypothesis and of tests of the hypothesis

Cerebral hemorrhage produced by ruptured dissecting aneurysm in miliary aneurysm

Immediate, Irreversible, Posttraumatic Coma

The Presence, Origin, and Significance of Aβ Peptide in the Cell Bodies of Neurons

Vasodilation of brain surface arterioles by blockade of Na-H+ antiport and its inhibition by inhibitors of KATP channel openers

Dimethylsulfoxide and ethanol, commonly used diluents, prevent dilation of pial arterioles by openers of KATP ion channels

Platelet adhesion and aggregation without endothelial denudation or exposure of basal lamina and/or collagen

Evidence for a KATP ion channel link in the inhibition of hypercapnic dilation of pial arterioles by 7-nitroindazole and tetrodotoxin

Failure of standards in reporting the composition of artificial cerebral spinal fluid in studies of the pial blood vessels

Selective impairment of response to acetylcholine after ischemia/reperfusion in mice

Role of platelet-endothelial cell adhesion molecule (PECAM) in platelet adhesion/aggregation over injured but not denuded endothelium in vivo and ex vivo

Dilation of rat brain arterioles by hypercapnia in vivo can occur even after blockade of guanylate cyclase by ODQ

Tetrahydrobiopterin, a cofactor for nitric oxide synthase, produces endothelium-dependent dilation of mouse pial arterioles

Antisense Evidence for Two Functionally Active Forms of Nitric Oxide Synthase in Brain Microvascular Endothelium

Re: Role of endothelial nitric oxide and smooth muscle potassium channels in cerebral arteriolar dilation in response to acidosis

Re: Disconnection of the Ascending Arousal System in Traumatic Coma. J Neuropathol Exp Neurol 2013;72:505-523/Author's Reply

Selective depression of endothelium-dependent dilations during cerebral ischemia

A review of vasomotor responses of arterioles on the surface of the mouse brain: the necessary prelude to studies using genetically manipulated mice

Is the EDRF in the Cerebral Circulation NO? Its Release by Shear and the Dangers in Interpreting the Effects of NOS Inhibitors

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