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Neuroinflammation and Central Sensitization in Chronic and Widespread Pain

Anesthesiology (Philadelphia), 2018-08, Vol.129 (2), p.343-366

2018

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Acute inflammatory response via neutrophil activation protects against the development of chronic pain

Science translational medicine, 2022-05, Vol.14 (644), p.eabj9954

2022

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β2- and β3-adrenergic receptors drive COMT-dependent pain by increasing production of nitric oxide and cytokines

Pain (Amsterdam), 2014-07, Vol.155 (7), p.1346-1355

2014

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Vagus nerve stimulation rescues persistent pain following orthopedic surgery in adult mice

Pain (Amsterdam), 2024-08, Vol.165 (8), p.e80-e92

2024

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Nuclear factor-kappa B regulates pain and COMT expression in a rodent model of inflammation

Brain, behavior, and immunity, 2015-11, Vol.50, p.196-202

2015

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Genetic architecture of human pain perception

Trends in genetics, 2007-12, Vol.23 (12), p.605-613

2007

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Transforming Growth Factor-β-Activated Kinase 1 (TAK1) Mediates Chronic Pain and Cytokine Production in Mouse Models of Inflammatory, Neuropathic, and Primary Pain

The journal of pain, 2023-09, Vol.24 (9), p.1633-1644

2023

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Alternative Splicing of G Protein–Coupled Receptors: Relevance to Pain Management

Mayo Clinic proceedings, 2015-08, Vol.90 (8), p.1135-1151

2015

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Sensory Neuron-TRPV4 Modulates Temporomandibular Disorder Pain Via CGRP in Mice

The journal of pain, 2023-05, Vol.24 (5), p.782-795

2023

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A mouse model of chronic primary pain that integrates clinically relevant genetic vulnerability, stress, and minor injury

Science translational medicine, 2024-04, Vol.16 (742)

2024

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Idiopathic pain disorders – Pathways of vulnerability

Pain (Amsterdam), 2006-08, Vol.123 (3), p.226-230

2006

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Sustained stimulation of β2- and β3-adrenergic receptors leads to persistent functional pain and neuroinflammation

Brain, behavior, and immunity, 2018-10, Vol.73, p.520-532

2018

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A Novel Alternatively Spliced Isoform of the Mu-Opioid Receptor: Functional Antagonism

Molecular pain, 2010-06, Vol.6 (1), p.33-33

2010

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miR-374 family is a key regulator of chronic primary pain onset

Pain reports, 2024-12, Vol.9 (6), p.e1199

2024

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Multi-ethnic GWAS and meta-analysis of sleep quality identify MPP6 as a novel gene that functions in sleep center neurons

Sleep (New York, N.Y.), 2021-03, Vol.44 (3), p.1

2021

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Low enzymatic activity haplotypes of the human catechol-O-methyltransferase gene: enrichment for marker SNPs

PloS one, 2009-04, Vol.4 (4), p.e5237-e5237

2009

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Catechol -O-methyltransferase inhibition increases pain sensitivity through activation of both β2-and β3-adrenergic receptors

Pain (Amsterdam), 2007-04, Vol.128 (3), p.199-208

2007

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Characterization of NF-Kb-Mediated Inhibition of Catechol-O-Methyltransferase

Molecular pain, 2009-03, Vol.5 (1), p.13-13

2009

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Anatomic Patterns of Mucosal and Muscular Pain Sensitivity in Provoked Vestibulodynia

The journal of pain, 2024-04, Vol.25 (4), p.1-1

2024

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Adipocyte Adrb3 Receptor Mediates Mechanical Hypersensitivity in Chronic Overlapping Pain Conditions

The journal of pain, 2022-05, Vol.23 (5), p.18-18

2022

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Neonatal Chronic Hind Paw Inflammation Alters Sensitization to Intradermal Capsaicin in Adult Rats: A Behavioral and Immunocytochemical Study

The journal of pain, 2005-12, Vol.6 (12), p.798-808

2005

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Activation of Beta-3 Adrenergic Receptors with the Selective Agonist Mirabegron Elicits Multi-Site Mechanical Hypersensitivity, Grimace, and Increased Interleukin-6 Levels in Mice

The journal of pain, 2024-04, Vol.25 (4), p.8-8

2024

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The Adrb3 Selective Agonist Mirabegron Elicits Multi-Site Mechanical Hypersensitivity And Grimace In Mice

The journal of pain, 2023-04, Vol.24 (4), p.28-29

2023

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IL-23/IL-17A/TRPV1 axis produces mechanical pain via macrophage-sensory neuron crosstalk in female mice

Neuron (Cambridge, Mass.), 2021-09, Vol.109 (17), p.2691-2706.e5

2021

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Basal and carrageenan-induced pain behavior in Sprague–Dawley, Lewis and Fischer rats

Physiology & behavior, 2005-06, Vol.85 (2), p.177-186

2005

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Neuroinflammation and Central Sensitization in Chronic and Widespread Pain

Acute inflammatory response via neutrophil activation protects against the development of chronic pain

β2- and β3-adrenergic receptors drive COMT-dependent pain by increasing production of nitric oxide and cytokines

Vagus nerve stimulation rescues persistent pain following orthopedic surgery in adult mice

Nuclear factor-kappa B regulates pain and COMT expression in a rodent model of inflammation

Genetic architecture of human pain perception

Transforming Growth Factor-β-Activated Kinase 1 (TAK1) Mediates Chronic Pain and Cytokine Production in Mouse Models of Inflammatory, Neuropathic, and Primary Pain

Alternative Splicing of G Protein–Coupled Receptors: Relevance to Pain Management

Sensory Neuron-TRPV4 Modulates Temporomandibular Disorder Pain Via CGRP in Mice

A mouse model of chronic primary pain that integrates clinically relevant genetic vulnerability, stress, and minor injury

Idiopathic pain disorders – Pathways of vulnerability

Sustained stimulation of β2- and β3-adrenergic receptors leads to persistent functional pain and neuroinflammation

A Novel Alternatively Spliced Isoform of the Mu-Opioid Receptor: Functional Antagonism

miR-374 family is a key regulator of chronic primary pain onset

Multi-ethnic GWAS and meta-analysis of sleep quality identify MPP6 as a novel gene that functions in sleep center neurons

Low enzymatic activity haplotypes of the human catechol-O-methyltransferase gene: enrichment for marker SNPs

Catechol -O-methyltransferase inhibition increases pain sensitivity through activation of both β2-and β3-adrenergic receptors

Characterization of NF-Kb-Mediated Inhibition of Catechol-O-Methyltransferase

Anatomic Patterns of Mucosal and Muscular Pain Sensitivity in Provoked Vestibulodynia

Adipocyte Adrb3 Receptor Mediates Mechanical Hypersensitivity in Chronic Overlapping Pain Conditions

Neonatal Chronic Hind Paw Inflammation Alters Sensitization to Intradermal Capsaicin in Adult Rats: A Behavioral and Immunocytochemical Study

Activation of Beta-3 Adrenergic Receptors with the Selective Agonist Mirabegron Elicits Multi-Site Mechanical Hypersensitivity, Grimace, and Increased Interleukin-6 Levels in Mice

The Adrb3 Selective Agonist Mirabegron Elicits Multi-Site Mechanical Hypersensitivity And Grimace In Mice

IL-23/IL-17A/TRPV1 axis produces mechanical pain via macrophage-sensory neuron crosstalk in female mice

Basal and carrageenan-induced pain behavior in Sprague–Dawley, Lewis and Fischer rats

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