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Warning Signs in the Development of Speech, Language, and Communication: When to Refer to a Speech-Language Pathologist

Journal of child and adolescent psychiatric nursing, 2011-08, Vol.24 (3), p.175-184

Wankoff, Lorain S.

2011

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Retinoblastoma protein represses transcription by recruiting a histone deacetylase

Nature (London), 1998-02, Vol.391 (6667), p.601-605

1998

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Gene therapy via trans-splicing for LMNA -related congenital muscular dystrophy (L-CMD)

Neuromuscular disorders : NMD, 2015-10, Vol.25, p.S280-S280

2015

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T.P.24 Correction of the GNE Myopathy M712T founder mutation by trans-splicing

Neuromuscular disorders : NMD, 2012-10, Vol.22 (9), p.859-860

2012

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P.20.8 AAV genome loss from dystrophic mouse muscles during AAV-U7snRNA-mediated exon skipping therapy

Neuromuscular disorders : NMD, 2013-10, Vol.23 (9), p.841-841

2013

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G.P.93

Neuromuscular disorders : NMD, 2014-10, Vol.24 (9), p.822-823

2014

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P4.51 Correction of FKRP function via RNA trans -splicing

Neuromuscular disorders : NMD, 2011-10, Vol.21 (9), p.719-720

2011

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P04 Correction of FKRP function via RNA trans-splicing

Neuromuscular disorders : NMD, 2011, Vol.21, p.S8-S8

2011

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O.14 Exon exchange approach to repair Duchenne dystrophin transcripts

Neuromuscular disorders : NMD, 2010-10, Vol.20 (9), p.639-639

2010

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T.P.1.05 Trans-splicing approaches to repair Duchenne dystrophin transcripts

Neuromuscular disorders : NMD, 2009-09, Vol.19 (8), p.578-578

2009

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Subnuclear localization and mitotic phosphorylation of HIRA, the human homologue of Saccharomyces cerevisiae transcriptional regulators Hir1p/Hir2p

Biochemical journal, 2001-09, Vol.358 (Pt 2), p.447-455

2001

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DMD TREATMENT

Neuromuscular disorders : NMD, 2019-10, Vol.29, p.S164-S165

2019

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DMD TREATMENT: P.334PPMO pre-treatment is beneficial for AAV-based gene therapy in Duchenne muscular dystrophy

Neuromuscular disorders : NMD, 2019-10, Vol.29, p.S164-S165

2019

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G.P.314 - Gene therapy via trans-splicing for LMNA-related congenital muscular dystrophy (L-CMD)

Neuromuscular disorders : NMD, 2015-10, Vol.25, p.S280-S280

2015

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MicroRNAs involved in nNOS regulation in dystrophic context

Neuromuscular disorders : NMD, 2017-10, Vol.27, p.S166-S167

2017

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P.237 - MicroRNAs involved in nNOS regulation in dystrophic context

Neuromuscular disorders : NMD, 2017-10, Vol.27, p.S166-S167

2017

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G.P.93 : Entry and intracellular trafficking of adeno-associated viral 8 (AAV8) vector in DMD muscular cells

Neuromuscular disorders : NMD, 2014-10, Vol.24 (9-10), p.822-823

2014

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Delivery is key: lessons learnt from developing splice‐switching antisense therapies

EMBO molecular medicine, 2017-05, Vol.9 (5), p.545-557

2017

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HIRA, a mammalian homologue of Saccharomyces cerevisiae transcriptional co-repressors, interacts with Pax3

Nature genetics, 1998-09, Vol.20 (1), p.74-77

1998

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Core Histones and HIRIP3, a Novel Histone-Binding Protein, Directly Interact with WD Repeat Protein HIRA

Molecular and Cellular Biology, 1998-09, Vol.18 (9), p.5546-5556

1998

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The Impact of Personality on College Student Connectedness, Course Satisfaction, and Mental Well-Being

Psi Beta Research Journal - Brief Reports, 2023-10, Vol.3 (1), p.1-8

2023

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Innovative Methods in Language Intervention: Treatment Outcome Measures--Can the Data Support the Claims?

PRO-ED, Inc, 2005

Szabo Wankoff, Lorain

2005

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Three-Year Efficacy of Complex Insulin Regimens in Type 2 Diabetes

The New England journal of medicine, 2009-10, Vol.361 (18), p.1736-1747

2009

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Structural Organization of the WD repeat protein-encoding gene HIRA in the DiGeorge syndrome critical region of human chromosome 22

Genome research, 1996-01, Vol.6 (1), p.43-50

1996

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Plasticité chromatinienne, contrôle de l'expression génique et pathologie humaine

M.S. Médecine sciences, 2000, Vol.16 (1), p.69

2000

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Warning Signs in the Development of Speech, Language, and Communication: When to Refer to a Speech-Language Pathologist

Retinoblastoma protein represses transcription by recruiting a histone deacetylase

Gene therapy via trans-splicing for LMNA -related congenital muscular dystrophy (L-CMD)

T.P.24 Correction of the GNE Myopathy M712T founder mutation by trans-splicing

P.20.8 AAV genome loss from dystrophic mouse muscles during AAV-U7snRNA-mediated exon skipping therapy

G.P.93

P4.51 Correction of FKRP function via RNA trans -splicing

P04 Correction of FKRP function via RNA trans-splicing

O.14 Exon exchange approach to repair Duchenne dystrophin transcripts

T.P.1.05 Trans-splicing approaches to repair Duchenne dystrophin transcripts

Subnuclear localization and mitotic phosphorylation of HIRA, the human homologue of Saccharomyces cerevisiae transcriptional regulators Hir1p/Hir2p

DMD TREATMENT

DMD TREATMENT: P.334PPMO pre-treatment is beneficial for AAV-based gene therapy in Duchenne muscular dystrophy

G.P.314 - Gene therapy via trans-splicing for LMNA-related congenital muscular dystrophy (L-CMD)

MicroRNAs involved in nNOS regulation in dystrophic context

P.237 - MicroRNAs involved in nNOS regulation in dystrophic context

G.P.93 : Entry and intracellular trafficking of adeno-associated viral 8 (AAV8) vector in DMD muscular cells

Delivery is key: lessons learnt from developing splice‐switching antisense therapies

HIRA, a mammalian homologue of Saccharomyces cerevisiae transcriptional co-repressors, interacts with Pax3

Core Histones and HIRIP3, a Novel Histone-Binding Protein, Directly Interact with WD Repeat Protein HIRA

The Impact of Personality on College Student Connectedness, Course Satisfaction, and Mental Well-Being

Innovative Methods in Language Intervention: Treatment Outcome Measures--Can the Data Support the Claims?

Three-Year Efficacy of Complex Insulin Regimens in Type 2 Diabetes

Structural Organization of the WD repeat protein-encoding gene HIRA in the DiGeorge syndrome critical region of human chromosome 22

Plasticité chromatinienne, contrôle de l'expression génique et pathologie humaine

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