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Systemic Adenosine Triphosphate Impairs Neutrophil Chemotaxis and Host Defense in Sepsis

Critical care medicine, 2017-01, Vol.45 (1), p.e97-e104

2017

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Author Correction: NADH oxidase-dependent CD39 expression by CD8+ T cells modulates interferon gamma responses via generation of adenosine

Nature communications, 2020-06, Vol.11 (1), p.3036-3036, Article 3036

2020

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ATP release and autocrine signaling through P2X4 receptors regulate γδ T cell activation

Journal of leukocyte biology, 2012-10, Vol.92 (4), p.787-794

2012

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Adenosine Triphosphate Release is Required for Toll-Like Receptor-Induced Monocyte/Macrophage Activation, Inflammasome Signaling, Interleukin-1β Production, and the Host Immune Response to Infection

Critical care medicine, 2018-12, Vol.46 (12), p.e1183-e1189

2018

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Purinergic P2Y2 receptors modulate endothelial sprouting

Cellular and molecular life sciences : CMLS, 2020-03, Vol.77 (5), p.885-901

2020

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mTOR and differential activation of mitochondria orchestrate neutrophil chemotaxis

The Journal of experimental medicine, 2015-10, Vol.212 (11), p.21211-21211OIA93

2015

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Hypertonic saline increases γδT cell-mediated killing of activated neutrophils

Critical care medicine, 2008-12, Vol.36 (12), p.3220-3225

2008

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Surface expression of HSP72 by LPS‐stimulated neutrophils facilitates γδT cell‐mediated killing

European journal of immunology, 2006-03, Vol.36 (3), p.712-721

2006

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Autocrine regulation of T-cell activation by ATP release and P2X₇ receptors

The FASEB journal, 2009-06, Vol.23 (6), p.1685-1693

2009

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Autocrine regulation of T‐cell activation by ATP release and P2X 7 receptors

The FASEB journal, 2009-06, Vol.23 (6), p.1685-1693

2009

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ATP release from influenza-infected lungs enhances neutrophil activation and promotes disease progression

The Journal of infectious diseases, 2023-10

2023

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P2Y11 receptors support T cell activation by directing mitochondrial trafficking to the immune synapse

Journal of leukocyte biology, 2020-06, Vol.109 (3), p.497-508

2020

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Roles of Heat Shock Proteins and γδT Cells in Inflammation

American journal of respiratory cell and molecular biology, 2008-11, Vol.39 (5), p.509-513

2008

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E. coli use LPS as decoy to impair neutrophil chemotaxis and defeat antimicrobial host defense

Journal of leukocyte biology, 2019-08, Vol.106 (6), p.1211-1219

2019

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ATP release is required for Toll-like receptor-induced monocyte/macrophage activation, NLRP3 inflammasome signaling, IL-1β production, and the host immune response to infection

Critical care medicine, 2018-12, Vol.46 (12), p.e1183-e1189

2018

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Systemic ATP impairs neutrophil chemotaxis and host defense in sepsis

Critical care medicine, 2017-01, Vol.45 (1), p.e97-e104

2017

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Purinergic signaling and immune responses in sepsis

Clinical therapeutics, 2016-05, Vol.38 (5), p.1054-1065

2016

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Whole-Blood Assay to Measure Oxidative Burst and Degranulation of Neutrophils for Monitoring Trauma Patients

European journal of trauma, 2005-08, Vol.31 (4), p.379-388

2005

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Roles of Heat Shock Proteins and {gamma}{delta}T Cells in Inflammation

American journal of respiratory cell and molecular biology, 2008-11, Vol.39 (5), p.509

2008

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Purinergic P2Y 2 receptors modulate endothelial sprouting

Cellular and molecular life sciences : CMLS, 2020-03, Vol.77 (5), p.885

2020

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CD39 Expression Identifies Terminally Exhausted CD8 + T Cells: e1005177

PLoS pathogens, 2015-10, Vol.11 (10)

2015

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CD39 expression identifies terminally exhausted [CD8.sup.+] T cells

PLoS pathogens, 2015-10, Vol.11 (10)

2015

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Pre-hospital hypertonic saline resuscitation attenuates the activation and promotes apoptosis of neutrophils in patients with severe traumatic brain injury

Shock (Augusta, Ga.), 2013-11, Vol.40 (5), p.366-374

2013

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E‐NTPDase1 and alkaline phosphatase control chemotaxis of human neutrophils by generating adenosine from released ATP

The FASEB journal, 2008-03, Vol.22 (S1)

2008

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Adrenergic receptor activation involves ATP release and feedback through purinergic receptors

American Journal of Physiology: Cell Physiology, 2010, Vol.68 (5)

2010

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Systemic Adenosine Triphosphate Impairs Neutrophil Chemotaxis and Host Defense in Sepsis

Author Correction: NADH oxidase-dependent CD39 expression by CD8+ T cells modulates interferon gamma responses via generation of adenosine

ATP release and autocrine signaling through P2X4 receptors regulate γδ T cell activation

Adenosine Triphosphate Release is Required for Toll-Like Receptor-Induced Monocyte/Macrophage Activation, Inflammasome Signaling, Interleukin-1β Production, and the Host Immune Response to Infection

Purinergic P2Y2 receptors modulate endothelial sprouting

mTOR and differential activation of mitochondria orchestrate neutrophil chemotaxis

Hypertonic saline increases γδT cell-mediated killing of activated neutrophils

Surface expression of HSP72 by LPS‐stimulated neutrophils facilitates γδT cell‐mediated killing

Autocrine regulation of T-cell activation by ATP release and P2X₇ receptors

Autocrine regulation of T‐cell activation by ATP release and P2X 7 receptors

ATP release from influenza-infected lungs enhances neutrophil activation and promotes disease progression

P2Y11 receptors support T cell activation by directing mitochondrial trafficking to the immune synapse

Roles of Heat Shock Proteins and γδT Cells in Inflammation

E. coli use LPS as decoy to impair neutrophil chemotaxis and defeat antimicrobial host defense

ATP release is required for Toll-like receptor-induced monocyte/macrophage activation, NLRP3 inflammasome signaling, IL-1β production, and the host immune response to infection

Systemic ATP impairs neutrophil chemotaxis and host defense in sepsis

Purinergic signaling and immune responses in sepsis

Whole-Blood Assay to Measure Oxidative Burst and Degranulation of Neutrophils for Monitoring Trauma Patients

Roles of Heat Shock Proteins and {gamma}{delta}T Cells in Inflammation

Purinergic P2Y 2 receptors modulate endothelial sprouting

CD39 Expression Identifies Terminally Exhausted CD8 + T Cells: e1005177

CD39 expression identifies terminally exhausted [CD8.sup.+] T cells

Pre-hospital hypertonic saline resuscitation attenuates the activation and promotes apoptosis of neutrophils in patients with severe traumatic brain injury

E‐NTPDase1 and alkaline phosphatase control chemotaxis of human neutrophils by generating adenosine from released ATP

Adrenergic receptor activation involves ATP release and feedback through purinergic receptors

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