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Comparison of the IKr blockers moxifloxacin, dofetilide and E‐4031 in five screening models of pro‐arrhythmia reveals lack of specificity of isolated cardiomyocytes

British journal of pharmacology, 2012-01, Vol.165 (2), p.467-478

2012

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Determining the role of uremic toxins on cardiac electrophysiology and pro-arrhythmia during chronic kidney disease

Cardiovascular research, 2022-06, Vol.118 (Supplement_1)

2022

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Computational modeling identifies the cellular electromechanical effects of disrupted intracellular calcium handling in arrhythmogenic cardiomyopathy patients

Cardiovascular research, 2022-06, Vol.118 (Supplement_1)

2022

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Buccal mucosa cells as a potential diagnostic tool to study onset and progression of arrhythmogenic cardiomyopathy

Cardiovascular research, 2022-06, Vol.118 (Supplement_1)

2022

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The anti‐protozoal drug pentamidine blocks KIR2.x‐mediated inward rectifier current by entering the cytoplasmic pore region of the channel

British journal of pharmacology, 2010-04, Vol.159 (7), p.1532-1541

2010

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Determining the role of uremic toxins on cardiac electrophysiology and pro-arrhythmia during chronic kidney disease

Europace (London, England), 2022-05, Vol.24 (Supplement_1)

2022

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A polymorphism in Histidine-Rich Calcium Binding Protein as second hit in Phospholamban Cardiomyopathy

Cardiovascular research, 2022-06, Vol.118 (Supplement_1)

2022

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Fibrosis biomarkers as a predictive tool for clinical outcome in PLNR14Del patients

Europace (London, England), 2021-05, Vol.23 (Supplement_3)

2021

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Computational modeling identifies the cellular electromechanical effects of disrupted intracellular calcium handling in arrhythmogenic cardiomyopathy patients

Europace (London, England), 2022-05, Vol.24 (Supplement_1)

2022

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P132Decreasing patchy fibrosis using combined CaMKII inhibition and anti-fibrotic eplerenone treatment tempers arrhythmogenesis in chronic pressure overloaded mice

Cardiovascular research, 2018-04, Vol.114 (suppl_1), p.S34-S34

2018

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Buccal mucosa cells as a diagnostic tool in patients with arrhythmogenic cardiomyopathy

Europace (London, England), 2021-05, Vol.23 (Supplement_3)

2021

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A polymorphism in histidine-rich calcium binding protein as second hit in phospholamban cardiomyopathy

Europace (London, England), 2022-05, Vol.24 (Supplement_1)

2022

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Mouse model of SCN5A-linked hereditary lenègre's disease : Age-related conduction slowing and myocardial fibrosis

Circulation (New York, N.Y.), 2005-04, Vol.111 (14), p.1738-1746

2005

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P316Optogenetic sensors in zebrafish hearts as novel in vivo electrophysiological readout tools to study cardiac arrhythmogenesis

Cardiovascular research, 2018-04, Vol.114 (suppl_1), p.S81-S81

2018

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Impaired impulse propagation in Scn5a-knockout mice : Combined contribution of excitability, connexin expression, and tissue architecture in relation to aging

Circulation (New York, N.Y.), 2005-09, Vol.112 (13), p.1927-1935

2005

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P504Arrhythmogenesis is tempered by combined CaMKII inhibition with anti-fibrotic eplerenone treatment in a model of chronic pressure overload

Europace (London, England), 2017-06, Vol.19 (suppl_3), p.iii114-iii114

2017

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Comparison of the I Kr blockers moxifloxacin, dofetilide and E‐4031 in five screening models of pro‐arrhythmia reveals lack of specificity of isolated cardiomyocytes

British journal of pharmacology, 2012-01, Vol.165 (2), p.467-478

2012

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Cardiac gap junction channels: modulation of expression and channel properties

Cardiovascular research, 2001-08, Vol.51 (2), p.217-229

2001

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Larger cell size in rabbits with heart failure increases myocardial conduction velocity and QRS duration

Circulation (New York, N.Y.), 2006-02, Vol.113 (6), p.806-813

2006

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P791A common co-morbidity modulates disease expression and treatment efficacy in inherited cardiac sodium channelopathy

Europace (London, England), 2018-03, Vol.20 (suppl_1), p.i140-i140

2018

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Impaired conduction in the bundle branches of mouse hearts lacking the gap junction protein connexin40

Circulation (New York, N.Y.), 2001-03, Vol.103 (11), p.1591-1598

2001

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Alternative promoter usage and splicing of the human SCN5A gene contribute to transcript heterogeneity

DNA and cell biology, 2010-10, Vol.29 (10), p.577-587

2010

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Hypoxia, electrical uncoupling, and conduction slowing: Role of conduction reserve

Cardiovascular research, 2005-04, Vol.66 (1), p.9-11

2005

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The anti‐protozoal drug pentamidine blocks K IR 2.x‐mediated inward rectifier current by entering the cytoplasmic pore region of the channel

British journal of pharmacology, 2010-04, Vol.159 (7), p.1532-1541

2010

Volltextzugriff (PDF)

Remodeling of Gap Junctions in Mouse Hearts Hypertrophied by Forced Retinoic Acid Signaling

Journal of molecular and cellular cardiology, 2002-10, Vol.34 (10), p.1411-1423

2002

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Comparison of the IKr blockers moxifloxacin, dofetilide and E‐4031 in five screening models of pro‐arrhythmia reveals lack of specificity of isolated cardiomyocytes

Determining the role of uremic toxins on cardiac electrophysiology and pro-arrhythmia during chronic kidney disease

Computational modeling identifies the cellular electromechanical effects of disrupted intracellular calcium handling in arrhythmogenic cardiomyopathy patients

Buccal mucosa cells as a potential diagnostic tool to study onset and progression of arrhythmogenic cardiomyopathy

The anti‐protozoal drug pentamidine blocks KIR2.x‐mediated inward rectifier current by entering the cytoplasmic pore region of the channel

Determining the role of uremic toxins on cardiac electrophysiology and pro-arrhythmia during chronic kidney disease

A polymorphism in Histidine-Rich Calcium Binding Protein as second hit in Phospholamban Cardiomyopathy

Fibrosis biomarkers as a predictive tool for clinical outcome in PLNR14Del patients

Computational modeling identifies the cellular electromechanical effects of disrupted intracellular calcium handling in arrhythmogenic cardiomyopathy patients

P132Decreasing patchy fibrosis using combined CaMKII inhibition and anti-fibrotic eplerenone treatment tempers arrhythmogenesis in chronic pressure overloaded mice

Buccal mucosa cells as a diagnostic tool in patients with arrhythmogenic cardiomyopathy

A polymorphism in histidine-rich calcium binding protein as second hit in phospholamban cardiomyopathy

Mouse model of SCN5A-linked hereditary lenègre's disease : Age-related conduction slowing and myocardial fibrosis

P316Optogenetic sensors in zebrafish hearts as novel in vivo electrophysiological readout tools to study cardiac arrhythmogenesis

Impaired impulse propagation in Scn5a-knockout mice : Combined contribution of excitability, connexin expression, and tissue architecture in relation to aging

P504Arrhythmogenesis is tempered by combined CaMKII inhibition with anti-fibrotic eplerenone treatment in a model of chronic pressure overload

Comparison of the I Kr blockers moxifloxacin, dofetilide and E‐4031 in five screening models of pro‐arrhythmia reveals lack of specificity of isolated cardiomyocytes

Cardiac gap junction channels: modulation of expression and channel properties

Larger cell size in rabbits with heart failure increases myocardial conduction velocity and QRS duration

P791A common co-morbidity modulates disease expression and treatment efficacy in inherited cardiac sodium channelopathy

Impaired conduction in the bundle branches of mouse hearts lacking the gap junction protein connexin40

Alternative promoter usage and splicing of the human SCN5A gene contribute to transcript heterogeneity

Hypoxia, electrical uncoupling, and conduction slowing: Role of conduction reserve

The anti‐protozoal drug pentamidine blocks K IR 2.x‐mediated inward rectifier current by entering the cytoplasmic pore region of the channel

Remodeling of Gap Junctions in Mouse Hearts Hypertrophied by Forced Retinoic Acid Signaling

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