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Increased Matrix Metalloproteinase-9 Predicts Poor Wound Healing in Diabetic Foot Ulcers

Diabetes care, 2009, Vol.32 (1), p.117-119

2009

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Disruption of E-Cadherin by Matrix Metalloproteinase Directly Mediates Epithelial-Mesenchymal Transition Downstream of Transforming Growth Factor-β1 in Renal Tubular Epithelial Cells

The American journal of pathology, 2009-08, Vol.175 (2), p.580-591

2009

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Comparison of digital and traditional skin wound closure assessment methods in mice

Laboratory Animal Research, 2023, 39(4), , pp.308-316

2023

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Insulin treatment prevents wounding associated changes in tissue and circulating neutrophil MMP-9 and NGAL in diabetic rats

PloS one, 2017-02, Vol.12 (2), p.e0170951-e0170951

2017

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Effect of low-volume exercise on hepatic steatosis in adults with obesity plus normal glucose, prediabetes or type 2 diabetes: a randomised controlled trial

BMJ Open Sport & Exercise Medicine, 2024-10, Vol.10 (4), p.e001878

2024

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Mesangial cell-derived factors alter monocyte activation and function through inflammatory pathways: possible pathogenic role in diabetic nephropathy

American journal of physiology. Renal physiology, 2009-11, Vol.297 (5), p.F1229-F1237

2009

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Alterations in Monocyte CD16 in Association with Diabetes Complications

Mediators of Inflammation, 2012-01, Vol.2012 (2012), p.317-326-133

2012

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Alterations of CD163 expression in the complications of diabetes: A systematic review

Journal of diabetes and its complications, 2022-04, Vol.36 (4), p.108150-108150, Article 108150

2022

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Increased Matrix Metalloproteinase-9 Predicts Poor Wound Healing in Diabetic Foot Ulcers

Diabetes care, 2009-11, Vol.32 (11), p.e137-e137

2009

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Blockade of High-Fat Diet Proteomic Phenotypes Using Exercise as Prevention or Treatment

Molecular & cellular proteomics, 2021-01, Vol.20, p.100027, Article 100027

2021

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Circulating CD31+ Angiogenic T cells are reduced in prediabetes and increase with exercise training

Journal of diabetes and its complications, 2024-11, Vol.38 (11), p.108868, Article 108868

2024

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Monocyte phenotype as a predictive marker for wound healing in diabetes-related foot ulcers

Journal of diabetes and its complications, 2021-05, Vol.35 (5), p.107889-107889, Article 107889

2021

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Blockade of High-Fat Diet Proteomic Phenotypes Using Exercise as Prevention or Treatment

Molecular & cellular proteomics, 2021, Vol.20, p.100027

2021

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Do diabetes-related foot ulcer wound fluid measures have clinical utility as biomarkers for healing? A systematic review

Journal of wound care, 2023-04, Vol.32 (Sup4a), p.xlvii-lxii

2023

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Circulating soluble CD163 as a potential biomarker of diabetes complications

Journal of diabetes and its complications, 2023-08, Vol.37 (8), p.108525-108525, Article 108525

2023

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284-OR: Hepatocyte-Specific CCN2 Gene Deletion Prevents NASH Fibrosis in a Mouse Model of High-Fat Feeding and Diabetes

Diabetes (New York, N.Y.), 2021-06, Vol.70 (Supplement_1)

2021

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233-OR: Circulating Inflammatory Biomarkers Are Not Improved with Combined Low-Volume High-Intensity Interval Training and Progressive Resistance Training in Prediabetes

Diabetes (New York, N.Y.), 2021-06, Vol.70 (Supplement_1)

2021

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Change in Circulating Monocyte Profile with Foot Ulcer Healing in Diabetic Patients

Diabetes (New York, N.Y.), 2018-07, Vol.67 (Supplement_1)

2018

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Monocyte CD163 is altered in association with diabetic complications: possible protective role

Journal of leukocyte biology, 2016-12, Vol.100 (6), p.1375-1383

2016

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Method for Analysis of Matrix Degradation by CCN2 Through the MMP/TIMP System

Methods in molecular biology (Clifton, N.J.), 2017, Vol.1489, p.523

2017

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Reduction of ARNT in myeloid cells causes immune suppression and delayed wound healing

American Journal of Physiology: Cell Physiology, 2014-08, Vol.307 (4), p.C349-C357

2014

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Increased matrix metalloproteinase-9 predicts poor wound healing in diabetic foot ulcers: Response to Muller et al

Diabetes care, 2009-11, Vol.32 (11), p.e137-e137

2009

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Mesangial cell-derived factors alter monocyte activation and function through inflammatory pathways: possible pathogenic role in diabetic nephropathy

American journal of physiology. Renal physiology, 2009, Vol.66 (5)

2009

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Activation of Macrophage Promatrix Metalloproteinase-9 by Lipopolysaccharide-Associated Proteinases

The Journal of immunology (1950), 2002-03, Vol.168 (5), p.2449-2455

2002

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RETHINKING THE MANAGEMENT OF HEALTH IN PEOPLE WITH DIABETES-RELATED FOOT ULCERS FROM AN AEP PERSPECTIVE: AN UPDATE FROM THE DFUEX STUDY

Journal of clinical exercise physiology, 2024-05, Vol.13 (s2), p.352-352

2024

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Increased Matrix Metalloproteinase-9 Predicts Poor Wound Healing in Diabetic Foot Ulcers

Disruption of E-Cadherin by Matrix Metalloproteinase Directly Mediates Epithelial-Mesenchymal Transition Downstream of Transforming Growth Factor-β1 in Renal Tubular Epithelial Cells

Comparison of digital and traditional skin wound closure assessment methods in mice

Insulin treatment prevents wounding associated changes in tissue and circulating neutrophil MMP-9 and NGAL in diabetic rats

Effect of low-volume exercise on hepatic steatosis in adults with obesity plus normal glucose, prediabetes or type 2 diabetes: a randomised controlled trial

Mesangial cell-derived factors alter monocyte activation and function through inflammatory pathways: possible pathogenic role in diabetic nephropathy

Alterations in Monocyte CD16 in Association with Diabetes Complications

Alterations of CD163 expression in the complications of diabetes: A systematic review

Increased Matrix Metalloproteinase-9 Predicts Poor Wound Healing in Diabetic Foot Ulcers

Blockade of High-Fat Diet Proteomic Phenotypes Using Exercise as Prevention or Treatment

Circulating CD31+ Angiogenic T cells are reduced in prediabetes and increase with exercise training

Monocyte phenotype as a predictive marker for wound healing in diabetes-related foot ulcers

Blockade of High-Fat Diet Proteomic Phenotypes Using Exercise as Prevention or Treatment

Do diabetes-related foot ulcer wound fluid measures have clinical utility as biomarkers for healing? A systematic review

Circulating soluble CD163 as a potential biomarker of diabetes complications

284-OR: Hepatocyte-Specific CCN2 Gene Deletion Prevents NASH Fibrosis in a Mouse Model of High-Fat Feeding and Diabetes

233-OR: Circulating Inflammatory Biomarkers Are Not Improved with Combined Low-Volume High-Intensity Interval Training and Progressive Resistance Training in Prediabetes

Change in Circulating Monocyte Profile with Foot Ulcer Healing in Diabetic Patients

Monocyte CD163 is altered in association with diabetic complications: possible protective role

Method for Analysis of Matrix Degradation by CCN2 Through the MMP/TIMP System

Reduction of ARNT in myeloid cells causes immune suppression and delayed wound healing

Increased matrix metalloproteinase-9 predicts poor wound healing in diabetic foot ulcers: Response to Muller et al

Mesangial cell-derived factors alter monocyte activation and function through inflammatory pathways: possible pathogenic role in diabetic nephropathy

Activation of Macrophage Promatrix Metalloproteinase-9 by Lipopolysaccharide-Associated Proteinases

RETHINKING THE MANAGEMENT OF HEALTH IN PEOPLE WITH DIABETES-RELATED FOOT ULCERS FROM AN AEP PERSPECTIVE: AN UPDATE FROM THE DFUEX STUDY

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