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Promotion of cellular senescence by THG-1/TSC22D4 knockout through activation of JUNB

Biochemical and biophysical research communications, 2020-02, Vol.522 (4), p.897-902

2020

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PMEPA1/TMEPAI knockout impairs tumour growth and lung metastasis in MDA-MB-231 cells without changing monolayer culture cell growth

Journal of biochemistry (Tokyo), 2019-05, Vol.165 (5), p.411-414

2019

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Glycoprotein nmb Is Exposed on the Surface of Dormant Breast Cancer Cells and Induces Stem Cell-like Properties

Cancer research (Chicago, Ill.), 2018-11, Vol.78 (22), p.6424-6435

2018

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Glycoprotein NMB promotes tumor formation and malignant progression of laryngeal squamous cell carcinoma

Cancer science, 2022-09, Vol.113 (9), p.3244-3254

2022

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TMEPAI/PMEPA1 enhances tumorigenic activities in lung cancer cells

Cancer science, 2014-03, Vol.105 (3), p.334-341

2014

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Regulation of c‐MYC transcriptional activity by transforming growth factor‐beta 1‐stimulated clone 22

Cancer science, 2018-02, Vol.109 (2), p.395-402

2018

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Phosphorylation status determines the opposing functions of Smad2/Smad3 as STAT3 cofactors in TH17 differentiation

Nature communications, 2015-07, Vol.6 (1), p.7600-7600, Article 7600

2015

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Role of the kringle‐like domain in glycoprotein NMB for its tumorigenic potential

Cancer science, 2019-07, Vol.110 (7), p.2237-2246

2019

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The possible repositioning of an oral anti-arthritic drug, auranofin, for Nrf2-activating therapy: The demonstration of Nrf2-dependent anti-oxidative action using a zebrafish model

Free radical biology & medicine, 2018-02, Vol.115, p.405-411

2018

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PCSK9 Promotes Cardiovascular Diseases: Recent Evidence about Its Association with Platelet Activation-Induced Myocardial Infarction

Life (Basel, Switzerland), 2022-01, Vol.12 (2), p.190

2022

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Cell-surface GPNMB and induction of stemness

Oncotarget, 2018-12, Vol.9 (99), p.37289-37290

2018

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Regulation of the TMEPAI promoter by TCF7L2: the C-terminal tail of TCF7L2 is essential to activate the TMEPAI gene

Journal of biochemistry (Tokyo), 2016-01, Vol.159 (1), p.27-30

2016

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THG-1 suppresses SALL4 degradation to induce stemness genes and tumorsphere formation through antagonizing NRBP1 in squamous cell carcinoma cells

Biochemical and biophysical research communications, 2020-03, Vol.523 (2), p.307-314

2020

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Glycoprotein non–metastatic melanoma protein B functions with growth factor signaling to induce tumorigenesis through its serine phosphorylation

Cancer science, 2021-10, Vol.112 (10), p.4187-4197

2021

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Repression of Smad3 by Stat3 and c-Ski/SnoN induces gefitinib resistance in lung adenocarcinoma

Biochemical and biophysical research communications, 2017-03, Vol.484 (2), p.269-277

2017

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TGFβ selects for pro‐stemness over pro‐invasive phenotypes during cancer cell epithelial–mesenchymal transition

Molecular oncology, 2022-06, Vol.16 (12), p.2330-2354

2022

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PMEPA1/TMEPAI Is a Unique Tumorigenic Activator of AKT Promoting Proteasomal Degradation of PHLPP1 in Triple-Negative Breast Cancer Cells

Cancers, 2021-09, Vol.13 (19), p.4934

2021

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Smad2/Smad3 in endothelium is indispensable for vascular stability via S1PR1 and N-cadherin expressions

Blood, 2012-05, Vol.119 (22), p.5320-5328

2012

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Inhibition of endothelial cell activation by bHLH protein E2-2 and its impairment of angiogenesis

Blood, 2010-05, Vol.115 (20), p.4138-4147

2010

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Activin receptor‐like kinase5 inhibition suppresses mouse melanoma by ubiquitin degradation of Smad4, thereby derepressing eomesodermin in cytotoxic T lymphocytes

EMBO molecular medicine, 2013-11, Vol.5 (11), p.1720-1739

2013

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Recent Update on PCSK9 and Platelet Activation Experimental Research Methods: In Vitro and In Vivo Studies

Journal of cardiovascular development and disease, 2022-08, Vol.9 (8), p.258

2022

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c-myc Is a Downstream Target of the Smad Pathway

The Journal of biological chemistry, 2002-01, Vol.277 (1), p.854-861

2002

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Interference of E2‐2‐mediated effect in endothelial cells by FAM96B through its limited expression of E2‐2

Cancer science, 2011-10, Vol.102 (10), p.1808-1814

2011

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Alternatively Spliced Variant of Smad2 Lacking Exon 3

The Journal of biological chemistry, 1999-01, Vol.274 (2), p.703-709

1999

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Promotion of cellular senescence by THG-1/TSC22D4 knockout through activation of JUNB

PMEPA1/TMEPAI knockout impairs tumour growth and lung metastasis in MDA-MB-231 cells without changing monolayer culture cell growth

Glycoprotein nmb Is Exposed on the Surface of Dormant Breast Cancer Cells and Induces Stem Cell-like Properties

Glycoprotein NMB promotes tumor formation and malignant progression of laryngeal squamous cell carcinoma

TMEPAI/PMEPA1 enhances tumorigenic activities in lung cancer cells

Regulation of c‐MYC transcriptional activity by transforming growth factor‐beta 1‐stimulated clone 22

Phosphorylation status determines the opposing functions of Smad2/Smad3 as STAT3 cofactors in TH17 differentiation

Role of the kringle‐like domain in glycoprotein NMB for its tumorigenic potential

The possible repositioning of an oral anti-arthritic drug, auranofin, for Nrf2-activating therapy: The demonstration of Nrf2-dependent anti-oxidative action using a zebrafish model

PCSK9 Promotes Cardiovascular Diseases: Recent Evidence about Its Association with Platelet Activation-Induced Myocardial Infarction

Cell-surface GPNMB and induction of stemness

Regulation of the TMEPAI promoter by TCF7L2: the C-terminal tail of TCF7L2 is essential to activate the TMEPAI gene

THG-1 suppresses SALL4 degradation to induce stemness genes and tumorsphere formation through antagonizing NRBP1 in squamous cell carcinoma cells

Glycoprotein non–metastatic melanoma protein B functions with growth factor signaling to induce tumorigenesis through its serine phosphorylation

Repression of Smad3 by Stat3 and c-Ski/SnoN induces gefitinib resistance in lung adenocarcinoma

Disruption of entire Cables2 locus leads to embryonic lethality by diminished Rps21 gene expression and enhanced p53 pathway

TGFβ selects for pro‐stemness over pro‐invasive phenotypes during cancer cell epithelial–mesenchymal transition

PMEPA1/TMEPAI Is a Unique Tumorigenic Activator of AKT Promoting Proteasomal Degradation of PHLPP1 in Triple-Negative Breast Cancer Cells

Smad2/Smad3 in endothelium is indispensable for vascular stability via S1PR1 and N-cadherin expressions

Inhibition of endothelial cell activation by bHLH protein E2-2 and its impairment of angiogenesis

Activin receptor‐like kinase5 inhibition suppresses mouse melanoma by ubiquitin degradation of Smad4, thereby derepressing eomesodermin in cytotoxic T lymphocytes

Recent Update on PCSK9 and Platelet Activation Experimental Research Methods: In Vitro and In Vivo Studies

c-myc Is a Downstream Target of the Smad Pathway

Interference of E2‐2‐mediated effect in endothelial cells by FAM96B through its limited expression of E2‐2

Alternatively Spliced Variant of Smad2 Lacking Exon 3

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