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Modulation of the endoplasmic reticulum–mitochondria interface in Alzheimer’s disease and related models

Proceedings of the National Academy of Sciences - PNAS, 2013-05, Vol.110 (19), p.7916-7921

2013

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Amyloid-β accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism

Journal of neuroinflammation, 2023-02, Vol.20 (1), p.43-43, Article 43

2023

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MIEF1/2 orchestrate mitochondrial dynamics through direct engagement with both the fission and fusion machineries

BMC biology, 2021-10, Vol.19 (1), p.229-229, Article 229

2021

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Mechanism of Peptide Binding and Cleavage by the Human Mitochondrial Peptidase Neurolysin

Journal of molecular biology, 2018-02, Vol.430 (3), p.348-362

2018

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Strategic role for mitochondria in Alzheimer's disease and cancer

Antioxidants & redox signaling, 2012-06, Vol.16 (12), p.1476-1491

2012

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Center for Alzheimer Research at Karolinska Institutet, Sweden

Alzheimer's & dementia, 2023-12, Vol.19 (S23), p.n/a

2023

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Beyond the critical point: An overview of excitotoxicity, calcium overload and the downstream consequences

Neuroscience letters, 2018-01, Vol.663, p.79-85

2018

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Alterations in mitochondria-endoplasmic reticulum connectivity in human brain biopsies from idiopathic normal pressure hydrocephalus patients

Acta neuropathologica communications, 2018-10, Vol.6 (1), p.102-102, Article 102

2018

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Amyloid-β peptides are generated in mitochondria-associated endoplasmic reticulum membranes

Journal of Alzheimer's disease, 2015-01, Vol.43 (2), p.369-374

2015

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Biomarkers for apoptosis in Alzheimer's disease

International journal of geriatric psychiatry, 2005-02, Vol.20 (2), p.101-105

2005

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Presenilin-1 is located in rat mitochondria

Biochemical and biophysical research communications, 2002-07, Vol.295 (3), p.766-770

2002

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Isolation of Mitochondria-Associated Membranes (MAM) from Mouse Brain Tissue

Methods in molecular biology (Clifton, N.J.), 2017-01, Vol.1567, p.53

2017

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Mitochondrial hypermetabolism precedes impaired autophagy and synaptic disorganization in App knock-in Alzheimer mouse models

Molecular psychiatry, 2023-09, Vol.28 (9), p.3966-3981

2023

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Amyloid-[beta] accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism

Journal of neuroinflammation, 2023-02, Vol.20 (1)

2023

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The Sigma-1 Receptor Mediates Pridopidine Rescue of Mitochondrial Function in Huntington Disease Models

Neurotherapeutics, 2021-04, Vol.18 (2), p.1017-1038

2021

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Mitochondrial Alterations in Neurons Derived from the Murine AppNL-F Knock-In Model of Alzheimer's Disease

Journal of Alzheimer's disease, 2022-01, Vol.90 (2), p.565-583

2022

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Rethinking Alzheimer's disease therapy: are mitochondria the key?

Journal of Alzheimer's disease, 2010-01, Vol.20 Suppl 2 (s2), p.S579-S590

2010

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Degradation of the Amyloid β-Protein by the Novel Mitochondrial Peptidasome, PreP

The Journal of biological chemistry, 2006-09, Vol.281 (39), p.29096-29104

2006

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Dimebon (latrepirdine) enhances mitochondrial function and protects neuronal cells from death

Journal of Alzheimer's disease, 2010-01, Vol.21 (2), p.389-402

2010

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THE EFFECT OF AMYLOID β-PEPTIDE ON ER-MITOCHONDRIA CONTACT IN ALZHEIMER’S DISEASE

Alzheimer's & dementia, 2017-07, Vol.13 (7), p.P886-P886

2017

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Nicastrin, Presenilin, APH-1, and PEN-2 Form Active γ-Secretase Complexes in Mitochondria

The Journal of biological chemistry, 2004-12, Vol.279 (49), p.51654-51660

2004

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Apoptosis and Necrosis: Two Distinct Events Induced, Respectively, by Mild and Intense Insults with N-Methyl-D-Aspartate or Nitric Oxide/Superoxide in Cortical Cell Cultures

Proceedings of the National Academy of Sciences - PNAS, 1995-08, Vol.92 (16), p.7162-7166

1995

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Reduction of PINK 1 or DJ ‐1 impair mitochondrial motility in neurites and alter ER ‐mitochondria contacts

Journal of cellular and molecular medicine, 2018-11, Vol.22 (11), p.5439-5449

2018

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TOM70 Sustains Cell Bioenergetics by Promoting IP3R3-Mediated ER to Mitochondria Ca 2+ Transfer

Current biology, 2018-02, Vol.28 (3), p.369

2018

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Modulation of the endoplasmic reticulum–mitochondria interface in Alzheimer’s disease and related models

Amyloid-β accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism

MIEF1/2 orchestrate mitochondrial dynamics through direct engagement with both the fission and fusion machineries

Mechanism of Peptide Binding and Cleavage by the Human Mitochondrial Peptidase Neurolysin

Strategic role for mitochondria in Alzheimer's disease and cancer

Neuronal cell-based high-throughput screen for enhancers of mitochondrial function reveals luteolin as a modulator of mitochondria-endoplasmic reticulum coupling

Center for Alzheimer Research at Karolinska Institutet, Sweden

Beyond the critical point: An overview of excitotoxicity, calcium overload and the downstream consequences

Alterations in mitochondria-endoplasmic reticulum connectivity in human brain biopsies from idiopathic normal pressure hydrocephalus patients

Amyloid-β peptides are generated in mitochondria-associated endoplasmic reticulum membranes

Biomarkers for apoptosis in Alzheimer's disease

Presenilin-1 is located in rat mitochondria

Isolation of Mitochondria-Associated Membranes (MAM) from Mouse Brain Tissue

Mitochondrial hypermetabolism precedes impaired autophagy and synaptic disorganization in App knock-in Alzheimer mouse models

Amyloid-[beta] accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism

The Sigma-1 Receptor Mediates Pridopidine Rescue of Mitochondrial Function in Huntington Disease Models

Mitochondrial Alterations in Neurons Derived from the Murine AppNL-F Knock-In Model of Alzheimer's Disease

Rethinking Alzheimer's disease therapy: are mitochondria the key?

Degradation of the Amyloid β-Protein by the Novel Mitochondrial Peptidasome, PreP

Dimebon (latrepirdine) enhances mitochondrial function and protects neuronal cells from death

THE EFFECT OF AMYLOID β-PEPTIDE ON ER-MITOCHONDRIA CONTACT IN ALZHEIMER’S DISEASE

Nicastrin, Presenilin, APH-1, and PEN-2 Form Active γ-Secretase Complexes in Mitochondria

Apoptosis and Necrosis: Two Distinct Events Induced, Respectively, by Mild and Intense Insults with N-Methyl-D-Aspartate or Nitric Oxide/Superoxide in Cortical Cell Cultures

Reduction of PINK 1 or DJ ‐1 impair mitochondrial motility in neurites and alter ER ‐mitochondria contacts

TOM70 Sustains Cell Bioenergetics by Promoting IP3R3-Mediated ER to Mitochondria Ca 2+ Transfer

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