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Delineation of the extent of coronary atherosclerosis by high-frequency epicardial echocardiography

The New England journal of medicine, 1987-02, Vol.316 (6), p.304-309

1987

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Overlapping sequential pulses : a new waveform for transthoracic defibrillation

Circulation (New York, N.Y.), 1994-05, Vol.89 (5), p.2369-2379

1994

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Effect of lidocaine and bretylium on energy requirements for transthoracic defibrillation: Experimental studies

Journal of the American College of Cardiology, 1986-02, Vol.7 (2), p.397-405

1986

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High frequency epicardial echocardiography for coronary artery evaluation: In vitro and in vivo validation of arterial lumen and wall thickness measurements

Journal of the American College of Cardiology, 1986-09, Vol.8 (3), p.600-606

1986

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Two-dimensional echocardiography in experimental coronary stenosis. II. Relationship between systolic wall thinning and regional myocardial perfusion in severe coronary stenosis

Circulation (New York, N.Y.), 1982-09, Vol.66 (3), p.603-611

1982

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Mechanisms responsible for decline in transthoracic impedance after DC shocks

American journal of physiology. Heart and circulatory physiology, 1989-10, Vol.257 (4), p.H1180-H1183

1989

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Current-based transthoracic defibrillation

The American journal of cardiology, 1996-11, Vol.78 (10), p.1113-1118

1996

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Gene differences influencing visual system function and behavior

Behavior genetics, 1974-06, Vol.4 (2), p.165-170

1974

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Is transthoracic impedance arrythmia specific ? Experimental studies

The American heart journal, 1994, Vol.127 (4), p.842-847

1994

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Effect of age on diastolic left ventricular filling at rest and during inotripic stimulation and acute systemic hypertension : experimental studies in conscious beagles

The American heart journal, 1990, Vol.120 (1), p.73-81

1990

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Effect of calcium channel blockers on hemodynamic responses to defibrillation

The American heart journal, 1989-01, Vol.177 (3), p.569-576

1989

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Relations between 2-dimensional echocardiographic wall thickening abnormalities, myocardial infarct size and coronary risk area in normal and hypertrophied myocardium in dogs

The American journal of cardiology, 1983-12, Vol.52 (10), p.1318-1325

1983

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Mechanisms responsible for decline in transthoracic impedance after DC shocks

American journal of physiology. Heart and circulatory physiology, 1989, Vol.26 (4), p.H1180-H1183

1989

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Ultrasound Characterization of Acoustic Properties of Acute Intracardiac Thrombi: Studies in a New Experimental Model

Journal of the American Society of Echocardiography, 1988-07, Vol.1 (4), p.264-270

1988

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Transient myocardial ischemia: experimental echocardiographic demonstration and evaluation of myocardial contraction abnormalities

Canadian journal of cardiology, 1986-07, Vol.Suppl A, p.136A-141A

1986

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Relations between 2-dimensional echocardiographic wall thickening abnormalities, myocardial infarct size and coronary risk area in normal and hypertrophied myocardium in dogs

The American journal of cardiology, 1984, Vol.52 (10), p.1318-1325

1984

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Duvelisib Eliminates CLL B Cells, Impairs CLL-Supporting Cells, and Overcomes Ibrutinib Resistance in a Xenograft Model

Clinical cancer research, 2023-05, Vol.29 (10), p.1984-1995

2023

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CLECL1+ CLL Cells: A Key Subset That Drives CLL Survival and Proliferation and Shapes a Th2-Biased Tumor Microenvironment

Blood, 2022-11, Vol.140 (Supplement 1), p.1528-1529

2022

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Serum Proteomic Analyses Suggest That the HMGB1 and Other Inflammatory Pathways Are Operational in MBL and Are Less in Overt CLL

Blood, 2021-11, Vol.138 (Supplement 1), p.2625-2625

2021

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Identification of Three Unique Clusters of Serum Proteins with Distinctive Functionalities in IGHV-Mutated MBL

Blood, 2022-11, Vol.140 (Supplement 1), p.9859-9861

2022

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Chronic lymphocytic leukemia–like monoclonal B-cell lymphocytosis exhibits an increased inflammatory signature that is reduced in early-stage chronic lymphocytic leukemia

Experimental hematology, 2021-03, Vol.95, p.68-80

2021

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Dual Inhibition of PI3K-δ and PI3K-γ By Duvelisib Eliminates CLL B Cells, Impairs CLL-Supporting Cells, and Overcomes Ibrutinib Resistance in a Patient-Derived Xenograft Model

Blood, 2018-11, Vol.132 (Supplement 1), p.4420-4420

2018

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Abstract 960: Mechanisms underlying primary ibrutinib sensitivity in CLL

Cancer research (Chicago, Ill.), 2017-07, Vol.77 (13_Supplement), p.960-960

2017

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Deciphering the CXCL9-CXCL10-CXCL11/CXCR3 Axis in CLL-like Monoclonal B-Cell Lymphocytosis and Chronic Lymphocytic Leukemia: A New Target for Immune Activation?

Blood, 2019-11, Vol.134 (Supplement_1), p.3029-3029

2019

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Gene Expression and Cytokine Analyses Identify Markers of Progression from CLL-like Monoclonal B-Cell Lymphocytosis to Chronic Lymphocytic Leukemia

Blood, 2019-11, Vol.134 (Supplement_1), p.3027-3027

2019

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Delineation of the extent of coronary atherosclerosis by high-frequency epicardial echocardiography

Overlapping sequential pulses : a new waveform for transthoracic defibrillation

Effect of lidocaine and bretylium on energy requirements for transthoracic defibrillation: Experimental studies

High frequency epicardial echocardiography for coronary artery evaluation: In vitro and in vivo validation of arterial lumen and wall thickness measurements

Two-dimensional echocardiography in experimental coronary stenosis. II. Relationship between systolic wall thinning and regional myocardial perfusion in severe coronary stenosis

Mechanisms responsible for decline in transthoracic impedance after DC shocks

Current-based transthoracic defibrillation

Gene differences influencing visual system function and behavior

Is transthoracic impedance arrythmia specific ? Experimental studies

Effect of age on diastolic left ventricular filling at rest and during inotripic stimulation and acute systemic hypertension : experimental studies in conscious beagles

Effect of calcium channel blockers on hemodynamic responses to defibrillation

Relations between 2-dimensional echocardiographic wall thickening abnormalities, myocardial infarct size and coronary risk area in normal and hypertrophied myocardium in dogs

Mechanisms responsible for decline in transthoracic impedance after DC shocks

Ultrasound Characterization of Acoustic Properties of Acute Intracardiac Thrombi: Studies in a New Experimental Model

Transient myocardial ischemia: experimental echocardiographic demonstration and evaluation of myocardial contraction abnormalities

Relations between 2-dimensional echocardiographic wall thickening abnormalities, myocardial infarct size and coronary risk area in normal and hypertrophied myocardium in dogs

Duvelisib Eliminates CLL B Cells, Impairs CLL-Supporting Cells, and Overcomes Ibrutinib Resistance in a Xenograft Model

CLECL1+ CLL Cells: A Key Subset That Drives CLL Survival and Proliferation and Shapes a Th2-Biased Tumor Microenvironment

Serum Proteomic Analyses Suggest That the HMGB1 and Other Inflammatory Pathways Are Operational in MBL and Are Less in Overt CLL

Identification of Three Unique Clusters of Serum Proteins with Distinctive Functionalities in IGHV-Mutated MBL

Chronic lymphocytic leukemia–like monoclonal B-cell lymphocytosis exhibits an increased inflammatory signature that is reduced in early-stage chronic lymphocytic leukemia

Dual Inhibition of PI3K-δ and PI3K-γ By Duvelisib Eliminates CLL B Cells, Impairs CLL-Supporting Cells, and Overcomes Ibrutinib Resistance in a Patient-Derived Xenograft Model

Abstract 960: Mechanisms underlying primary ibrutinib sensitivity in CLL

Deciphering the CXCL9-CXCL10-CXCL11/CXCR3 Axis in CLL-like Monoclonal B-Cell Lymphocytosis and Chronic Lymphocytic Leukemia: A New Target for Immune Activation?

Gene Expression and Cytokine Analyses Identify Markers of Progression from CLL-like Monoclonal B-Cell Lymphocytosis to Chronic Lymphocytic Leukemia

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