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Thrombin‐activatable fibrinolysis inhibitor (TAFI, plasma procarboxypeptidase B, procarboxypeptidase R, procarboxypeptidase U)

Journal of thrombosis and haemostasis, 2003-07, Vol.1 (7), p.1566-1574

2003

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A randomized cross-over study on the effects of levonorgestrel- and desogestrel-containing oral contraceptives on the anticoagulant pathways

Thrombosis and haemostasis, 2000-07, Vol.84 (1), p.15-21

2000

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Effects on coagulation of levonorgestrel- and desogestrel-containing low dose oral contraceptives: a cross-over study

Thrombosis and haemostasis, 2000-07, Vol.84 (1), p.4-8

2000

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Regulation of fibrinolysis in plasma by TAFI and protein C is dependent on the concentration of thrombomodulin

Thrombosis and haemostasis, 2001-01, Vol.85 (1), p.5-11

2001

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Plasma TAFI levels influence the clot lysis time in healthy individuals in the presence of an intact intrinsic pathway of coagulation

Thrombosis and haemostasis, 1998-11, Vol.80 (5), p.829-835

1998

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Decreased active von Willebrand factor level owing to shear stress in aortic stenosis patients

Journal of thrombosis and haemostasis, 2011-05, Vol.9 (5), p.953-958

2011

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The defective down regulation of fibrinolysis in haemophilia A can be restored by increasing the TAFI plasma concentration

Thrombosis and haemostasis, 2001-10, Vol.86 (4), p.1035-1039

2001

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Increased fibrinolytic activity during use of oral contraceptives is counteracted by an enhanced factor XI-independent down regulation of fibrinolysis: a randomized cross-over study of two low-dose oral contraceptives

Thrombosis and haemostasis, 2000-07, Vol.84 (1), p.9-14

2000

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Impaired healing of cutaneous wounds and colonic anastomoses in mice lacking thrombin‐activatable fibrinolysis inhibitor

Journal of thrombosis and haemostasis, 2003-10, Vol.1 (10), p.2087-2096

2003

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Feedback Activation of Factor XI by Thrombin in Plasma Results in Additional Formation of Thrombin That Protects Fibrin Clots From Fibrinolysis

Blood, 1995-10, Vol.86 (8), p.3035-3042

1995

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Effects of second and third generation oral contraceptives and their respective progestagens on the coagulation system in the absence or presence of the factor V Leiden mutation

Thrombosis and haemostasis, 2002-02, Vol.87 (2), p.199-205

2002

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Factor VIII and von Willebrand factor

Thrombosis and haemostasis, 1998-03, Vol.79 (3), p.456-465

1998

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Decreased plasma sensitivity to activated protein C by oral contraceptives is associated with decreases in plasma glucosylceramide

Journal of thrombosis and haemostasis, 2005-05, Vol.3 (5), p.935-938

2005

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Antiphospholipid Antibodies Directed Against a Combination of Phospholipids With Prothrombin, Protein C, or Protein S: An Explanation for Their Pathogenic Mechanism?

Blood, 1993-05, Vol.81 (10), p.2618-2625

1993

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Protein C inhibitor regulates the thrombin-thrombomodulin complex in the up- and down regulation of TAFI activation

Thrombosis and haemostasis, 2001-10, Vol.86 (4), p.1057-1064

2001

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The role of protein S in the activation of thrombin activatable fibrinolysis inhibitor (TAFI) and regulation of fibrinolysis

Thrombosis and haemostasis, 2001-10, Vol.86 (4), p.1040-1046

2001

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Unraveling the mystery of von Willebrand factor

Journal of thrombosis and haemostasis, 2006-03, Vol.4 (3), p.489-495

2006

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Thrombin-Activatable Fibrinolysis Inhibitor (TAFI, Plasma Procarboxypeptidase B, Procarboxypeptidase R, Procarboxypeptidase U)

Thrombosis research, 2001-03, Vol.101 (5), p.329-354

2001

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Reduced activity of TAFI (thrombin‐activatable fibrinolysis inhibitor) in acute promyelocytic leukaemia

British journal of haematology, 2000-03, Vol.108 (3), p.518-523

2000

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Factor XI dependent and independent activation of thrombin activatable fibrinolysis inhibitor (TAFI) in plasma associated with clot formation

Thrombosis and haemostasis, 1999-12, Vol.82 (6), p.1703-1708

1999

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Factor XI and protection of the fibrin clot against lysis--a role for the intrinsic pathway of coagulation in fibrinolysis

Thrombosis and haemostasis, 1998-07, Vol.80 (1), p.24-27

1998

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Tumor growth and metastasis are not affected in thrombin‐activatable fibrinolysis inhibitor‐deficient mice

Journal of thrombosis and haemostasis, 2004-05, Vol.2 (5), p.769-779

2004

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Role of isoleucine residues 182 and 183 in thrombin‐activatable fibrinolysis inhibitor

Journal of thrombosis and haemostasis, 2005-06, Vol.3 (6), p.1293-1300

2005

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The Affinity and Stoichiometry of Binding of Human Factor VIII to von Willebrand Factor

Blood, 1995-06, Vol.85 (11), p.3150-3157

1995

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C4b-binding protein inhibits the factor V-dependent but not the factor V-independent cofactor activity of protein S in the activated protein C-mediated inactivation of factor VIIIa

Thrombosis and haemostasis, 2001-05, Vol.85 (5), p.761-765

2001

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Thrombin‐activatable fibrinolysis inhibitor (TAFI, plasma procarboxypeptidase B, procarboxypeptidase R, procarboxypeptidase U)

A randomized cross-over study on the effects of levonorgestrel- and desogestrel-containing oral contraceptives on the anticoagulant pathways

Effects on coagulation of levonorgestrel- and desogestrel-containing low dose oral contraceptives: a cross-over study

Regulation of fibrinolysis in plasma by TAFI and protein C is dependent on the concentration of thrombomodulin

Plasma TAFI levels influence the clot lysis time in healthy individuals in the presence of an intact intrinsic pathway of coagulation

Decreased active von Willebrand factor level owing to shear stress in aortic stenosis patients

The defective down regulation of fibrinolysis in haemophilia A can be restored by increasing the TAFI plasma concentration

Increased fibrinolytic activity during use of oral contraceptives is counteracted by an enhanced factor XI-independent down regulation of fibrinolysis: a randomized cross-over study of two low-dose oral contraceptives

Impaired healing of cutaneous wounds and colonic anastomoses in mice lacking thrombin‐activatable fibrinolysis inhibitor

Feedback Activation of Factor XI by Thrombin in Plasma Results in Additional Formation of Thrombin That Protects Fibrin Clots From Fibrinolysis

Effects of second and third generation oral contraceptives and their respective progestagens on the coagulation system in the absence or presence of the factor V Leiden mutation

Factor VIII and von Willebrand factor

Decreased plasma sensitivity to activated protein C by oral contraceptives is associated with decreases in plasma glucosylceramide

Antiphospholipid Antibodies Directed Against a Combination of Phospholipids With Prothrombin, Protein C, or Protein S: An Explanation for Their Pathogenic Mechanism?

Protein C inhibitor regulates the thrombin-thrombomodulin complex in the up- and down regulation of TAFI activation

The role of protein S in the activation of thrombin activatable fibrinolysis inhibitor (TAFI) and regulation of fibrinolysis

Unraveling the mystery of von Willebrand factor

Thrombin-Activatable Fibrinolysis Inhibitor (TAFI, Plasma Procarboxypeptidase B, Procarboxypeptidase R, Procarboxypeptidase U)

Reduced activity of TAFI (thrombin‐activatable fibrinolysis inhibitor) in acute promyelocytic leukaemia

Factor XI dependent and independent activation of thrombin activatable fibrinolysis inhibitor (TAFI) in plasma associated with clot formation

Factor XI and protection of the fibrin clot against lysis--a role for the intrinsic pathway of coagulation in fibrinolysis

Tumor growth and metastasis are not affected in thrombin‐activatable fibrinolysis inhibitor‐deficient mice

Role of isoleucine residues 182 and 183 in thrombin‐activatable fibrinolysis inhibitor

The Affinity and Stoichiometry of Binding of Human Factor VIII to von Willebrand Factor

C4b-binding protein inhibits the factor V-dependent but not the factor V-independent cofactor activity of protein S in the activated protein C-mediated inactivation of factor VIIIa

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