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Proteasome Function Is Regulated by Cyclic AMP-dependent Protein Kinase through Phosphorylation of Rpt6

The Journal of biological chemistry, 2007-08, Vol.282 (31), p.22460-22471

2007

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O-GlcNAc Modification Is an Endogenous Inhibitor of the Proteasome

Cell, 2003-12, Vol.115 (6), p.715-725

2003

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Post-translational modification by O-GlcNAc: Another way to change protein function

Journal of cellular biochemistry, 2006-08, Vol.98 (5), p.1062-1075

Kudlow, Jeffrey E.

2006

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Characterization of the Histone Acetyltransferase (HAT) Domain of a Bifunctional Protein with Activable O-GlcNAcase and HAT Activities

The Journal of biological chemistry, 2004-12, Vol.279 (51), p.53665-53673

2004

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O-Linkage of N-Acetylglucosamine to Sp1 Activation Domain Inhibits Its Transcriptional Capability

Proceedings of the National Academy of Sciences - PNAS, 2001-06, Vol.98 (12), p.6611-6616

2001

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Phosphorylation of Mouse Glutamine-Fructose-6-phosphate Amidotransferase 2 (GFAT2) by cAMP-dependent Protein Kinase Increases the Enzyme Activity

The Journal of biological chemistry, 2004-07, Vol.279 (29), p.29988-29993

2004

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Keratin 14 Cre transgenic mice authenticate keratin 14 as an oocyte-expressed protein

Genesis (New York, N.Y. : 2000), 2004-04, Vol.38 (4), p.176-181

2004

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Alloxan is an inhibitor of the enzyme O-linked N-acetylglucosamine transferase

Biochemical and biophysical research communications, 2002-04, Vol.293 (1), p.207-212

2002

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Streptozotocin inhibits O-GlcNAcase via the production of a transition state analog

Biochemical and biophysical research communications, 2006-02, Vol.340 (2), p.526-534

2006

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Recruitment of O-GlcNAc Transferase to Promoters by Corepressor mSin3A : Coupling Protein O-GlcNAcylation to Transcriptional Repression

Cell, 2002-07, Vol.110 (1), p.69-80

2002

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O-GlcNAc Integrates the Proteasome and Transcriptome To Regulate Nuclear Hormone Receptors

Molecular and Cellular Biology, 2006-11, Vol.26 (22), p.8539-8550

2006

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The Histone Acetyltransferase NCOAT Contains a Zinc Finger-like Motif Involved in Substrate Recognition

The Journal of biological chemistry, 2006-02, Vol.281 (7), p.3918-3925

2006

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Targeted Expression of a Dominant Negative Epidermal Growth Factor Receptor in the Mammary Gland of Transgenic Mice Inhibits Pubertal Mammary Duct Development

Molecular endocrinology (Baltimore, Md.), 1997-11, Vol.11 (12), p.1766-1781

1997

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Glucose Stimulates Protein Modification by O-Linked GlcNAc in Pancreatic β Cells: Linkage of O-Linked GlcNAc to β Cell Death

Proceedings of the National Academy of Sciences - PNAS, 2000-03, Vol.97 (6), p.2820-2825

2000

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Disrupting the enzyme complex regulating O-GlcNAcylation blocks signaling and development

Glycobiology (Oxford), 2006-06, Vol.16 (6), p.551-563

2006

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Stage-Sensitive Blockade of Pituitary Somatomammotrope Development by Targeted Expression of a Dominant Negative Epidermal Growth Factor Receptor in Transgenic Mice

Molecular endocrinology (Baltimore, Md.), 2001-04, Vol.15 (4), p.600-613

2001

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Streptozotocin, an O-GlcNAcase inhibitor, blunts insulin and growth hormone secretion

Molecular and cellular endocrinology, 2002-08, Vol.194 (1), p.135-146

2002

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O-GlcNAcylation enhances FOXO4 transcriptional regulation in response to stress

FEBS letters, 2010-01, Vol.584 (1), p.49-54

2010

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Metabolic Control of Proteasome Function

Physiology (Bethesda, Md.), 2007-12, Vol.22 (6), p.373-379

2007

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O glycosylation of an Sp1-derived peptide blocks known Sp1 protein interactions

Molecular and Cellular Biology, 1997-11, Vol.17 (11), p.6472-6480

1997

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Location and characterization of the O-GlcNAcase active site

Biochimica et biophysica acta, 2006-05, Vol.1760 (5), p.829-839

2006

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Increased O-GlcNAc causes disrupted lens fiber cell differentiation and cataracts

Biochemical and biophysical research communications, 2009-09, Vol.387 (1), p.70-76

2009

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Muscle-specific overexpression of NCOATGK, splice variant of O-GlcNAcase, induces skeletal muscle atrophy

American Journal of Physiology: Cell Physiology, 2011-03, Vol.300 (3), p.C456-C465

2011

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The role of O-linked protein glycosylation in beta-cell dysfunction

International journal of molecular medicine, 2002-11, Vol.10 (5), p.535

2002

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Phosphorylation of Human Glutamine:Fructose-6-phosphate Amidotransferase by cAMP-dependent Protein Kinase at Serine 205 Blocks the Enzyme Activity

The Journal of biological chemistry, 2000-07, Vol.275 (29), p.21981-21987

2000

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Proteasome Function Is Regulated by Cyclic AMP-dependent Protein Kinase through Phosphorylation of Rpt6

O-GlcNAc Modification Is an Endogenous Inhibitor of the Proteasome

Post-translational modification by O-GlcNAc: Another way to change protein function

Characterization of the Histone Acetyltransferase (HAT) Domain of a Bifunctional Protein with Activable O-GlcNAcase and HAT Activities

O-Linkage of N-Acetylglucosamine to Sp1 Activation Domain Inhibits Its Transcriptional Capability

Phosphorylation of Mouse Glutamine-Fructose-6-phosphate Amidotransferase 2 (GFAT2) by cAMP-dependent Protein Kinase Increases the Enzyme Activity

Keratin 14 Cre transgenic mice authenticate keratin 14 as an oocyte-expressed protein

Alloxan is an inhibitor of the enzyme O-linked N-acetylglucosamine transferase

Streptozotocin inhibits O-GlcNAcase via the production of a transition state analog

Recruitment of O-GlcNAc Transferase to Promoters by Corepressor mSin3A : Coupling Protein O-GlcNAcylation to Transcriptional Repression

O-GlcNAc Integrates the Proteasome and Transcriptome To Regulate Nuclear Hormone Receptors

The Histone Acetyltransferase NCOAT Contains a Zinc Finger-like Motif Involved in Substrate Recognition

Targeted Expression of a Dominant Negative Epidermal Growth Factor Receptor in the Mammary Gland of Transgenic Mice Inhibits Pubertal Mammary Duct Development

Glucose Stimulates Protein Modification by O-Linked GlcNAc in Pancreatic β Cells: Linkage of O-Linked GlcNAc to β Cell Death

Disrupting the enzyme complex regulating O-GlcNAcylation blocks signaling and development

Stage-Sensitive Blockade of Pituitary Somatomammotrope Development by Targeted Expression of a Dominant Negative Epidermal Growth Factor Receptor in Transgenic Mice

Streptozotocin, an O-GlcNAcase inhibitor, blunts insulin and growth hormone secretion

O-GlcNAcylation enhances FOXO4 transcriptional regulation in response to stress

Metabolic Control of Proteasome Function

O glycosylation of an Sp1-derived peptide blocks known Sp1 protein interactions

Location and characterization of the O-GlcNAcase active site

Increased O-GlcNAc causes disrupted lens fiber cell differentiation and cataracts

Muscle-specific overexpression of NCOATGK, splice variant of O-GlcNAcase, induces skeletal muscle atrophy

The role of O-linked protein glycosylation in beta-cell dysfunction

Phosphorylation of Human Glutamine:Fructose-6-phosphate Amidotransferase by cAMP-dependent Protein Kinase at Serine 205 Blocks the Enzyme Activity

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