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The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2020-12, Vol.26 (1), p.1-98

2020

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Carbon monoxide provides protection against hyperoxic lung injury

American journal of physiology. Lung cellular and molecular physiology, 1999-04, Vol.276 (4), p.688-L694

1999

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Correction to: GTS-21, an α7nAChR agonist, increases pulmonary bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2021-08, Vol.27 (1), p.93-93, Article 93

2021

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From nicotine to the cholinergic anti-inflammatory reflex - Can nicotine alleviate the dysregulated inflammation in COVID-19?

Journal of immunotoxicology, 2021-01, Vol.18 (1), p.23-29

2021

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HMGB-1 as a therapeutic target for infectious and inflammatory disorders

Shock (Augusta, Ga.), 2006, Vol.25 (1), p.4-11

2006

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Exogenous administration of heme oxygenase-1 by gene transfer provides protection against hyperoxia-induced lung injury

The Journal of clinical investigation, 1999-04, Vol.103 (7), p.1047-1054

1999

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Retraction Note: The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2020-12, Vol.26 (1), p.132-132, Article 132

2020

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Hyperoxia sensing: From molecular mechanisms to significance in disease

Journal of immunotoxicology, 2010-12, Vol.7 (4), p.239-254

2010

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Correction to: 2‑O, 3‑O desulfated heparin (ODSH) increases bacterial clearance and attenuates lung injury in cystic fibrosis by restoring HMGB1‑compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2021-09, Vol.27 (1), p.98-98, Article 98

2021

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Expression of Concern to: The α7 nicotine acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2020-02, Vol.26 (1), p.16-16, Article 16

2020

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Our Fragile Water Planet: An Introduction to the Earth Sciences

Mantell, C

1, 1976

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Signal Transduction Pathways in Hyperoxia-Induced Lung Cell Death

Molecular Genetics and Metabolism, 2000-09, Vol.71 (1-2), p.359-370

2000

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Carbon Nanomaterials Stimulate HMGB1 Release From Macrophages and Induce Cell Migration and Invasion

Toxicological sciences, 2019-12, Vol.172 (2), p.398-410

2019

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The nitric oxide donor, (Z)-1-[N-(2-aminoethyl)-N-(2-ammonioethyl)amino]diazen-1-ium-1,2-diolate (DETA-NONOate/D-NO), increases survival by attenuating hyperoxia-compromised innate immunity in bacterial clearance in a mouse model of ventilator-associated pneumonia

Biochemical pharmacology, 2020-06, Vol.176, p.113817-113817, Article 113817

2020

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Mitogen-activated protein kinase pathway mediates hyperoxia-induced apoptosis in cultured macrophage cells

American journal of physiology. Lung cellular and molecular physiology, 1999-09, Vol.277 (3), p.589-L595

1999

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Alpha-chemokine receptor blockade reduces high mobility group box 1 protein-induced lung inflammation and injury and improves survival in sepsis

American journal of physiology. Lung cellular and molecular physiology, 2005-10, Vol.289 (4), p.L583-L590

2005

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Nitric Oxide Regulates Gene Expression in Cancers by Controlling Histone Posttranslational Modifications

Cancer research (Chicago, Ill.), 2015-12, Vol.75 (24), p.5299-5308

2015

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A32 MICROBIAL INTERACTIONS AND HOST DEFENSE: Host And Pathogen Factors Modulate Antibacterial Properties Of Histones And Non-Histone Protein Hmgb1

American journal of respiratory and critical care medicine, 2015-01, Vol.191, p.1

2015

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GAT107-mediated α7 nicotinic acetylcholine receptor signaling attenuates inflammatory lung injury and mortality in a mouse model of ventilator-associated pneumonia by alleviating macrophage mitochondrial oxidative stress via reducing MnSOD-S-glutathionylation

Redox biology, 2023-04, Vol.60, p.102614-102614, Article 102614

2023

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C73 OXIDANTS: Hyperoxia-Compromised Phagocytic Function In Macrophages Is Improved By Salicylates Via Inhibition Of Hmgb1 Release

American journal of respiratory and critical care medicine, 2016-01, Vol.193, p.1

2016

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Hyperoxia-induced Cell Death in the Lung-the Correlation of Apoptosis, Necrosis, and Inflammation

Annals of the New York Academy of Sciences, 1999-12, Vol.887 (1), p.171-180

1999

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Partially-desulfated heparin improves survival in Pseudomonas pneumonia by enhancing bacterial clearance and ameliorating lung injury

Journal of immunotoxicology, 2014-07, Vol.11 (3), p.260-267

2014

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Pulmonary apoptosis in aged and oxygen-tolerant rats exposed to hyperoxia

American journal of physiology. Lung cellular and molecular physiology, 1998-07, Vol.275 (1), p.14-L20

1998

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A37 CYSTIC FIBROSIS: IMMUNE REGULATORY PATHWAYS: Cf Airway Epithelia Contribute To The Compromised Innate Immunity Against Bacterial Infection

American journal of respiratory and critical care medicine, 2016-01, Vol.193, p.1

2016

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A57 LUNG INJURY, REPAIR, AND FIBROSIS: THE PLOT THICKENS FOR THREE'S COMPANY: Mnsod Prevents Hyperoxia-Induced Cell Death Via Erk Activation

American journal of respiratory and critical care medicine, 2015-01, Vol.191, p.1

2015

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The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Carbon monoxide provides protection against hyperoxic lung injury

Correction to: GTS-21, an α7nAChR agonist, increases pulmonary bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

From nicotine to the cholinergic anti-inflammatory reflex - Can nicotine alleviate the dysregulated inflammation in COVID-19?

HMGB-1 as a therapeutic target for infectious and inflammatory disorders

Exogenous administration of heme oxygenase-1 by gene transfer provides protection against hyperoxia-induced lung injury

Retraction Note: The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Hyperoxia sensing: From molecular mechanisms to significance in disease

Correction to: 2‑O, 3‑O desulfated heparin (ODSH) increases bacterial clearance and attenuates lung injury in cystic fibrosis by restoring HMGB1‑compromised macrophage function

Expression of Concern to: The α7 nicotine acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Our Fragile Water Planet: An Introduction to the Earth Sciences

Signal Transduction Pathways in Hyperoxia-Induced Lung Cell Death

Carbon Nanomaterials Stimulate HMGB1 Release From Macrophages and Induce Cell Migration and Invasion

The nitric oxide donor, (Z)-1-[N-(2-aminoethyl)-N-(2-ammonioethyl)amino]diazen-1-ium-1,2-diolate (DETA-NONOate/D-NO), increases survival by attenuating hyperoxia-compromised innate immunity in bacterial clearance in a mouse model of ventilator-associated pneumonia

Mitogen-activated protein kinase pathway mediates hyperoxia-induced apoptosis in cultured macrophage cells

Alpha-chemokine receptor blockade reduces high mobility group box 1 protein-induced lung inflammation and injury and improves survival in sepsis

Nitric Oxide Regulates Gene Expression in Cancers by Controlling Histone Posttranslational Modifications

A32 MICROBIAL INTERACTIONS AND HOST DEFENSE: Host And Pathogen Factors Modulate Antibacterial Properties Of Histones And Non-Histone Protein Hmgb1

GAT107-mediated α7 nicotinic acetylcholine receptor signaling attenuates inflammatory lung injury and mortality in a mouse model of ventilator-associated pneumonia by alleviating macrophage mitochondrial oxidative stress via reducing MnSOD-S-glutathionylation

C73 OXIDANTS: Hyperoxia-Compromised Phagocytic Function In Macrophages Is Improved By Salicylates Via Inhibition Of Hmgb1 Release

Hyperoxia-induced Cell Death in the Lung-the Correlation of Apoptosis, Necrosis, and Inflammation

Partially-desulfated heparin improves survival in Pseudomonas pneumonia by enhancing bacterial clearance and ameliorating lung injury

Pulmonary apoptosis in aged and oxygen-tolerant rats exposed to hyperoxia

A37 CYSTIC FIBROSIS: IMMUNE REGULATORY PATHWAYS: Cf Airway Epithelia Contribute To The Compromised Innate Immunity Against Bacterial Infection

A57 LUNG INJURY, REPAIR, AND FIBROSIS: THE PLOT THICKENS FOR THREE'S COMPANY: Mnsod Prevents Hyperoxia-Induced Cell Death Via Erk Activation

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