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The CDK9 Inhibitor Dinaciclib Exerts Potent Apoptotic and Antitumor Effects in Preclinical Models of MLL-Rearranged Acute Myeloid Leukemia

Cancer research (Chicago, Ill.), 2016-03, Vol.76 (5), p.1158-1169

2016

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Functional Identification of Tumor-Suppressor Genes through an In Vivo RNA Interference Screen in a Mouse Lymphoma Model

Cancer cell, 2009-10, Vol.16 (4), p.324-335

2009

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Histone Acetyltransferase Activity of MOF Is Required for MLL-AF9 Leukemogenesis

Cancer research (Chicago, Ill.), 2017-04, Vol.77 (7), p.1753-1762

2017

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Id2 and E Proteins Orchestrate the Initiation and Maintenance of MLL-Rearranged Acute Myeloid Leukemia

Cancer cell, 2016-07, Vol.30 (1), p.59-74

2016

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Functional Analysis of Non-Genetic Resistance to Platinum in Epithelial Ovarian Cancer Reveals a Role for the MBD3-NuRD Complex in Resistance Development

Cancers, 2021-07, Vol.13 (15), p.3801

2021

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Polycomb protein RING1A limits hematopoietic differentiation in myelodysplastic syndromes

Oncotarget, 2017-12, Vol.8 (70), p.115002-115017

2017

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Cancer stem cell definitions and terminology: the devil is in the details

Nature reviews. Cancer, 2012-11, Vol.12 (11), p.767

2012

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MLL3 Is a Haploinsufficient 7q Tumor Suppressor in Acute Myeloid Leukemia

Cancer cell, 2014-05, Vol.25 (5), p.652-665

2014

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Selective Requirement of MYB for Oncogenic Hyperactivation of a Translocated Enhancer in Leukemia

Cancer discovery, 2021-11, Vol.11 (11), p.2868-2883

2021

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Small-molecule inhibition of BRD4 as a new potent approach to eliminate leukemic stem- and progenitor cells in acute myeloid leukemia (AML)

Oncotarget, 2012-12, Vol.3 (12), p.1588-1599

2012

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TCEA1 regulates the proliferative potential of mouse myeloid cells

Experimental cell research, 2018-09, Vol.370 (2), p.551-560

2018

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Cancer Evolution: A Multifaceted Affair

Cancer discovery, 2024-01, Vol.14 (1), p.36

2024

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Development of siRNA payloads to target KRAS-mutant cancer

Cancer discovery, 2014-10, Vol.4 (10), p.1182-1197

2014

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CDK6 Antagonizes p53-Induced Responses during Tumorigenesis

Cancer discovery, 2018-07, Vol.8 (7), p.884-897

2018

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Drug-induced inhibition of phosphorylation of STAT5 overrides drug resistance in neoplastic mast cells

Leukemia, 2018-04, Vol.32 (4), p.1016-1022

2018

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MEK1 is required for the development of NRAS-driven leukemia

Oncotarget, 2016-12, Vol.7 (49), p.80113-80130

2016

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Leukemia Cell of Origin Influences Apoptotic Priming and Sensitivity to LSD1 Inhibition

Cancer discovery, 2020-10, Vol.10 (10), p.1500-1513

2020

Volltextzugriff (PDF)

Direct and indirect targeting of MYC to treat acute myeloid leukemia

Cancer chemotherapy and pharmacology, 2015-07, Vol.76 (1), p.35-46

2015

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Identification of bromodomain-containing protein-4 as a novel marker and epigenetic target in mast cell leukemia

Leukemia, 2015-11, Vol.29 (11), p.2230-2237

2015

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Combining the differentiating effect of panobinostat with the apoptotic effect of arsenic trioxide leads to significant survival benefit in a model of t(8;21) acute myeloid leukemia

Clinical epigenetics, 2015-01, Vol.7 (1), p.2-2, Article 2

2015

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The Polycomb complex PRC2 supports aberrant self-renewal in a mouse model of MLL-AF9;NrasG12D acute myeloid leukemia

Oncogene, 2013-02, Vol.32 (7), p.930-938

2013

Volltextzugriff (PDF)

Effective use of high-dose chemotherapy and autologous stem cell rescue for relapsed adult Wilms' tumor and a novel alteration in intron 1 of the WT1 gene

Journal of pediatric hematology/oncology, 2004-12, Vol.26 (12), p.820

2004

Volltextzugriff (PDF)

Markers of tumor angiogenesis and proteolysis independently define high- and low-risk subsets of node-negative breast cancer patients

Journal of clinical oncology, 1998-09, Vol.16 (9), p.3129-3136

1998

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The CDK9 Inhibitor Dinaciclib Exerts Potent Apoptotic and Antitumor Effects in Preclinical Models of MLL-Rearranged Acute Myeloid Leukemia

Functional Identification of Tumor-Suppressor Genes through an In Vivo RNA Interference Screen in a Mouse Lymphoma Model

Histone Acetyltransferase Activity of MOF Is Required for MLL-AF9 Leukemogenesis

Id2 and E Proteins Orchestrate the Initiation and Maintenance of MLL-Rearranged Acute Myeloid Leukemia

Functional Analysis of Non-Genetic Resistance to Platinum in Epithelial Ovarian Cancer Reveals a Role for the MBD3-NuRD Complex in Resistance Development

Polycomb protein RING1A limits hematopoietic differentiation in myelodysplastic syndromes

Cancer stem cell definitions and terminology: the devil is in the details

MLL3 Is a Haploinsufficient 7q Tumor Suppressor in Acute Myeloid Leukemia

Acquired resistance to anti-MAPK targeted therapy confers an immune-evasive tumor microenvironment and cross-resistance to immunotherapy in melanoma

Selective Requirement of MYB for Oncogenic Hyperactivation of a Translocated Enhancer in Leukemia

Small-molecule inhibition of BRD4 as a new potent approach to eliminate leukemic stem- and progenitor cells in acute myeloid leukemia (AML)

TCEA1 regulates the proliferative potential of mouse myeloid cells

Cancer Evolution: A Multifaceted Affair

Development of siRNA payloads to target KRAS-mutant cancer

CDK6 Antagonizes p53-Induced Responses during Tumorigenesis

Drug-induced inhibition of phosphorylation of STAT5 overrides drug resistance in neoplastic mast cells

MEK1 is required for the development of NRAS-driven leukemia

Leukemia Cell of Origin Influences Apoptotic Priming and Sensitivity to LSD1 Inhibition

Direct and indirect targeting of MYC to treat acute myeloid leukemia

Identification of bromodomain-containing protein-4 as a novel marker and epigenetic target in mast cell leukemia

Combining the differentiating effect of panobinostat with the apoptotic effect of arsenic trioxide leads to significant survival benefit in a model of t(8;21) acute myeloid leukemia

The Polycomb complex PRC2 supports aberrant self-renewal in a mouse model of MLL-AF9;NrasG12D acute myeloid leukemia

Effective use of high-dose chemotherapy and autologous stem cell rescue for relapsed adult Wilms' tumor and a novel alteration in intron 1 of the WT1 gene

Markers of tumor angiogenesis and proteolysis independently define high- and low-risk subsets of node-negative breast cancer patients

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