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Abstract 12275: Knock-down of Insulin-like Growth Factor-1 Receptor in the Endothelium Decreases Anti-oxidant Capacity of Aorta, Leading to Increased Atherosclerosis

Circulation (New York, N.Y.), 2015-11, Vol.132 (Suppl_3 Suppl 3), p.A12275-A12275

2015

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Abstract 13092: Macrophage Specific IGF1 Overexpression on ApoE Deficient Mice Decreases Atherosclerosis, Reduces CXCL12 Chemokine Expression, and Increases Cholesterol Efflux

Circulation (New York, N.Y.), 2021-11, Vol.144 (Suppl_1)

2021

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Abstract 14917: Insulin Growth Factor 1 Reduces Matrix Metalloproteinase Expression in Macrophages and Induces a Stable Atherosclerotic Plaque Phenotype

Circulation (New York, N.Y.), 2018-11, Vol.138 (Suppl_1 Suppl 1), p.A14917-A14917

2018

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Abstract 17103: Insulin-like Growth Factor I (IGF-1) Suppresses Chemokines and Reduces Recruitment of Monocytes into Atherosclerotic Plaque: Potential Mechanism Mediating IGF-1-induced Atheroprotection

Circulation (New York, N.Y.), 2016-11, Vol.134 (Suppl_1 Suppl 1), p.A17103-A17103

2016

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Abstract 17104: Smooth Muscle Protein 22 alpha Promoter-driven Insulin-like Growth Factor I (IGF-1) Receptor Deficiency Increases Atherosclerosis and Promotes Atherosclerotic Plaque Destabilization

Circulation (New York, N.Y.), 2016-11, Vol.134 (Suppl_1 Suppl 1), p.A17104-A17104

2016

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Abstract 464: Knockout of Insulin-like Growth Factor-1 Receptor in Monocyte/Macrophage Promotes Atherosclerosis and Induces an Unstable Plaque Phenotype

Arteriosclerosis, thrombosis, and vascular biology, 2013-05, Vol.33 (suppl_1)

2013

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Abstract 5276: Insulin-like Growth Factor I Downregulates Vascular Smooth Muscle and Macrophage 12/15-Lipoxygenase Expression: Potential Mechanism for Igf-1-induced Atheroprotection

Circulation (New York, N.Y.), 2009-11, Vol.120 (suppl_18)

2009

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Aging: Aging, Atherosclerosis, and IGF-1

The journals of gerontology. Series A, Biological sciences and medical sciences, 2012-06, Vol.67A (6), p.626

2012

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Aging, atherosclerosis, and IGF-1

The journals of gerontology. Series A, Biological sciences and medical sciences, 2012-06, Vol.67 (6), p.626-639

2012

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Beta(1) integrins participate in the hypertrophic response of rat ventricular myocytes

Circulation research, 1998-06, Vol.82 (11), p.1160

1998

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beta1 Integrins Participate in the Hypertrophic Response of Rat Ventricular Myocytes

Circulation research, 1998-06, Vol.82 (11), p.1160-1172

1998

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Bovine Angiotensin Converting Enzyme cDNA Cloning and Regulation: Increased Expression During Endothelial Cell Growth Arrest

Circulation research, 1992-06, Vol.70 (6), p.1274-1281

1992

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Cardiac myocyte-specific excision of the (beta)1 integrin gene results in myocardial fibrosis and cardiac failure

Circulation research, 2002-03, Vol.90 (4), p.458

2002

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Cardiac myocyte-specific excision of the beta1 integrin gene results in myocardial fibrosis and cardiac failure

Circulation research, 2002-03, Vol.90 (4), p.458-464

2002

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Cardiac Myocyte-Specific Excision of the β1 Integrin Gene Results in Myocardial Fibrosis and Cardiac Failure

Circulation research, 2002-03, Vol.90 (4), p.458-464

2002

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Characterization of genes encoding rat tonin and a kallikrein-like serine protease

Biochemistry (Easton), 1989-06, Vol.28 (13), p.5334-5343

1989

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Combined deficiency of dystrophin and beta1 integrin in the cardiac myocyte causes myocardial dysfunction, fibrosis and calcification

Circulation research, 2008-05, Vol.102 (9), p.1109

2008

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Combined Deficiency of Dystrophin and β1 Integrin in the Cardiac Myocyte Causes Myocardial Dysfunction, Fibrosis and Calcification

Circulation research, 2008-05, Vol.102 (9), p.1109-1117

2008

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Disruption of Integrin Function in the Murine Myocardium Leads to Perinatal Lethality, Fibrosis, and Abnormal Cardiac Performance

The American journal of pathology, 2001-03, Vol.158 (3), p.1079-1090

2001

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Endothelial Expression of Beta‐1 Integrin Is Required for Embryonic Vascular Patterning and Postnatal Vascular Remodeling

The FASEB journal, 2008-03, Vol.22 (S1)

2008

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Endothelial expression of beta1 integrin is required for embryonic vascular patterning and postnatal vascular remodeling

Molecular and cellular biology, 2008-01, Vol.28 (2), p.794

2008

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Endothelial Expression of β1 Integrin Is Required for Embryonic Vascular Patterning and Postnatal Vascular Remodeling

Molecular and Cellular Biology, 2008-01, Vol.28 (2), p.794-802

2008

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Features of Stable Plaque Phenotype Are Increased by Macrophage Specific Insulin‐Like Growth Factor 1

The FASEB journal, 2020-04, Vol.34 (S1), p.1-1

2020

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Fibroblast growth factor stimulates angiotensin converting enzyme expression in vascular smooth muscle cells. Possible mediator of the response to vascular injury

The Journal of clinical investigation, 1995-01, Vol.95 (1), p.377-387

1995

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Genomic DNA 5′ to the mouse and human angiotensin-converting enzyme genes contains two distinct regions of conserved sequence

Biochemical and biophysical research communications, 1990-03, Vol.167 (3), p.1128-1133

1990

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Abstract 12275: Knock-down of Insulin-like Growth Factor-1 Receptor in the Endothelium Decreases Anti-oxidant Capacity of Aorta, Leading to Increased Atherosclerosis

Abstract 13092: Macrophage Specific IGF1 Overexpression on ApoE Deficient Mice Decreases Atherosclerosis, Reduces CXCL12 Chemokine Expression, and Increases Cholesterol Efflux

Abstract 14917: Insulin Growth Factor 1 Reduces Matrix Metalloproteinase Expression in Macrophages and Induces a Stable Atherosclerotic Plaque Phenotype

Abstract 17103: Insulin-like Growth Factor I (IGF-1) Suppresses Chemokines and Reduces Recruitment of Monocytes into Atherosclerotic Plaque: Potential Mechanism Mediating IGF-1-induced Atheroprotection

Abstract 17104: Smooth Muscle Protein 22 alpha Promoter-driven Insulin-like Growth Factor I (IGF-1) Receptor Deficiency Increases Atherosclerosis and Promotes Atherosclerotic Plaque Destabilization

Abstract 464: Knockout of Insulin-like Growth Factor-1 Receptor in Monocyte/Macrophage Promotes Atherosclerosis and Induces an Unstable Plaque Phenotype

Abstract 5276: Insulin-like Growth Factor I Downregulates Vascular Smooth Muscle and Macrophage 12/15-Lipoxygenase Expression: Potential Mechanism for Igf-1-induced Atheroprotection

Aging: Aging, Atherosclerosis, and IGF-1

Aging, atherosclerosis, and IGF-1

Beta(1) integrins participate in the hypertrophic response of rat ventricular myocytes

beta1 Integrins Participate in the Hypertrophic Response of Rat Ventricular Myocytes

Bovine Angiotensin Converting Enzyme cDNA Cloning and Regulation: Increased Expression During Endothelial Cell Growth Arrest

Cardiac myocyte-specific excision of the (beta)1 integrin gene results in myocardial fibrosis and cardiac failure

Cardiac myocyte-specific excision of the beta1 integrin gene results in myocardial fibrosis and cardiac failure

Cardiac Myocyte-Specific Excision of the β1 Integrin Gene Results in Myocardial Fibrosis and Cardiac Failure

Characterization of genes encoding rat tonin and a kallikrein-like serine protease

Combined deficiency of dystrophin and beta1 integrin in the cardiac myocyte causes myocardial dysfunction, fibrosis and calcification

Combined Deficiency of Dystrophin and β1 Integrin in the Cardiac Myocyte Causes Myocardial Dysfunction, Fibrosis and Calcification

Disruption of Integrin Function in the Murine Myocardium Leads to Perinatal Lethality, Fibrosis, and Abnormal Cardiac Performance

Endothelial Expression of Beta‐1 Integrin Is Required for Embryonic Vascular Patterning and Postnatal Vascular Remodeling

Endothelial expression of beta1 integrin is required for embryonic vascular patterning and postnatal vascular remodeling

Endothelial Expression of β1 Integrin Is Required for Embryonic Vascular Patterning and Postnatal Vascular Remodeling

Features of Stable Plaque Phenotype Are Increased by Macrophage Specific Insulin‐Like Growth Factor 1

Fibroblast growth factor stimulates angiotensin converting enzyme expression in vascular smooth muscle cells. Possible mediator of the response to vascular injury

Genomic DNA 5′ to the mouse and human angiotensin-converting enzyme genes contains two distinct regions of conserved sequence

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