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Dietary Antioxidants Significantly Attenuate Hyperoxia-Induced Acute Inflammatory Lung Injury by Enhancing Macrophage Function via Reducing the Accumulation of Airway HMGB1

International journal of molecular sciences, 2020-02, Vol.21 (3), p.977

2020

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A Standardized Botanical Composition Mitigated Acute Inflammatory Lung Injury and Reduced Mortality through Extracellular HMGB1 Reduction

Molecules (Basel, Switzerland), 2023-09, Vol.28 (18), p.6560

2023

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Correction to: GTS-21, an α7nAChR agonist, increases pulmonary bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2021-08, Vol.27 (1), p.93-93, Article 93

2021

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From nicotine to the cholinergic anti-inflammatory reflex - Can nicotine alleviate the dysregulated inflammation in COVID-19?

Journal of immunotoxicology, 2021-01, Vol.18 (1), p.23-29

2021

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Retraction Note: The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2020-12, Vol.26 (1), p.132-132, Article 132

2020

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Correction to: 2‑O, 3‑O desulfated heparin (ODSH) increases bacterial clearance and attenuates lung injury in cystic fibrosis by restoring HMGB1‑compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2021-09, Vol.27 (1), p.98-98, Article 98

2021

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Expression of Concern to: The α7 nicotine acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2020-02, Vol.26 (1), p.16-16, Article 16

2020

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The Role of HMGB1, a Nuclear Damage-Associated Molecular Pattern Molecule, in the Pathogenesis of Lung Diseases

Antioxidants & redox signaling, 2019-11, Vol.31 (13), p.954-993

2019

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Our Fragile Water Planet: An Introduction to the Earth Sciences

Mantell, C

1, 1976

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Carbon Nanomaterials Stimulate HMGB1 Release From Macrophages and Induce Cell Migration and Invasion

Toxicological sciences, 2019-12, Vol.172 (2), p.398-410

2019

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Dual effects of supplemental oxygen on pulmonary infection, inflammatory lung injury, and neuromodulation in aging and COVID-19

Free radical biology & medicine, 2022-09, Vol.190, p.247-263

2022

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The nitric oxide donor, (Z)-1-[N-(2-aminoethyl)-N-(2-ammonioethyl)amino]diazen-1-ium-1,2-diolate (DETA-NONOate/D-NO), increases survival by attenuating hyperoxia-compromised innate immunity in bacterial clearance in a mouse model of ventilator-associated pneumonia

Biochemical pharmacology, 2020-06, Vol.176, p.113817-113817, Article 113817

2020

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In memoriam Steven Yue Qian (1960–2019)

Redox biology, 2020-10, Vol.37, p.101821, Article 101821

2020

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Nitric Oxide Regulates Gene Expression in Cancers by Controlling Histone Posttranslational Modifications

Cancer research (Chicago, Ill.), 2015-12, Vol.75 (24), p.5299-5308

2015

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A32 MICROBIAL INTERACTIONS AND HOST DEFENSE: Host And Pathogen Factors Modulate Antibacterial Properties Of Histones And Non-Histone Protein Hmgb1

American journal of respiratory and critical care medicine, 2015-01, Vol.191, p.1

2015

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Hyperoxia Inhibits Oxidant-induced Apoptosis in Lung Epithelial Cells

The Journal of biological chemistry, 2001-01, Vol.276 (1), p.569-575

2001

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Introduction to serial reviews on redox signaling in immune function and cellular responses in lung injury and diseases

Free radical biology & medicine, 2006-07, Vol.41 (1), p.1-3

Mantell, Lin L.

2006

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Interleukin-11 and Interleukin-6 Protect Cultured Human Endothelial Cells from H2O2-Induced Cell Death

American journal of respiratory cell and molecular biology, 2003-10, Vol.29 (4), p.513

2003

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C73 OXIDANTS: Hyperoxia-Compromised Phagocytic Function In Macrophages Is Improved By Salicylates Via Inhibition Of Hmgb1 Release

American journal of respiratory and critical care medicine, 2016-01, Vol.193, p.1

2016

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Overexpression of Manganese Superoxide Dismutase Protects Lung Epithelial Cells against Oxidant Injury

American journal of respiratory cell and molecular biology, 2001-04, Vol.24 (4), p.436-441

2001

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Ascorbic Acid Attenuates Hyperoxia-Compromised Host Defense against Pulmonary Bacterial Infection

American journal of respiratory cell and molecular biology, 2016-10, Vol.55 (4), p.511-520

2016

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Nuclear Factor-κB Is Activated by Hyperoxia but Does Not Protect from Cell Death

The Journal of biological chemistry, 1997-08, Vol.272 (33), p.20646-20649

1997

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A37 CYSTIC FIBROSIS: IMMUNE REGULATORY PATHWAYS: Cf Airway Epithelia Contribute To The Compromised Innate Immunity Against Bacterial Infection

American journal of respiratory and critical care medicine, 2016-01, Vol.193, p.1

2016

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A57 LUNG INJURY, REPAIR, AND FIBROSIS: THE PLOT THICKENS FOR THREE'S COMPANY: Mnsod Prevents Hyperoxia-Induced Cell Death Via Erk Activation

American journal of respiratory and critical care medicine, 2015-01, Vol.191, p.1

2015

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Unscheduled apoptosis during acute inflammatory lung injury

Cell death and differentiation, 1997-10, Vol.4 (7), p.600-607

1997

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Dietary Antioxidants Significantly Attenuate Hyperoxia-Induced Acute Inflammatory Lung Injury by Enhancing Macrophage Function via Reducing the Accumulation of Airway HMGB1

A Standardized Botanical Composition Mitigated Acute Inflammatory Lung Injury and Reduced Mortality through Extracellular HMGB1 Reduction

Correction to: GTS-21, an α7nAChR agonist, increases pulmonary bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

From nicotine to the cholinergic anti-inflammatory reflex - Can nicotine alleviate the dysregulated inflammation in COVID-19?

Retraction Note: The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Correction to: 2‑O, 3‑O desulfated heparin (ODSH) increases bacterial clearance and attenuates lung injury in cystic fibrosis by restoring HMGB1‑compromised macrophage function

Expression of Concern to: The α7 nicotine acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

The Role of HMGB1, a Nuclear Damage-Associated Molecular Pattern Molecule, in the Pathogenesis of Lung Diseases

Our Fragile Water Planet: An Introduction to the Earth Sciences

Carbon Nanomaterials Stimulate HMGB1 Release From Macrophages and Induce Cell Migration and Invasion

Dual effects of supplemental oxygen on pulmonary infection, inflammatory lung injury, and neuromodulation in aging and COVID-19

The nitric oxide donor, (Z)-1-[N-(2-aminoethyl)-N-(2-ammonioethyl)amino]diazen-1-ium-1,2-diolate (DETA-NONOate/D-NO), increases survival by attenuating hyperoxia-compromised innate immunity in bacterial clearance in a mouse model of ventilator-associated pneumonia

In memoriam Steven Yue Qian (1960–2019)

Nitric Oxide Regulates Gene Expression in Cancers by Controlling Histone Posttranslational Modifications

A32 MICROBIAL INTERACTIONS AND HOST DEFENSE: Host And Pathogen Factors Modulate Antibacterial Properties Of Histones And Non-Histone Protein Hmgb1

Hyperoxia Inhibits Oxidant-induced Apoptosis in Lung Epithelial Cells

Introduction to serial reviews on redox signaling in immune function and cellular responses in lung injury and diseases

Interleukin-11 and Interleukin-6 Protect Cultured Human Endothelial Cells from H2O2-Induced Cell Death

C73 OXIDANTS: Hyperoxia-Compromised Phagocytic Function In Macrophages Is Improved By Salicylates Via Inhibition Of Hmgb1 Release

Overexpression of Manganese Superoxide Dismutase Protects Lung Epithelial Cells against Oxidant Injury

Ascorbic Acid Attenuates Hyperoxia-Compromised Host Defense against Pulmonary Bacterial Infection

Nuclear Factor-κB Is Activated by Hyperoxia but Does Not Protect from Cell Death

A37 CYSTIC FIBROSIS: IMMUNE REGULATORY PATHWAYS: Cf Airway Epithelia Contribute To The Compromised Innate Immunity Against Bacterial Infection

A57 LUNG INJURY, REPAIR, AND FIBROSIS: THE PLOT THICKENS FOR THREE'S COMPANY: Mnsod Prevents Hyperoxia-Induced Cell Death Via Erk Activation

Unscheduled apoptosis during acute inflammatory lung injury

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