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The α-Ketoglutarate Dehydrogenase Complex as a Hub of Plasticity in Neurodegeneration and Regeneration

International journal of molecular sciences, 2022-10, Vol.23 (20), p.12403

2022

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Serum Metabolomic and Lipidomic Profiling Reveals Novel Biomarkers of Efficacy for Benfotiamine in Alzheimer's Disease

International journal of molecular sciences, 2021-12, Vol.22 (24), p.13188

2021

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Benfotiamine and Cognitive Decline in Alzheimer's Disease: Results of a Randomized Placebo-Controlled Phase IIa Clinical Trial

Journal of Alzheimer's disease, 2020-01, Vol.78 (3), p.989

2020

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Novel Metabolic Abnormalities in the Tricarboxylic Acid Cycle in Peripheral Cells From Huntington's Disease Patients

PloS one, 2016-09, Vol.11 (9), p.e0160384

2016

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Pharmacological thiamine levels as a therapeutic approach in Alzheimer's disease

Frontiers in medicine, 2022-10, Vol.9, p.1033272-1033272

2022

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Obituary: In Memory of Myron Flint Beal, MD, November 6, 1950–June 12, 2021

Movement disorders, 2021-09, Vol.36 (9), p.2003-2004

2021

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Thiamine and Oxidants Interact to Modify Cellular Calcium Stores

Neurochemical research, 2010-12, Vol.35 (12), p.2107-2116

2010

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Mitochondria/metabolic reprogramming in the formation of neurons from peripheral cells: Cause or consequence and the implications to their utility

Neurochemistry international, 2018-07, Vol.117, p.65-76

2018

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Selective NADH communication from α-ketoglutarate dehydrogenase to mitochondrial transhydrogenase prevents reactive oxygen species formation under reducing conditions in the heart

Basic research in cardiology, 2020-09, Vol.115 (5), p.53, Article 53

2020

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The human brain acetylome reveals that decreased acetylation of mitochondrial proteins associates with Alzheimer's disease

Journal of neurochemistry, 2021-07, Vol.158 (2), p.282-296

2021

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Mild metabolic perturbations alter succinylation of mitochondrial proteins

Journal of neuroscience research, 2017-11, Vol.95 (11), p.2244-2252

2017

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Reductions in the mitochondrial enzyme [alpha]-ketoglutarate dehydrogenase complex in neurodegenerative disease - beneficial or detrimental?

Journal of neurochemistry, 2016-12, Vol.139 (5), p.823

2016

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Selective linkage of mitochondrial enzymes to intracellular calcium stores differs between human‐induced pluripotent stem cells, neural stem cells, and neurons

Journal of neurochemistry, 2021-03, Vol.156 (6), p.867-879

2021

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Nutritional metabolism and cerebral bioenergetics in Alzheimer's disease and related dementias

Alzheimer's & dementia, 2023-03, Vol.19 (3), p.1041-1066

2023

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Inactivation and Reactivation of the Mitochondrial α-Ketoglutarate Dehydrogenase Complex

The Journal of biological chemistry, 2011-05, Vol.286 (20), p.17640-17648

2011

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Thiamine-dependent processes and treatment strategies in neurodegeneration

Antioxidants & redox signaling, 2007-10, Vol.9 (10), p.1605

2007

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Oxidative stress in Alzheimer's disease

Neurobiology of aging, 2005-05, Vol.26 (5), p.575-578

2005

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Oxidant-induced Changes in Mitochondria and Calcium Dynamics in the Pathophysiology of Alzheimer's Disease

Annals of the New York Academy of Sciences, 2008-12, Vol.1147 (1), p.221-232

2008

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Mild mitochondrial metabolic deficits by α-ketoglutarate dehydrogenase inhibition cause prominent changes in intracellular autophagic signaling: Potential role in the pathobiology of Alzheimer's disease

Neurochemistry international, 2016-06, Vol.96, p.32-45

2016

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Novel functions of the α-ketoglutarate dehydrogenase complex may mediate diverse oxidant-induced changes in mitochondrial enzymes associated with Alzheimer's disease

Biochimica et biophysica acta, 2008-04, Vol.1782 (4), p.229-238

2008

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Interactions of oxidative stress with cellular calcium dynamics and glucose metabolism in Alzheimer's disease

Free radical biology & medicine, 2002-06, Vol.32 (11), p.1061-1070

Gibson, Gary E

2002

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Mild reduction in the activity of the α‐ketoglutarate dehydrogenase complex elevates GABA shunt and glycolysis

Journal of neurochemistry, 2009-05, Vol.109 (s1), p.214-221

2009

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Cerebrometabolic aspects of delirium in relationship to dementia

Dementia and geriatric cognitive disorders, 1999-09, Vol.10 (5), p.335

1999

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Impaired mitochondrial energy metabolism as a novel risk factor for selective onset and progression of dementia in oldest-old subjects

Neuropsychiatric disease and treatment, 2015-01, Vol.11 (default), p.565-574

2015

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The α-Ketoglutarate Dehydrogenase Complex as a Hub of Plasticity in Neurodegeneration and Regeneration

Serum Metabolomic and Lipidomic Profiling Reveals Novel Biomarkers of Efficacy for Benfotiamine in Alzheimer's Disease

Benfotiamine and Cognitive Decline in Alzheimer's Disease: Results of a Randomized Placebo-Controlled Phase IIa Clinical Trial

Protocol for a seamless phase 2A-phase 2B randomized double-blind placebo-controlled trial to evaluate the safety and efficacy of benfotiamine in patients with early Alzheimer's disease (BenfoTeam)

Novel Metabolic Abnormalities in the Tricarboxylic Acid Cycle in Peripheral Cells From Huntington's Disease Patients

Pharmacological thiamine levels as a therapeutic approach in Alzheimer's disease

Obituary: In Memory of Myron Flint Beal, MD, November 6, 1950–June 12, 2021

Thiamine and Oxidants Interact to Modify Cellular Calcium Stores

Mitochondria/metabolic reprogramming in the formation of neurons from peripheral cells: Cause or consequence and the implications to their utility

Selective NADH communication from α-ketoglutarate dehydrogenase to mitochondrial transhydrogenase prevents reactive oxygen species formation under reducing conditions in the heart

The human brain acetylome reveals that decreased acetylation of mitochondrial proteins associates with Alzheimer's disease

Mild metabolic perturbations alter succinylation of mitochondrial proteins

Reductions in the mitochondrial enzyme [alpha]-ketoglutarate dehydrogenase complex in neurodegenerative disease - beneficial or detrimental?

Selective linkage of mitochondrial enzymes to intracellular calcium stores differs between human‐induced pluripotent stem cells, neural stem cells, and neurons

Nutritional metabolism and cerebral bioenergetics in Alzheimer's disease and related dementias

Inactivation and Reactivation of the Mitochondrial α-Ketoglutarate Dehydrogenase Complex

Thiamine-dependent processes and treatment strategies in neurodegeneration

Oxidative stress in Alzheimer's disease

Oxidant-induced Changes in Mitochondria and Calcium Dynamics in the Pathophysiology of Alzheimer's Disease

Mild mitochondrial metabolic deficits by α-ketoglutarate dehydrogenase inhibition cause prominent changes in intracellular autophagic signaling: Potential role in the pathobiology of Alzheimer's disease

Novel functions of the α-ketoglutarate dehydrogenase complex may mediate diverse oxidant-induced changes in mitochondrial enzymes associated with Alzheimer's disease

Interactions of oxidative stress with cellular calcium dynamics and glucose metabolism in Alzheimer's disease

Mild reduction in the activity of the α‐ketoglutarate dehydrogenase complex elevates GABA shunt and glycolysis

Cerebrometabolic aspects of delirium in relationship to dementia

Impaired mitochondrial energy metabolism as a novel risk factor for selective onset and progression of dementia in oldest-old subjects

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