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Gain-of-function mutation in Nav1.7 in familial erythromelalgia induces bursting of sensory neurons

Brain (London, England : 1878), 2005-08, Vol.128 (8), p.1847-1854

2005

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International Spinal Cord Injury Pain Classification: part I. Background and description

Spinal cord, 2012-06, Vol.50 (6), p.413-417

2012

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Nav1.7-related small fiber neuropathy: impaired slow-inactivation and DRG neuron hyperexcitability

Neurology, 2012-05, Vol.78 (21), p.1635

2012

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NaV1.7 Gain-of-Function Mutations as a Continuum: A1632E Displays Physiological Changes Associated with Erythromelalgia and Paroxysmal Extreme Pain Disorder Mutations and Produces Symptoms of Both Disorders

The Journal of neuroscience, 2008-10, Vol.28 (43), p.11079-11088

2008

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Ca2+ toxicity due to reverse Na+/Ca2+ exchange contributes to degeneration of neurites of DRG neurons induced by a neuropathy-associated Nav1.7 mutation

Journal of neurophysiology, 2015-09, Vol.114 (3), p.1554-1564

2015

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Genetic aspects of sodium channelopathy in small fiber neuropathy

Clinical genetics, 2012-10, Vol.82 (4), p.351-358

2012

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Persistent TTX-Resistant Na+ Current Affects Resting Potential and Response to Depolarization in Simulated Spinal Sensory Neurons

Journal of neurophysiology, 2001-09, Vol.86 (3), p.1351-1364

2001

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International Spinal Cord Injury Pain (ISCIP) Classification: Part 2. Initial validation using vignettes

Spinal cord, 2012-06, Vol.50 (6), p.404-412

2012

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NaN, a Novel Voltage-Gated Na Channel, is Expressed Preferentially in Peripheral Sensory Neurons and Down-Regulated after Axotomy

Proceedings of the National Academy of Sciences - PNAS, 1998-07, Vol.95 (15), p.8963-8968

1998

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Sodium Channels and Pain

Proceedings of the National Academy of Sciences - PNAS, 1999-07, Vol.96 (14), p.7635-7639

1999

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Rat NaV1.7 loss-of-function genetic model: Deficient nociceptive and neuropathic pain behavior with retained olfactory function and intra-epidermal nerve fibers

Molecular pain, 2019, Vol.15, p.1744806919881846-1744806919881846

2019

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GTP-induced tetrodotoxin-resistant Na+ current regulates excitability in mouse and rat small diameter sensory neurones

The Journal of physiology, 2003-04, Vol.548 (2), p.373-382

2003

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A Novel Persistent Tetrodotoxin-Resistant Sodium Current In SNS-Null And Wild-Type Small Primary Sensory Neurons

The Journal of neuroscience, 1999-12, Vol.19 (24), p.43-RC43

1999

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Nav1.9 expression in magnocellular neurosecretory cells of supraoptic nucleus

Experimental neurology, 2014-03, Vol.253, p.174-179

2014

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Distinct repriming and closed-state inactivation kinetics of Nav1.6 and Nav1.7 sodium channels in mouse spinal sensory neurons

The Journal of physiology, 2003-09, Vol.551 (3), p.741-750

2003

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Upregulation of a Silent Sodium Channel After Peripheral, but not Central, Nerve Injury in DRG Neurons

Journal of neurophysiology, 1999-11, Vol.82 (5), p.2776-2785

1999

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Intravenous administration of auto serum-expanded autologous mesenchymal stem cells in stroke

Brain (London, England : 1878), 2011-06, Vol.134 (Pt 6), p.1790-1807

2011

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Contribution of Na(v)1.8 sodium channels to action potential electrogenesis in DRG neurons

Journal of neurophysiology, 2001-08, Vol.86 (2), p.629

2001

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Down-Regulation of Transcripts for Na Channel α -SNS in Spinal Sensory Neurons Following Axotomy

Proceedings of the National Academy of Sciences - PNAS, 1996-12, Vol.93 (25), p.14950-14954

1996

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A gain-of-function mutation in the sodium channel gene Scn2a results in seizures and behavioral abnormalities

Neuroscience, 2001-01, Vol.102 (2), p.307-317

2001

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Altered Sodium Channel Expression in Second-Order Spinal Sensory Neurons Contributes to Pain after Peripheral Nerve Injury

The Journal of neuroscience, 2004-05, Vol.24 (20), p.4832-4839

2004

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Changes in Expression of Two Tetrodotoxin-Resistant Sodium Channels and Their Currents in Dorsal Root Ganglion Neurons after Sciatic Nerve Injury But Not Rhizotomy

The Journal of neuroscience, 2000-10, Vol.20 (19), p.7279-7289

2000

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Type III sodium channel mRNA is expressed in embryonic but not adult spinal sensory neurons, and is reexpressed following axotomy

Journal of neurophysiology, 1994-07, Vol.72 (1), p.466-470

1994

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Demyelinating diseases--new pathological insights, new therapeutic targets

The New England journal of medicine, 1998-01, Vol.338 (5), p.323-325

Waxman, S G

1998

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Nomenclature of voltage-gated sodium channels

Neuron (Cambridge, Mass.), 2000-11, Vol.28 (2), p.365-368

2000

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Gain-of-function mutation in Nav1.7 in familial erythromelalgia induces bursting of sensory neurons

International Spinal Cord Injury Pain Classification: part I. Background and description

Nav1.7-related small fiber neuropathy: impaired slow-inactivation and DRG neuron hyperexcitability

NaV1.7 Gain-of-Function Mutations as a Continuum: A1632E Displays Physiological Changes Associated with Erythromelalgia and Paroxysmal Extreme Pain Disorder Mutations and Produces Symptoms of Both Disorders

Ca2+ toxicity due to reverse Na+/Ca2+ exchange contributes to degeneration of neurites of DRG neurons induced by a neuropathy-associated Nav1.7 mutation

Genetic aspects of sodium channelopathy in small fiber neuropathy

Persistent TTX-Resistant Na+ Current Affects Resting Potential and Response to Depolarization in Simulated Spinal Sensory Neurons

International Spinal Cord Injury Pain (ISCIP) Classification: Part 2. Initial validation using vignettes

NaN, a Novel Voltage-Gated Na Channel, is Expressed Preferentially in Peripheral Sensory Neurons and Down-Regulated after Axotomy

Sodium Channels and Pain

Rat NaV1.7 loss-of-function genetic model: Deficient nociceptive and neuropathic pain behavior with retained olfactory function and intra-epidermal nerve fibers

GTP-induced tetrodotoxin-resistant Na+ current regulates excitability in mouse and rat small diameter sensory neurones

A Novel Persistent Tetrodotoxin-Resistant Sodium Current In SNS-Null And Wild-Type Small Primary Sensory Neurons

Nav1.9 expression in magnocellular neurosecretory cells of supraoptic nucleus

Distinct repriming and closed-state inactivation kinetics of Nav1.6 and Nav1.7 sodium channels in mouse spinal sensory neurons

Upregulation of a Silent Sodium Channel After Peripheral, but not Central, Nerve Injury in DRG Neurons

Intravenous administration of auto serum-expanded autologous mesenchymal stem cells in stroke

Contribution of Na(v)1.8 sodium channels to action potential electrogenesis in DRG neurons

Down-Regulation of Transcripts for Na Channel α -SNS in Spinal Sensory Neurons Following Axotomy

A gain-of-function mutation in the sodium channel gene Scn2a results in seizures and behavioral abnormalities

Altered Sodium Channel Expression in Second-Order Spinal Sensory Neurons Contributes to Pain after Peripheral Nerve Injury

Changes in Expression of Two Tetrodotoxin-Resistant Sodium Channels and Their Currents in Dorsal Root Ganglion Neurons after Sciatic Nerve Injury But Not Rhizotomy

Type III sodium channel mRNA is expressed in embryonic but not adult spinal sensory neurons, and is reexpressed following axotomy

Demyelinating diseases--new pathological insights, new therapeutic targets

Nomenclature of voltage-gated sodium channels

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