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Human extracellular microvesicles from renal tubules reverse kidney ischemia-reperfusion injury in rats

PloS one, 2018-08, Vol.13 (8), p.e0202550-e0202550

2018

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Impaired microvascular circulation in distant organs following renal ischemia

PloS one, 2023-06, Vol.18 (6), p.e0286543-e0286543

2023

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Improved Structure and Function in Autosomal Recessive Polycystic Rat Kidneys with Renal Tubular Cell Therapy

PloS one, 2015-07, Vol.10 (7), p.e0131677-e0131677

2015

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Renal Tubular Cell-Derived Extracellular Vesicles Accelerate the Recovery of Established Renal Ischemia Reperfusion Injury

Journal of the American Society of Nephrology, 2017-12, Vol.28 (12), p.3533-3544

2017

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Rapid Progression of Diabetic Nephropathy Is Linked to Inflammation and Episodes of Acute Renal Failure

American journal of nephrology, 2010-01, Vol.32 (5), p.469-475

2010

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Cardiac effects of renal ischemia

American journal of physiology. Renal physiology, 2023-01, Vol.324 (1), p.F64-F74

2023

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Renal, but not platelet or skin, extracellular vesicles decrease oxidative stress, enhance nascent peptide synthesis, and protect from ischemic renal injury

American journal of physiology. Renal physiology, 2023-08, Vol.325 (2), p.F164-F176

2023

Volltextzugriff (PDF)

Role of coagulation in persistent renal ischemia following reperfusion in an animal model

American journal of physiology. Renal physiology, 2022-11, Vol.323 (5), p.F590-F601

2022

Volltextzugriff (PDF)

LOX-1 and inflammation: a new mechanism for renal injury in obesity and diabetes

American journal of physiology. Renal physiology, 2008-05, Vol.294 (5), p.F1136-F1145

2008

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Microcirculation: nexus of comorbidities in diabetes

American journal of physiology. Renal physiology, 2007-08, Vol.293 (2), p.F486-F493

2007

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Renal C3 Complement Component: Feed Forward to Diabetic Kidney Disease

American journal of nephrology, 2015-01, Vol.41 (1), p.48-56

2015

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Intravenous renal cell transplantation with SAA1-positive cells prevents the progression of chronic renal failure in rats with ischemic-diabetic nephropathy

American journal of physiology. Renal physiology, 2013-12, Vol.305 (12), p.F1804-F1812

2013

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Comprehensive genomic profiling in diabetic nephropathy reveals the predominance of proinflammatory pathways

Physiological genomics, 2013-08, Vol.45 (16), p.710-719

2013

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Renal iron overload in rats with diabetic nephropathy

Physiological reports, 2015-12, Vol.3 (12), p.e12654-n/a

2015

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Postischemic inflammatory syndrome: a critical mechanism of progression in diabetic nephropathy

American journal of physiology. Renal physiology, 2009-10, Vol.297 (4), p.F923-F931

2009

Volltextzugriff (PDF)

Intravenous renal cell transplantation for rats with acute and chronic renal failure

American journal of physiology. Renal physiology, 2012-08, Vol.303 (3), p.F357-F365

2012

Volltextzugriff (PDF)

Intravenous cell therapy for acute renal failure with serum amyloid A protein-reprogrammed cells

American journal of physiology. Renal physiology, 2010-08, Vol.299 (2), p.F453-F464

2010

Volltextzugriff (PDF)

Treatment of the post-ischaemic inflammatory syndrome of diabetic nephropathy

Nephrology, dialysis, transplantation, 2010-10, Vol.25 (10), p.3204-3212

2010

Volltextzugriff (PDF)

Acute-phase response protein serum amyloid A stimulates renal tubule formation: studies in vitro and in vivo

American journal of physiology. Renal physiology, 2009-06, Vol.296 (6), p.F1355-F1363

2009

Volltextzugriff (PDF)

Mechanisms of Protection From Ischemic Injury With Renal Exosomes: FR-PO118

Journal of the American Society of Nephrology, 2022-11, Vol.33 (11S), p.359-359

2022

Volltextzugriff (PDF)

Distant Organ Effects of Renal Injury: TH-PO099

Journal of the American Society of Nephrology, 2022-11, Vol.33 (11S), p.77-77

2022

Volltextzugriff (PDF)

Studies of renal injury IV: The GLUT1 gene protects renal cells from cyclosporine A toxicity

Kidney international, 2002-07, Vol.62 (1), p.127-136

2002

Volltextzugriff (PDF)

Multiple Beneficial Effects of Renal Exosomes on Ischemic Injury: PO0142

Journal of the American Society of Nephrology, 2020-10, Vol.31 (10S), p.100-100

2020

Volltextzugriff (PDF)

Anti-LOX-1 therapy in rats with diabetes and dyslipidemia: ablation of renal vascular and epithelial manifestations

American journal of physiology. Renal physiology, 2008-01, Vol.294 (1), p.F110-F119

2008

Volltextzugriff (PDF)

Studies of renal injury III: Lipid-induced nephropathy in type II diabetes

Kidney international, 2000-01, Vol.57 (1), p.92-104

2000

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Human extracellular microvesicles from renal tubules reverse kidney ischemia-reperfusion injury in rats

Impaired microvascular circulation in distant organs following renal ischemia

Improved Structure and Function in Autosomal Recessive Polycystic Rat Kidneys with Renal Tubular Cell Therapy

Renal Tubular Cell-Derived Extracellular Vesicles Accelerate the Recovery of Established Renal Ischemia Reperfusion Injury

Rapid Progression of Diabetic Nephropathy Is Linked to Inflammation and Episodes of Acute Renal Failure

Cardiac effects of renal ischemia

Renal, but not platelet or skin, extracellular vesicles decrease oxidative stress, enhance nascent peptide synthesis, and protect from ischemic renal injury

Role of coagulation in persistent renal ischemia following reperfusion in an animal model

LOX-1 and inflammation: a new mechanism for renal injury in obesity and diabetes

Microcirculation: nexus of comorbidities in diabetes

Renal C3 Complement Component: Feed Forward to Diabetic Kidney Disease

Intravenous renal cell transplantation with SAA1-positive cells prevents the progression of chronic renal failure in rats with ischemic-diabetic nephropathy

Comprehensive genomic profiling in diabetic nephropathy reveals the predominance of proinflammatory pathways

Renal iron overload in rats with diabetic nephropathy

Postischemic inflammatory syndrome: a critical mechanism of progression in diabetic nephropathy

Intravenous renal cell transplantation for rats with acute and chronic renal failure

Intravenous cell therapy for acute renal failure with serum amyloid A protein-reprogrammed cells

Treatment of the post-ischaemic inflammatory syndrome of diabetic nephropathy

Acute-phase response protein serum amyloid A stimulates renal tubule formation: studies in vitro and in vivo

Mechanisms of Protection From Ischemic Injury With Renal Exosomes: FR-PO118

Distant Organ Effects of Renal Injury: TH-PO099

Studies of renal injury IV: The GLUT1 gene protects renal cells from cyclosporine A toxicity

Multiple Beneficial Effects of Renal Exosomes on Ischemic Injury: PO0142

Anti-LOX-1 therapy in rats with diabetes and dyslipidemia: ablation of renal vascular and epithelial manifestations

Studies of renal injury III: Lipid-induced nephropathy in type II diabetes

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