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Animal models of Diamond-Blackfan anemia: updates and challenges

Haematologica (Roma), 2023-05, Vol.108 (5), p.1222

2023

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Noncanonical TGF-beta pathways, mTORC1 and Abl, in renal interstitial fibrogenesis

American Journal of Physiology - Renal Physiology, 2010, Vol.298 (1), p.F142-F149

2010

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Activation of nemo-like kinase in diamond blackfan anemia suppresses early erythropoiesis by preventing mitochondrial biogenesis

The Journal of biological chemistry, 2024-08, Vol.300 (8), p.107542, Article 107542

2024

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Beyond mRNA: The role of non-coding RNAs in normal and aberrant hematopoiesis

Molecular genetics and metabolism, 2017-11, Vol.122 (3), p.28-38

2017

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Downregulation of SATB1 by miRNAs reduces megakaryocyte/erythroid progenitor expansion in preclinical models of Diamond–Blackfan anemia

Experimental hematology, 2022-07, Vol.111, p.66-78

2022

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Signaling Pathways That Regulate Normal and Aberrant Red Blood Cell Development

Genes, 2021-10, Vol.12 (10), p.1646

2021

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Imatinib mesylate inhibits the profibrogenic activity of TGF-beta and prevents bleomycin-mediated lung fibrosis

The Journal of clinical investigation, 2004-11, Vol.114 (9), p.1308-1316

2004

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SATB1 Chromatin Loops Regulate Megakaryocyte/Erythroid Progenitor Expansion by Facilitating HSP70 and GATA1 Induction

Stem cells (Dayton, Ohio), 2023-06, Vol.41 (6), p.560-569

2023

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The active component of ginseng, ginsenoside Rb1, improves erythropoiesis in models of Diamond–Blackfan anemia by targeting Nemo-like kinase

The Journal of biological chemistry, 2021-09, Vol.297 (3), p.100988-100988, Article 100988

2021

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Imatinib mesylate blocks a non‐Smad TGF‐β pathway and reduces renal fibrogenesis in vivo

The FASEB journal, 2005-01, Vol.19 (1), p.1-11

2005

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Innate immune system activation in zebrafish and cellular models of Diamond Blackfan Anemia

Scientific reports, 2018-03, Vol.8 (1), p.5165-11, Article 5165

2018

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Metformin-induced suppression of Nemo-like kinase improves erythropoiesis in preclinical models of Diamond–Blackfan anemia through induction of miR-26a

Experimental hematology, 2020-11, Vol.91, p.65-77

2020

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Retromer maintains basolateral distribution of the type II TGF-β receptor via the recycling endosome

Molecular biology of the cell, 2013-07, Vol.24 (14), p.2285-2298

2013

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Sorting nexin 9 differentiates ligand-activated Smad3 from Smad2 for nuclear import and transforming growth factor β signaling

Molecular biology of the cell, 2015-11, Vol.26 (21), p.3879-3891

2015

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Non-Smad Transforming Growth Factor-β Signaling Regulated by Focal Adhesion Kinase Binding the p85 Subunit of Phosphatidylinositol 3-Kinase

The Journal of biological chemistry, 2011-05, Vol.286 (20), p.17841-17850

2011

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Profibrotic TGFβ responses require the cooperative action of PDGF and ErbB receptor tyrosine kinases

The FASEB journal, 2013-11, Vol.27 (11), p.4444-4454

2013

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Transforming Growth Factor β Activation of c-Abl Is Independent of Receptor Internalization and Regulated by Phosphatidylinositol 3-Kinase and PAK2 in Mesenchymal Cultures

The Journal of biological chemistry, 2006-09, Vol.281 (38), p.27846-27854

2006

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Cell-Type-Specific Activation of PAK2 by Transforming Growth Factor β Independent of Smad2 and Smad3

Molecular and Cellular Biology, 2003-12, Vol.23 (23), p.8878-8889

2003

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Transforming growth factor-β activation of phosphatidylinositol 3-kinase is independent of Smad2 and Smad3 and regulates fibroblast responses via p21-activated kinase-2

Cancer research (Chicago, Ill.), 2005-11, Vol.65 (22), p.10431-10440

2005

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Distinct Roles for Mammalian Target of Rapamycin Complexes in the Fibroblast Response to Transforming Growth Factor-β

Cancer research (Chicago, Ill.), 2009, Vol.69 (1), p.84-93

2009

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Type II transforming growth factor-beta receptor recycling is dependent upon the clathrin adaptor protein Dab2

Molecular biology of the cell, 2010-11, Vol.21 (22), p.4009-4019

2010

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MMP9 inhibition increases erythropoiesis in RPS14-deficient del(5q) MDS models through suppression of TGF-β pathways

Blood advances, 2019-09, Vol.3 (18), p.2751-2763

2019

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Transforming growth factor β signaling via ras in mesenchymal cells requires p21-activated kinase 2 for extracellular signal-regulated kinase-dependent transcriptional responses

Cancer research (Chicago, Ill.), 2007-04, Vol.67 (8), p.3673-3682

2007

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Erbin and the NF2 Tumor Suppressor Merlin Cooperatively Regulate Cell-Type-Specific Activation of PAK2 by TGF-β

Developmental cell, 2009-03, Vol.16 (3), p.433-444

2009

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ERBB Receptor Activation Is Required for Profibrotic Responses to Transforming Growth Factor β

Cancer research (Chicago, Ill.), 2010-10, Vol.70 (19), p.7421-7430

2010

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Animal models of Diamond-Blackfan anemia: updates and challenges

Noncanonical TGF-beta pathways, mTORC1 and Abl, in renal interstitial fibrogenesis

Activation of nemo-like kinase in diamond blackfan anemia suppresses early erythropoiesis by preventing mitochondrial biogenesis

Beyond mRNA: The role of non-coding RNAs in normal and aberrant hematopoiesis

Downregulation of SATB1 by miRNAs reduces megakaryocyte/erythroid progenitor expansion in preclinical models of Diamond–Blackfan anemia

Signaling Pathways That Regulate Normal and Aberrant Red Blood Cell Development

Imatinib mesylate inhibits the profibrogenic activity of TGF-beta and prevents bleomycin-mediated lung fibrosis

SATB1 Chromatin Loops Regulate Megakaryocyte/Erythroid Progenitor Expansion by Facilitating HSP70 and GATA1 Induction

The active component of ginseng, ginsenoside Rb1, improves erythropoiesis in models of Diamond–Blackfan anemia by targeting Nemo-like kinase

Imatinib mesylate blocks a non‐Smad TGF‐β pathway and reduces renal fibrogenesis in vivo

Innate immune system activation in zebrafish and cellular models of Diamond Blackfan Anemia

Metformin-induced suppression of Nemo-like kinase improves erythropoiesis in preclinical models of Diamond–Blackfan anemia through induction of miR-26a

Retromer maintains basolateral distribution of the type II TGF-β receptor via the recycling endosome

Sorting nexin 9 differentiates ligand-activated Smad3 from Smad2 for nuclear import and transforming growth factor β signaling

Non-Smad Transforming Growth Factor-β Signaling Regulated by Focal Adhesion Kinase Binding the p85 Subunit of Phosphatidylinositol 3-Kinase

Profibrotic TGFβ responses require the cooperative action of PDGF and ErbB receptor tyrosine kinases

Transforming Growth Factor β Activation of c-Abl Is Independent of Receptor Internalization and Regulated by Phosphatidylinositol 3-Kinase and PAK2 in Mesenchymal Cultures

Cell-Type-Specific Activation of PAK2 by Transforming Growth Factor β Independent of Smad2 and Smad3

Transforming growth factor-β activation of phosphatidylinositol 3-kinase is independent of Smad2 and Smad3 and regulates fibroblast responses via p21-activated kinase-2

Distinct Roles for Mammalian Target of Rapamycin Complexes in the Fibroblast Response to Transforming Growth Factor-β

Type II transforming growth factor-beta receptor recycling is dependent upon the clathrin adaptor protein Dab2

MMP9 inhibition increases erythropoiesis in RPS14-deficient del(5q) MDS models through suppression of TGF-β pathways

Transforming growth factor β signaling via ras in mesenchymal cells requires p21-activated kinase 2 for extracellular signal-regulated kinase-dependent transcriptional responses

Erbin and the NF2 Tumor Suppressor Merlin Cooperatively Regulate Cell-Type-Specific Activation of PAK2 by TGF-β

ERBB Receptor Activation Is Required for Profibrotic Responses to Transforming Growth Factor β

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