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Inflammasome activation causes dual recruitment of NLRC4 and NLRP3 to the same macromolecular complex

Proceedings of the National Academy of Sciences - PNAS, 2014-05, Vol.111 (20), p.7403-7408

2014

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CARD9 negatively regulates NLRP3-induced IL-1β production on Salmonella infection of macrophages

Nature communications, 2016-09, Vol.7 (1), p.12874-12874, Article 12874

2016

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Insights into the molecular basis of the NOD2 signalling pathway

Open biology, 2014-12, Vol.4 (12), p.140178

2014

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The Innate Immune System: A Compositional and Functional Perspective

Monie, Tom

1, 2017

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Activating immunity: lessons from the TLRs and NLRs

Trends in biochemical sciences (Amsterdam. Regular ed.), 2009-11, Vol.34 (11), p.553-561

2009

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Mice, men and the relatives: cross-species studies underpin innate immunity

Open biology, 2012-04, Vol.2 (4), p.120015-120015

2012

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A dimer of the Toll-like receptor 4 cytoplasmic domain provides a specific scaffold for the recruitment of signalling adaptor proteins

PloS one, 2007-08, Vol.2 (8), p.e788

2007

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The N-terminal region of the human autophagy protein ATG16L1 contains a domain that folds into a helical structure consistent with formation of a coiled-coil

PloS one, 2013-09, Vol.8 (9), p.e76237-e76237

2013

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Cell Swelling and the NLRP3 Inflammasome

Immunity (Cambridge, Mass.), 2013-03, Vol.38 (3), p.399-399

2013

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Salmonella infection induces recruitment of Caspase-8 to the inflammasome to modulate IL-1β production

The Journal of immunology (1950), 2013-11, Vol.191 (10), p.5239-5246

2013

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NLR activation takes a direct route

Trends in biochemical sciences (Amsterdam. Regular ed.), 2013-03, Vol.38 (3), p.131-139

Monie, Tom P.

2013

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THE CONCISE GUIDE TO PHARMACOLOGY 2019/20: Catalytic receptors

British journal of pharmacology, 2019-12, Vol.176 (S1), p.S247-S296

2019

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Caspase-8 functions as a key mediator of inflammation and pro-IL-1β processing via both canonical and non-canonical pathways

Immunological reviews, 2015-05, Vol.265 (1), p.181-193

2015

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The Concise Guide to PHARMACOLOGY 2023/24: Catalytic receptors

British journal of pharmacology, 2023-10, Vol.180 (S2), p.S241-S288

2023

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Pathogen Sensing by Nucleotide-binding Oligomerization Domain-containing Protein 2 (NOD2) Is Mediated by Direct Binding to Muramyl Dipeptide and ATP

The Journal of biological chemistry, 2012-06, Vol.287 (27), p.23057-23067

2012

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Dynamic phosphorylation of RelA on Ser42 and Ser45 in response to TNFα stimulation regulates DNA binding and transcription

Open biology, 2016-07, Vol.6 (7), p.160055

2016

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Blau syndrome polymorphisms in NOD2 identify nucleotide hydrolysis and helical domain 1 as signalling regulators

FEBS letters, 2014-09, Vol.588 (18), p.3382-3389

2014

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Ultrasmall silica nanoparticles directly ligate the T cell receptor complex

Proceedings of the National Academy of Sciences - PNAS, 2020-01, Vol.117 (1), p.285-291

2020

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Structure and RNA Interactions of the N-Terminal RRM Domains of PTB

Structure (London), 2004-09, Vol.12 (9), p.1631-1643

2004

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International Union of Basic and Clinical Pharmacology. XCVI. Pattern recognition receptors in health and disease

Pharmacological reviews, 2015-04, Vol.67 (2), p.462-504

2015

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Interaction between NOD2 and CARD9 involves the NOD2 NACHT and the linker region between the NOD2 CARDs and NACHT domain

FEBS letters, 2014-08, Vol.588 (17), p.2830-2836

2014

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Engagement of Nucleotide-binding Oligomerization Domain-containing Protein 1 (NOD1) by Receptor-interacting Protein 2 (RIP2) Is Insufficient for Signal Transduction

The Journal of biological chemistry, 2014-08, Vol.289 (33), p.22900-22914

2014

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Intestinal APCs of the endogenous nanomineral pathway fail to express PD-L1 in Crohn's disease

Scientific reports, 2016-05, Vol.6 (1), p.26747-26747, Article 26747

2016

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Dysfunctional Crohn's Disease-Associated NOD2 Polymorphisms Cannot be Reliably Predicted on the Basis of RIPK2 Binding or Membrane Association

Frontiers in immunology, 2015-10, Vol.6, p.521-521

2015

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Allergens and Activation of the Toll-Like Receptor Response

Methods in molecular biology (Clifton, N.J.), 2016, Vol.1390, p.341

2016

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Inflammasome activation causes dual recruitment of NLRC4 and NLRP3 to the same macromolecular complex

CARD9 negatively regulates NLRP3-induced IL-1β production on Salmonella infection of macrophages

Insights into the molecular basis of the NOD2 signalling pathway

The Innate Immune System: A Compositional and Functional Perspective

Activating immunity: lessons from the TLRs and NLRs

Mice, men and the relatives: cross-species studies underpin innate immunity

A dimer of the Toll-like receptor 4 cytoplasmic domain provides a specific scaffold for the recruitment of signalling adaptor proteins

The N-terminal region of the human autophagy protein ATG16L1 contains a domain that folds into a helical structure consistent with formation of a coiled-coil

Cell Swelling and the NLRP3 Inflammasome

Salmonella infection induces recruitment of Caspase-8 to the inflammasome to modulate IL-1β production

NLR activation takes a direct route

THE CONCISE GUIDE TO PHARMACOLOGY 2019/20: Catalytic receptors

Caspase-8 functions as a key mediator of inflammation and pro-IL-1β processing via both canonical and non-canonical pathways

The Concise Guide to PHARMACOLOGY 2023/24: Catalytic receptors

Pathogen Sensing by Nucleotide-binding Oligomerization Domain-containing Protein 2 (NOD2) Is Mediated by Direct Binding to Muramyl Dipeptide and ATP

Dynamic phosphorylation of RelA on Ser42 and Ser45 in response to TNFα stimulation regulates DNA binding and transcription

Blau syndrome polymorphisms in NOD2 identify nucleotide hydrolysis and helical domain 1 as signalling regulators

Ultrasmall silica nanoparticles directly ligate the T cell receptor complex

Structure and RNA Interactions of the N-Terminal RRM Domains of PTB

International Union of Basic and Clinical Pharmacology. XCVI. Pattern recognition receptors in health and disease

Interaction between NOD2 and CARD9 involves the NOD2 NACHT and the linker region between the NOD2 CARDs and NACHT domain

Engagement of Nucleotide-binding Oligomerization Domain-containing Protein 1 (NOD1) by Receptor-interacting Protein 2 (RIP2) Is Insufficient for Signal Transduction

Intestinal APCs of the endogenous nanomineral pathway fail to express PD-L1 in Crohn's disease

Dysfunctional Crohn's Disease-Associated NOD2 Polymorphisms Cannot be Reliably Predicted on the Basis of RIPK2 Binding or Membrane Association

Allergens and Activation of the Toll-Like Receptor Response

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