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Acquired and genetic complement abnormalities play a critical role in dense deposit disease and other C3 glomerulopathies

Kidney international, 2012-08, Vol.82 (4), p.454-464

2012

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Renal microRNA- and RNA-profiles in progressive chronic kidney disease

European journal of clinical investigation, 2016-03, Vol.46 (3), p.213-226

2016

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Novel Biomarkers in Glomerular Disease

Advances in chronic kidney disease, 2014-03, Vol.21 (2), p.205-216

2014

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Primary glomerulonephritis with isolated C3 deposits: a new entity which shares common genetic risk factors with haemolytic uraemic syndrome

Journal of medical genetics, 2007-03, Vol.44 (3), p.193-199

2007

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RNA‐seq profiling of tubulointerstitial tissue reveals a potential therapeutic role of dual anti‐phosphatase 1 in glomerulonephritis

Journal of cellular and molecular medicine, 2022-06, Vol.26 (12), p.3364-3377

2022

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Identification of plasma miR-106a-5p and miR-30a-5p as potential biomarkers for mesangial proliferative glomerulonephritis

Clinical biochemistry, 2020-10, Vol.84, p.79-86

2020

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Immunohistological score of transcription factor 21 had a positive correlation with its urinary excretion and proteinuria in immunoglobulin A nephropathy

Histology and histopathology, 2021-10, Vol.36 (10), p.1093-1098

2021

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Treatment of C3 glomerulopathy with complement blockers

Seminars in thrombosis and hemostasis, 2014-06, Vol.40 (4), p.472

2014

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Molecular pathomechanisms of membranous nephropathy: from Heymann nephritis to alloimmunization

Journal of the American Society of Nephrology, 2005-05, Vol.16 (5), p.1205-1213

2005

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Familial C3 glomerulonephritis associated with mutations in the gene for complement factor B

Nephrology, dialysis, transplantation, 2015-05, Vol.30 (5), p.862-864

2015

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C3 glomerulopathy: what's in a name?

Kidney international, 2012-08, Vol.82 (4), p.379-381

2012

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The in situ expression of interleukin-8 in the normal human kidney and in different morphological forms of glomerulonephritis

American journal of kidney diseases, 2004-06, Vol.43 (6), p.983-998

2004

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The family of 14‐3‐3 proteins and specifically 14‐3‐3σ are up‐regulated during the development of renal pathologies

Journal of cellular and molecular medicine, 2018-09, Vol.22 (9), p.4139-4149

2018

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Circulating miR-103a-3p contributes to angiotensin II-induced renal inflammation and fibrosis via a SNRK/NF-κB/p65 regulatory axis

Nature communications, 2019-05, Vol.10 (1), p.2145-2145, Article 2145

2019

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C3 Glomerulonephritis Associated With Monoclonal Gammopathy: A Case Series

American journal of kidney diseases, 2013-09, Vol.62 (3), p.506-514

2013

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Short-chain fatty acids, GPR41 and GPR43 ligands, inhibit TNF-α-induced MCP-1 expression by modulating p38 and JNK signaling pathways in human renal cortical epithelial cells

Biochemical and biophysical research communications, 2017-04, Vol.486 (2), p.499-505

2017

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Cytokines: Names and Numbers You Should Care About

Clinical journal of the American Society of Nephrology, 2015-12, Vol.10 (12), p.2243-2254

2015

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C3 Glomerulopathy: Ten Years' Experience at Mayo Clinic

Mayo Clinic proceedings, 2018-08, Vol.93 (8), p.991-1008

2018

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Redefining C3 glomerulopathy: ‘C3 only’ is a bridge too far

Kidney international, 2013-02, Vol.83 (2), p.331-332

2013

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Transcription factor Nrf2 activation regulates NETosis, endothelial injury, and kidney disease in myeloperoxidase-positive antineutrophil cytoplasmic antibody-associated vasculitis

Kidney international, 2024-06, Vol.105 (6), p.1291-1305

2024

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L-FABP: A novel biomarker of kidney disease

Clinica chimica acta, 2015-05, Vol.445, p.85-90

2015

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Recurrent Glomerular Disease after Kidney Transplantation: Diagnostic and Management Dilemmas

Clinical journal of the American Society of Nephrology, 2021-11, Vol.16 (11), p.1730-1742

2021

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Early changes in gene expression that influence the course of primary glomerular disease

Kidney international, 2007-08, Vol.72 (3), p.337-347

2007

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immunodominant myeloperoxidase T-cell epitope induces local cell-mediated injury in antimyeloperoxidase glomerulonephritis

Proceedings of the National Academy of Sciences - PNAS, 2012-09, Vol.109 (39), p.E2615-E2624

2012

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Acquired and genetic complement abnormalities play a critical role in dense deposit disease and other C3 glomerulopathies

Renal microRNA- and RNA-profiles in progressive chronic kidney disease

Novel Biomarkers in Glomerular Disease

Primary glomerulonephritis with isolated C3 deposits: a new entity which shares common genetic risk factors with haemolytic uraemic syndrome

RNA‐seq profiling of tubulointerstitial tissue reveals a potential therapeutic role of dual anti‐phosphatase 1 in glomerulonephritis

Identification of plasma miR-106a-5p and miR-30a-5p as potential biomarkers for mesangial proliferative glomerulonephritis

Immunohistological score of transcription factor 21 had a positive correlation with its urinary excretion and proteinuria in immunoglobulin A nephropathy

Treatment of C3 glomerulopathy with complement blockers

Molecular pathomechanisms of membranous nephropathy: from Heymann nephritis to alloimmunization

Familial C3 glomerulonephritis associated with mutations in the gene for complement factor B

C3 glomerulopathy: what's in a name?

The in situ expression of interleukin-8 in the normal human kidney and in different morphological forms of glomerulonephritis

The family of 14‐3‐3 proteins and specifically 14‐3‐3σ are up‐regulated during the development of renal pathologies

Identification of a mutation in complement factor H-related protein 5 in patients of Cypriot origin with glomerulonephritis

Circulating miR-103a-3p contributes to angiotensin II-induced renal inflammation and fibrosis via a SNRK/NF-κB/p65 regulatory axis

C3 Glomerulonephritis Associated With Monoclonal Gammopathy: A Case Series

Short-chain fatty acids, GPR41 and GPR43 ligands, inhibit TNF-α-induced MCP-1 expression by modulating p38 and JNK signaling pathways in human renal cortical epithelial cells

Cytokines: Names and Numbers You Should Care About

C3 Glomerulopathy: Ten Years' Experience at Mayo Clinic

Redefining C3 glomerulopathy: ‘C3 only’ is a bridge too far

Transcription factor Nrf2 activation regulates NETosis, endothelial injury, and kidney disease in myeloperoxidase-positive antineutrophil cytoplasmic antibody-associated vasculitis

L-FABP: A novel biomarker of kidney disease

Recurrent Glomerular Disease after Kidney Transplantation: Diagnostic and Management Dilemmas

Early changes in gene expression that influence the course of primary glomerular disease

immunodominant myeloperoxidase T-cell epitope induces local cell-mediated injury in antimyeloperoxidase glomerulonephritis

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