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CT vs. bioluminescence: A comparison of imaging techniques for orthotopic prostate tumors in mice

PloS one, 2022-11, Vol.17 (11), p.e0277239-e0277239

2022

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MnTE-2-PyP Treatment, or NOX4 Inhibition, Protects against Radiation-Induced Damage in Mouse Primary Prostate Fibroblasts by Inhibiting the TGF-Beta 1 Signaling Pathway

Radiation research, 2017-03, Vol.187 (3), p.367

2017

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Upregulation of Nox4 induces a pro-survival Nrf2 response in cancer-associated fibroblasts that promotes tumorigenesis and metastasis, in part via Birc5 induction

Breast cancer research : BCR, 2022-07, Vol.24 (1), p.48-19, Article 48

2022

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MnTE-2-PyP reduces prostate cancer growth and metastasis by suppressing p300 activity and p300/HIF-1/CREB binding to the promoter region of the PAI-1 gene

Free radical biology & medicine, 2016-05, Vol.94, p.185-194

2016

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MnTE-2-PyP modulates thiol oxidation in a hydrogen peroxide-mediated manner in a human prostate cancer cell

Free radical biology & medicine, 2016-12, Vol.101, p.32-43

2016

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MnTE-2-PyP, a manganese porphyrin, reduces cytotoxicity caused by irradiation in a diabetic environment through the induction of endogenous antioxidant defenses

Redox biology, 2020-07, Vol.34, p.101542-101542, Article 101542

2020

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MnTE-2-PyP protects fibroblast mitochondria from hyperglycemia and radiation exposure

Redox biology, 2022-06, Vol.52, p.102301-102301, Article 102301

2022

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Exogenous APN protects normal tissues from radiation-induced oxidative damage and fibrosis in mice and prostate cancer patients with higher levels of APN have less radiation-induced toxicities

Redox biology, 2024-07, Vol.73, p.103219, Article 103219

2024

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The Addition of Manganese Porphyrins during Radiation Inhibits Prostate Cancer Growth and Simultaneously Protects Normal Prostate Tissue from Radiation Damage

Antioxidants, 2018-01, Vol.7 (1), p.21

2018

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Nanoformulation of the superoxide dismutase mimic, MnTnBuOE-2-PyP5+, prevents its acute hypotensive response

Redox biology, 2020-09, Vol.36, p.101610-101610, Article 101610

2020

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Reactive Oxygen Species Drive Epigenetic Changes in Radiation-Induced Fibrosis

Oxidative medicine and cellular longevity, 2019-01, Vol.2019 (2019), p.1-27

2019

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The SOD Mimic, MnTE-2-PyP, Protects from Chronic Fibrosis and Inflammation in Irradiated Normal Pelvic Tissues

Antioxidants, 2017-11, Vol.6 (4), p.87

2017

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Adipocytes protect fibroblasts from radiation-induced damage by adiponectin secretion

Scientific reports, 2020-07, Vol.10 (1), p.12616-12616, Article 12616

2020

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MnTE-2-PyP Suppresses Prostate Cancer Cell Growth via H2O2 Production

Antioxidants, 2020-06, Vol.9 (6), p.490

2020

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Sympathoinhibition and vasodilation contribute to the acute hypotensive response of the superoxide dismutase mimic, MnTnBuOE-2-PyP5+, in hypertensive animals

Advances in redox research : an official journal of the Society for Redox Biology and Medicine and the Society for Free Radical Research-Europe, 2021-12, Vol.3, p.100016, Article 100016

2021

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Manganese porphyrin, MnTE-2-PyP, treatment protects the prostate from radiation-induced fibrosis (RIF) by activating the NRF2 signaling pathway and enhancing SOD2 and sirtuin activity

Free radical biology & medicine, 2020-05, Vol.152, p.255-270

2020

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Dynamics of antioxidant heme oxygenase-1 and pro-oxidant p66Shc in promoting advanced prostate cancer progression

Free radical biology & medicine, 2022-11, Vol.193 (Pt 1), p.274-291

2022

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GM-CSF induces neuroprotective and anti-inflammatory responses in 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine intoxicated mice

Journal of neuroimmunology, 2013-12, Vol.265 (1), p.1-10

2013

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Lack of p53 function promotes radiation-induced mitotic catastrophe in mouse embryonic fibroblast cells

Cancer cell international, 2006-04, Vol.6 (1), p.11-11

2006

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p66Shc protein through a redox mechanism enhances the progression of prostate cancer cells towards castration-resistance

Free radical biology & medicine, 2019-08, Vol.139, p.24-34

2019

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Activation of Meiosis-Specific Genes Is Associated with Depolyploidization of Human Tumor Cells following Radiation-Induced Mitotic Catastrophe

Cancer research (Chicago, Ill.), 2009-03, Vol.69 (6), p.2296-2304

2009

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MnTnBuOE-2-PyP treatment protects from radioactive iodine (I-131) treatment-related side effects in thyroid cancer

Radiation and environmental biophysics, 2020-03, Vol.59 (1), p.99-109

2020

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Manganese Porphyrin and Radiotherapy Improves Local Tumor Response and Overall Survival in Orthotopic Murine Mammary Carcinoma Models

Radiation research, 2021-02, Vol.195 (2), p.128

2021

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MnTnBuOE-2-PyP protects normal colorectal fibroblasts from radiation damage and simultaneously enhances radio/chemotherapeutic killing of colorectal cancer cells

Oncotarget, 2016-06, Vol.7 (23), p.34532-34545

2016

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Polyploid tumour cells elicit paradiploid progeny through depolyploidizing divisions and regulated autophagic degradation

Cell biology international, 2011-07, Vol.35 (7), p.687-695

2011

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CT vs. bioluminescence: A comparison of imaging techniques for orthotopic prostate tumors in mice

MnTE-2-PyP Treatment, or NOX4 Inhibition, Protects against Radiation-Induced Damage in Mouse Primary Prostate Fibroblasts by Inhibiting the TGF-Beta 1 Signaling Pathway

Upregulation of Nox4 induces a pro-survival Nrf2 response in cancer-associated fibroblasts that promotes tumorigenesis and metastasis, in part via Birc5 induction

MnTE-2-PyP reduces prostate cancer growth and metastasis by suppressing p300 activity and p300/HIF-1/CREB binding to the promoter region of the PAI-1 gene

MnTE-2-PyP modulates thiol oxidation in a hydrogen peroxide-mediated manner in a human prostate cancer cell

MnTE-2-PyP, a manganese porphyrin, reduces cytotoxicity caused by irradiation in a diabetic environment through the induction of endogenous antioxidant defenses

MnTE-2-PyP protects fibroblast mitochondria from hyperglycemia and radiation exposure

Exogenous APN protects normal tissues from radiation-induced oxidative damage and fibrosis in mice and prostate cancer patients with higher levels of APN have less radiation-induced toxicities

The Addition of Manganese Porphyrins during Radiation Inhibits Prostate Cancer Growth and Simultaneously Protects Normal Prostate Tissue from Radiation Damage

Nanoformulation of the superoxide dismutase mimic, MnTnBuOE-2-PyP5+, prevents its acute hypotensive response

Reactive Oxygen Species Drive Epigenetic Changes in Radiation-Induced Fibrosis

The SOD Mimic, MnTE-2-PyP, Protects from Chronic Fibrosis and Inflammation in Irradiated Normal Pelvic Tissues

Adipocytes protect fibroblasts from radiation-induced damage by adiponectin secretion

MnTE-2-PyP Suppresses Prostate Cancer Cell Growth via H2O2 Production

Sympathoinhibition and vasodilation contribute to the acute hypotensive response of the superoxide dismutase mimic, MnTnBuOE-2-PyP5+, in hypertensive animals

Manganese porphyrin, MnTE-2-PyP, treatment protects the prostate from radiation-induced fibrosis (RIF) by activating the NRF2 signaling pathway and enhancing SOD2 and sirtuin activity

Dynamics of antioxidant heme oxygenase-1 and pro-oxidant p66Shc in promoting advanced prostate cancer progression

GM-CSF induces neuroprotective and anti-inflammatory responses in 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine intoxicated mice

Lack of p53 function promotes radiation-induced mitotic catastrophe in mouse embryonic fibroblast cells

p66Shc protein through a redox mechanism enhances the progression of prostate cancer cells towards castration-resistance

Activation of Meiosis-Specific Genes Is Associated with Depolyploidization of Human Tumor Cells following Radiation-Induced Mitotic Catastrophe

MnTnBuOE-2-PyP treatment protects from radioactive iodine (I-131) treatment-related side effects in thyroid cancer

Manganese Porphyrin and Radiotherapy Improves Local Tumor Response and Overall Survival in Orthotopic Murine Mammary Carcinoma Models

MnTnBuOE-2-PyP protects normal colorectal fibroblasts from radiation damage and simultaneously enhances radio/chemotherapeutic killing of colorectal cancer cells

Polyploid tumour cells elicit paradiploid progeny through depolyploidizing divisions and regulated autophagic degradation

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