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Fibrosarcoma-induced increase in macrophage tumor necrosis factor alpha synthesis suppresses T cell responses

Journal of leukocyte biology, 1993-08, Vol.54 (2), p.152-160

1993

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Tumor growth increases Ia- macrophage synthesis of tumor necrosis factor-alpha and prostaglandin E2: changes in macrophage suppressor activity

Journal of leukocyte biology, 1993-05, Vol.53 (5), p.550-558

1993

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Tumour Growth Causes Suppression of Autoreactive T‐Cell Proliferation by Disrupting Macrophage Responsiveness to Interferon‐γ

Scandinavian journal of immunology, 1994-01, Vol.39 (1), p.31-38

1994

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Interleukin-12 Overcomes Paclitaxel-Mediated Suppression of T-Cell Proliferation

Immunopharmacology and immunotoxicology, 1998-01, Vol.20 (4), p.473-492

1998

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Cytokines and suppressor macrophages cause tumor-bearing host CD8+ T cells to suppress recognition of allogeneic and syngeneic MHC class II molecules

Journal of leukocyte biology, 1992-12, Vol.52 (6), p.661-669

1992

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Tumor Growth Alters T Cell and Macrophage Production of and Responsiveness to Granulocyte-Macrophage Colony-Stimulating Factor: Partial Dysregulation through Interleukin-10

Cellular immunology, 1994-04, Vol.154 (2), p.342-357

1994

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Prostaglandin E2 production by Mac-2+ macrophages: tumor-induced population shift

Journal of leukocyte biology, 1987-12, Vol.42 (6), p.673-681

1987

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Tumor cell-derived TGF-β and IL-10 dysregulate paclitaxel-induced macrophage activation

Journal of leukocyte biology, 2001-01, Vol.69 (1), p.129-137

2001

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Changes in splenic macrophage Mac antigen expression during tumor growth: a kinetic study of accessory cell function and antigen-defined phenotypes

Journal of leukocyte biology, 1986-12, Vol.40 (6), p.709-724

1986

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Tumor growth and adherence change the expression of macrophage Mac-2

Cancer letters, 1993-04, Vol.69 (1), p.67-74

1993

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Tumor-Induced Macrophage Tumor Necrosis Factor-α Production Suppresses Autoreactive T Cell Proliferation

Immunobiology (1979), 1993-08, Vol.188 (4), p.430-445

1993

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Tumor modulation of autoreactivity: Decreased macrophage and autoreactive T cell interactions

Cellular immunology, 1990-04, Vol.127 (1), p.105-119

1990

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Tumor-induced alteration in macrophage accessory cell activity on autoreactive T cells

Cancer Immunology Immunotherapy, 1989-05, Vol.30 (3), p.170-176

1989

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Is celiac disease due to molecular mimicry between gliadin peptide-HLA class II molecule-T cell interactions and those of some unidentified superantigen?

Molecular immunology, 1997-05, Vol.34 (7), p.535-541

1997

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Shifts in macrophage (Mφ) surface phenotypes during tumor growth: Association of Mac-2 + and Mac-3 + Mφ with immunosuppressive activity

Cellular immunology, 1987-09, Vol.108 (2), p.255-268

1987

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Tumor Growth Alters Macrophage Responsiveness to Macrophage Colony-Stimulating Factor During Reactivity Against Allogeneic and Syngeneic Mhc Class II Molecules

Immunological investigations, 1993-01, Vol.22 (6-7), p.463-476

1993

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IA− Macrophages and Cytokine Networks Contribute to Tumor-Induced Suppression of CD4+ Autoreactive T Cells

Immunological investigations, 1993-01, Vol.22 (3), p.169-187

1993

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Macrophages stimulated by receptor-ligand interactions exhibit differences in cell-cycle kinetics during tumor growth: stimulation at Mac-1 and Mac-3 receptors alters DNA synthesis

Immunology letters, 1992-02, Vol.31 (3), p.217-225

1992

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Tumor growth changes responsiveness to and production of granulocyte-macrophage colony-stimulating factor during recognition of self MHC class II molecules

Oncology research, 1992, Vol.4 (11-12), p.455

1992

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Interleukin 2 (IL-2) activity during tumor growth: IL-2 production kinetics, absorption of and responses to exogenous IL-2

Cellular immunology, 1984-04, Vol.84 (2), p.228-239

1984

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Tumor-Induced Variations in a High Molecular Weight Inhibitory Monokine

Immunobiology (1979), 1989-02, Vol.178 (4), p.361-379

1989

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Normal and Tumor-Bearing Host Splenic Macrophage Responses to Lipopolysaccharide

Immunological investigations, 1990-01, Vol.19 (1), p.41-55

1990

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Tumor-induced modulation of macrophage class II MHC molecule mRNA expression

Molecular immunology, 1993-07, Vol.30 (10), p.911-920

1993

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Macrophage-derived prostaglandin E modulation of the mixed-lymphocyte reaction: An anomaly of increased production and decreased T-cell susceptibility during tumor growth

Cellular immunology, 1984-03, Vol.84 (1), p.1-13

1984

Volltextzugriff (PDF)

Promotion of Macrophage-Stimulated Autoreactive T Cell Proliferation by Interleukin-10: Counteraction of Macrophage Suppressor Activity During Tumor Growth

Immunobiology (1979), 1995-02, Vol.192 (3), p.155-171

1995

Volltextzugriff (PDF)

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Fibrosarcoma-induced increase in macrophage tumor necrosis factor alpha synthesis suppresses T cell responses

Tumor growth increases Ia- macrophage synthesis of tumor necrosis factor-alpha and prostaglandin E2: changes in macrophage suppressor activity

Tumour Growth Causes Suppression of Autoreactive T‐Cell Proliferation by Disrupting Macrophage Responsiveness to Interferon‐γ

Interleukin-12 Overcomes Paclitaxel-Mediated Suppression of T-Cell Proliferation

Cytokines and suppressor macrophages cause tumor-bearing host CD8+ T cells to suppress recognition of allogeneic and syngeneic MHC class II molecules

Tumor Growth Alters T Cell and Macrophage Production of and Responsiveness to Granulocyte-Macrophage Colony-Stimulating Factor: Partial Dysregulation through Interleukin-10

Prostaglandin E2 production by Mac-2+ macrophages: tumor-induced population shift

Tumor cell-derived TGF-β and IL-10 dysregulate paclitaxel-induced macrophage activation

Changes in splenic macrophage Mac antigen expression during tumor growth: a kinetic study of accessory cell function and antigen-defined phenotypes

Tumor growth and adherence change the expression of macrophage Mac-2

Tumor-Induced Macrophage Tumor Necrosis Factor-α Production Suppresses Autoreactive T Cell Proliferation

Tumor modulation of autoreactivity: Decreased macrophage and autoreactive T cell interactions

Tumor-induced alteration in macrophage accessory cell activity on autoreactive T cells

Is celiac disease due to molecular mimicry between gliadin peptide-HLA class II molecule-T cell interactions and those of some unidentified superantigen?

Shifts in macrophage (Mφ) surface phenotypes during tumor growth: Association of Mac-2 + and Mac-3 + Mφ with immunosuppressive activity

Tumor Growth Alters Macrophage Responsiveness to Macrophage Colony-Stimulating Factor During Reactivity Against Allogeneic and Syngeneic Mhc Class II Molecules

IA− Macrophages and Cytokine Networks Contribute to Tumor-Induced Suppression of CD4+ Autoreactive T Cells

Macrophages stimulated by receptor-ligand interactions exhibit differences in cell-cycle kinetics during tumor growth: stimulation at Mac-1 and Mac-3 receptors alters DNA synthesis

Tumor growth changes responsiveness to and production of granulocyte-macrophage colony-stimulating factor during recognition of self MHC class II molecules

Interleukin 2 (IL-2) activity during tumor growth: IL-2 production kinetics, absorption of and responses to exogenous IL-2

Tumor-Induced Variations in a High Molecular Weight Inhibitory Monokine

Normal and Tumor-Bearing Host Splenic Macrophage Responses to Lipopolysaccharide

Tumor-induced modulation of macrophage class II MHC molecule mRNA expression

Macrophage-derived prostaglandin E modulation of the mixed-lymphocyte reaction: An anomaly of increased production and decreased T-cell susceptibility during tumor growth

Promotion of Macrophage-Stimulated Autoreactive T Cell Proliferation by Interleukin-10: Counteraction of Macrophage Suppressor Activity During Tumor Growth

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