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PH motifs in PAR1&2 endow breast cancer growth

Nature communications, 2015-11, Vol.6 (1), p.8853-8853, Article 8853

2015

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PH-domains as central modulators driving tumor growth

Cell cycle (Georgetown, Tex.), 2016-03, Vol.15 (5), p.615-616

2016

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Protease-activated receptors (PARs) in cancer: Novel biased signaling and targets for therapy

Methods in cell biology, 2016, Vol.132, p.341-358

2016

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p53 controls hPar1 function and expression

Oncogene, 2008-11, Vol.27 (54), p.6866-6874

2008

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Heparanase promotes growth, angiogenesis and survival of primary breast tumors

International journal of cancer, 2006-04, Vol.118 (7), p.1609-1617

2006

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Binding of thrombin to subendothelial extracellular matrix: protection and expression of functional properties

The Journal of clinical investigation, 1989-10, Vol.84 (4), p.1096-1104

1989

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Identification of a Thrombin Sequence with Growth Factor Activity on Macrophages

Proceedings of the National Academy of Sciences - PNAS, 1986-02, Vol.83 (4), p.976-980

1986

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Oncogenic transformation induces tumor angiogenesis: a role for PAR1 activation

The FASEB journal, 2003-02, Vol.17 (2), p.163-174

2003

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Chemotactic Response of Monocytes to Thrombin

The Journal of cell biology, 1983-01, Vol.96 (1), p.282-285

1983

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Sequestration and Release of Basic Fibroblast Growth Factor

Annals of the New York Academy of Sciences, 1991-01, Vol.638 (1), p.207-220

1991

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Thrombin as a multifunctional protein: induction of cell adhesion and proliferation

American journal of respiratory cell and molecular biology, 1992-02, Vol.6 (2), p.123-130

1992

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Human protease-activated receptor 1 expression in malignant epithelia: a role in invasiveness

Arteriosclerosis, thrombosis, and vascular biology, 2003-06, Vol.23 (6), p.940-944

2003

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Human alpha-defensin regulates smooth muscle cell contraction: a role for low-density lipoprotein receptor-related protein/alpha 2-macroglobulin receptor

Blood, 2002-12, Vol.100 (12), p.4026

2002

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Identification of a novel functional androgen response element within hPar1 promoter: implications to prostate cancer progression

The FASEB journal, 2005, Vol.19 (1), p.62-72

2005

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Control of cell proliferation by heparan sulfate and heparin-binding growth factors

Thrombosis and haemostasis, 1995-07, Vol.74 (1), p.534-540

1995

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Thrombin chemotactic stimulation of HL-60 cells: Studies on thrombin responsiveness as a function of differentiation

Journal of cellular physiology, 1987-05, Vol.131 (2), p.255-261

1987

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Signalling pathways induced by protease‐activated receptors and integrins in T cells

Immunology, 2002-01, Vol.105 (1), p.35-46

2002

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Inhibition of acetylcholine receptor synthesis by thyroid hormones

Journal of endocrinology, 1984-05, Vol.101 (2), p.141-147

1984

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Chapter 16 - Protease-activated receptors (PARs) in cancer: Novel biased signaling and targets for therapy

Methods in cell biology, 2016-01, Vol.132, p.341-358

2016

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Extracellular sequestration and release of fibroblast growth factor: a regulatory mechanism?

Trends in biochemical sciences (Amsterdam. Regular ed.), 1991, Vol.16 (7), p.268-271

1991

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Gonadotropin Stimulation of MLS Human Epithelial Ovarian Carcinoma Cells Augments Cell Adhesion Mediated by CD44 and by αv-Integrin

Gynecologic oncology, 2002-02, Vol.84 (2), p.296-302

2002

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Prothrombin Conversion to Thrombin by Plasminogen Activator Residing in the Subendothelial Extracellular Matrix

Seminars in thrombosis and hemostasis, 1993-01, Vol.19 (4), p.405-411

1993

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Thrombin-Induced Release of Active Basic Fibroblast Growth Factor-Heparan Sulfate Complexes From Subendothelial Extracellular Matrix

Blood, 1993-06, Vol.81 (12), p.3324-3331

1993

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Protection of Thrombin Receptor Expression under Hypoxia

The Journal of biological chemistry, 2000-01, Vol.275 (4), p.2281-2287

2000

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Growth-promoting effects of esterolytically inactive thrombin on macrophages

Journal of cellular biochemistry, 1986, Vol.32 (4), p.261-272

1986

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PH motifs in PAR1&2 endow breast cancer growth

PH-domains as central modulators driving tumor growth

Protease-activated receptors (PARs) in cancer: Novel biased signaling and targets for therapy

p53 controls hPar1 function and expression

Heparanase promotes growth, angiogenesis and survival of primary breast tumors

Binding of thrombin to subendothelial extracellular matrix: protection and expression of functional properties

Identification of a Thrombin Sequence with Growth Factor Activity on Macrophages

Oncogenic transformation induces tumor angiogenesis: a role for PAR1 activation

Chemotactic Response of Monocytes to Thrombin

Sequestration and Release of Basic Fibroblast Growth Factor

Thrombin as a multifunctional protein: induction of cell adhesion and proliferation

Human protease-activated receptor 1 expression in malignant epithelia: a role in invasiveness

Human alpha-defensin regulates smooth muscle cell contraction: a role for low-density lipoprotein receptor-related protein/alpha 2-macroglobulin receptor

Identification of a novel functional androgen response element within hPar1 promoter: implications to prostate cancer progression

Control of cell proliferation by heparan sulfate and heparin-binding growth factors

Thrombin chemotactic stimulation of HL-60 cells: Studies on thrombin responsiveness as a function of differentiation

Signalling pathways induced by protease‐activated receptors and integrins in T cells

Inhibition of acetylcholine receptor synthesis by thyroid hormones

Chapter 16 - Protease-activated receptors (PARs) in cancer: Novel biased signaling and targets for therapy

Extracellular sequestration and release of fibroblast growth factor: a regulatory mechanism?

Gonadotropin Stimulation of MLS Human Epithelial Ovarian Carcinoma Cells Augments Cell Adhesion Mediated by CD44 and by αv-Integrin

Prothrombin Conversion to Thrombin by Plasminogen Activator Residing in the Subendothelial Extracellular Matrix

Thrombin-Induced Release of Active Basic Fibroblast Growth Factor-Heparan Sulfate Complexes From Subendothelial Extracellular Matrix

Protection of Thrombin Receptor Expression under Hypoxia

Growth-promoting effects of esterolytically inactive thrombin on macrophages

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