UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Key Characteristics of Carcinogens as a Basis for Organizing Data on Mechanisms of Carcinogenesis

Environmental health perspectives, 2016-06, Vol.124 (6), p.713-721

2016

Volltextzugriff (PDF)

Enhanced Radiation Sensitivity in HPV-Positive Head and Neck Cancer

Cancer research (Chicago, Ill.), 2013-08, Vol.73 (15), p.4791-4800

2013

Volltextzugriff (PDF)

The Hippo/YAP pathway interacts with EGFR signaling and HPV oncoproteins to regulate cervical cancer progression

EMBO molecular medicine, 2015-11, Vol.7 (11), p.1426-1449

2015

Volltextzugriff (PDF)

Stress keratin 17 enhances papillomavirus infection-induced disease by downregulating T cell recruitment

PLoS pathogens, 2020-01, Vol.16 (1), p.e1008206-e1008206

2020

Volltextzugriff (PDF)

Merkel cell polyomavirus large T antigen binding to pRb promotes skin hyperplasia and tumor development

PLoS pathogens, 2022-05, Vol.18 (5), p.e1010551

2022

Volltextzugriff (PDF)

MmuPV1-Induced Cutaneous Squamous Cell Carcinoma Arises Preferentially from Lgr5+ Epithelial Progenitor Cells

Viruses, 2022-08, Vol.14 (8), p.1751

2022

Volltextzugriff (PDF)

Mouse papillomavirus type 1 (MmuPV1) DNA is frequently integrated in benign tumors by microhomology-mediated end-joining

PLoS pathogens, 2021-08, Vol.17 (8), p.e1009812-e1009812

2021

Volltextzugriff (PDF)

Dysregulation of Autophagy Contributes to Anal Carcinogenesis

PloS one, 2016-10, Vol.11 (10), p.e0164273

2016

Volltextzugriff (PDF)

Differentiation-Dependent KLF4 Expression Promotes Lytic Epstein-Barr Virus Infection in Epithelial Cells

PLoS pathogens, 2015-10, Vol.11 (10), p.e1005195-e1005195

2015

Volltextzugriff (PDF)

Prevention and treatment of cervical cancer in mice using estrogen receptor antagonists

Proceedings of the National Academy of Sciences - PNAS, 2009-11, Vol.106 (46), p.19467-19472

2009

Volltextzugriff (PDF)

Current In Vitro and In Vivo Models to Study MCPyV-Associated MCC

Viruses, 2022-10, Vol.14 (10), p.2204

2022

Volltextzugriff (PDF)

Fundamental differences in cell cycle deregulation in human papillomavirus-positive and human papillomavirus-negative head/neck and cervical cancers

Cancer research (Chicago, Ill.), 2007-05, Vol.67 (10), p.4605-4619

2007

Volltextzugriff (PDF)

Estrogen Contributes to the Onset, Persistence, and Malignant Progression of Cervical Cancer in a Human Papillomavirus-Transgenic Mouse Model

Proceedings of the National Academy of Sciences - PNAS, 2005-02, Vol.102 (7), p.2490-2495

2005

Volltextzugriff (PDF)

Role of Rb-Dependent and Rb-Independent Functions of Papillomavirus E7 Oncogene in Head and Neck Cancer

Cancer research (Chicago, Ill.), 2007-12, Vol.67 (24), p.11585-11593

2007

Volltextzugriff (PDF)

The human papillomavirus E6 oncogene dysregulates the cell cycle and contributes to cervical carcinogenesis through two independent activities

Cancer research (Chicago, Ill.), 2007-02, Vol.67 (4), p.1626-1635

2007

Volltextzugriff (PDF)

ΔNp63α promotes Epstein-Barr virus latency in undifferentiated epithelial cells

PLoS pathogens, 2021-11, Vol.17 (11), p.e1010045-e1010045

2021

Volltextzugriff (PDF)

Type 1 and Type 2 Epstein-Barr viruses induce proliferation, and inhibit differentiation, in infected telomerase-immortalized normal oral keratinocytes

PLoS pathogens, 2022-10, Vol.18 (10), p.e1010868

2022

Volltextzugriff (PDF)

Tumourigenesis driven by the human papillomavirus type 16 Asian-American e6 variant in a three-dimensional keratinocyte model

PloS one, 2014-07, Vol.9 (7), p.e101540

2014

Volltextzugriff (PDF)

Deficiencies in the Fanconi Anemia DNA Damage Response Pathway Increase Sensitivity to HPV-Associated Head and Neck Cancer

Cancer research (Chicago, Ill.), 2010-12, Vol.70 (23), p.9959-9968

2010

Volltextzugriff (PDF)

Inactivating All Three Rb Family Pocket Proteins Is Insufficient to Initiate Cervical Cancer

Cancer research (Chicago, Ill.), 2012-10, Vol.72 (20), p.5418-5427

2012

Volltextzugriff (PDF)

Development and Characterization of HPV-Positive and HPV-Negative Head and Neck Squamous Cell Carcinoma Tumorgrafts

Clinical cancer research, 2013-02, Vol.19 (4), p.855-864

2013

Volltextzugriff (PDF)

Identification of Biomarkers That Distinguish Human Papillomavirus (HPV)-Positive versus HPV-Negative Head and Neck Cancers in a Mouse Model

Proceedings of the National Academy of Sciences - PNAS, 2006-09, Vol.103 (38), p.14152-14157

2006

Volltextzugriff (PDF)

Sonoporation delivery of monoclonal antibodies against human papillomavirus 16 E6 restores p53 expression in transformed cervical keratinocytes

PloS one, 2012-11, Vol.7 (11), p.e50730

2012

Volltextzugriff (PDF)

High incidence of HPV-associated head and neck cancers in FA deficient mice is associated with E7's induction of DNA damage through its inactivation of pocket proteins

PloS one, 2013-09, Vol.8 (9), p.e75056-e75056

2013

Volltextzugriff (PDF)

Cervical Cancers Require the Continuous Expression of the Human Papillomavirus Type 16 E7 Oncoprotein Even in the Presence of the Viral E6 Oncoprotein

Cancer research (Chicago, Ill.), 2012-08, Vol.72 (16), p.4008-4016

2012

Volltextzugriff (PDF)

1 – 25 von 69

Suche als RSS-Feed

Aktive Filter

SpracheDeutsch

ThemaTumors

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(69)
Mit Volltextzugriff
(69)

Publikationsform

Artikel
(69)

Sprache

Englisch
(69)

Erscheinungsjahr

n.n

Thema

Science & Technology
(62)
Life Sciences & Biomedicine
(52)
Humans
(48)
Animals
(40)
Mice
(39)
Oncology
(38)
Female
(31)
Biological And Medical Sciences
(30)
Medical Sciences
(30)
Mice, Transgenic
(26)
Cancer
(25)
Human Papillomavirus
(24)
Pharmacology. Drug Treatments
(19)
Proteins
(19)
Antineoplastic Agents
(18)
Gene Expression
(17)
Human Papillomavirus 16
(16)
Oncogene Proteins, Viral - Genetics
(15)
Rodents
(14)

14 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

Key Characteristics of Carcinogens as a Basis for Organizing Data on Mechanisms of Carcinogenesis

Enhanced Radiation Sensitivity in HPV-Positive Head and Neck Cancer

The Hippo/YAP pathway interacts with EGFR signaling and HPV oncoproteins to regulate cervical cancer progression

Stress keratin 17 enhances papillomavirus infection-induced disease by downregulating T cell recruitment

Merkel cell polyomavirus large T antigen binding to pRb promotes skin hyperplasia and tumor development

MmuPV1-Induced Cutaneous Squamous Cell Carcinoma Arises Preferentially from Lgr5+ Epithelial Progenitor Cells

Mouse papillomavirus type 1 (MmuPV1) DNA is frequently integrated in benign tumors by microhomology-mediated end-joining

Dysregulation of Autophagy Contributes to Anal Carcinogenesis

Differentiation-Dependent KLF4 Expression Promotes Lytic Epstein-Barr Virus Infection in Epithelial Cells

Prevention and treatment of cervical cancer in mice using estrogen receptor antagonists

Current In Vitro and In Vivo Models to Study MCPyV-Associated MCC

Fundamental differences in cell cycle deregulation in human papillomavirus-positive and human papillomavirus-negative head/neck and cervical cancers

Estrogen Contributes to the Onset, Persistence, and Malignant Progression of Cervical Cancer in a Human Papillomavirus-Transgenic Mouse Model

Role of Rb-Dependent and Rb-Independent Functions of Papillomavirus E7 Oncogene in Head and Neck Cancer

The human papillomavirus E6 oncogene dysregulates the cell cycle and contributes to cervical carcinogenesis through two independent activities

ΔNp63α promotes Epstein-Barr virus latency in undifferentiated epithelial cells

Type 1 and Type 2 Epstein-Barr viruses induce proliferation, and inhibit differentiation, in infected telomerase-immortalized normal oral keratinocytes

Tumourigenesis driven by the human papillomavirus type 16 Asian-American e6 variant in a three-dimensional keratinocyte model

Deficiencies in the Fanconi Anemia DNA Damage Response Pathway Increase Sensitivity to HPV-Associated Head and Neck Cancer

Inactivating All Three Rb Family Pocket Proteins Is Insufficient to Initiate Cervical Cancer

Development and Characterization of HPV-Positive and HPV-Negative Head and Neck Squamous Cell Carcinoma Tumorgrafts

Identification of Biomarkers That Distinguish Human Papillomavirus (HPV)-Positive versus HPV-Negative Head and Neck Cancers in a Mouse Model

Sonoporation delivery of monoclonal antibodies against human papillomavirus 16 E6 restores p53 expression in transformed cervical keratinocytes

High incidence of HPV-associated head and neck cancers in FA deficient mice is associated with E7's induction of DNA damage through its inactivation of pocket proteins

Cervical Cancers Require the Continuous Expression of the Human Papillomavirus Type 16 E7 Oncoprotein Even in the Presence of the Viral E6 Oncoprotein

Aktive Filter

Suchergebnisse filtern