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Enhanced Radiation Sensitivity in HPV-Positive Head and Neck Cancer

Cancer research (Chicago, Ill.), 2013-08, Vol.73 (15), p.4791-4800

2013

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Fundamental differences in cell cycle deregulation in human papillomavirus-positive and human papillomavirus-negative head/neck and cervical cancers

Cancer research (Chicago, Ill.), 2007-05, Vol.67 (10), p.4605-4619

2007

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Role of Rb-Dependent and Rb-Independent Functions of Papillomavirus E7 Oncogene in Head and Neck Cancer

Cancer research (Chicago, Ill.), 2007-12, Vol.67 (24), p.11585-11593

2007

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The human papillomavirus E6 oncogene dysregulates the cell cycle and contributes to cervical carcinogenesis through two independent activities

Cancer research (Chicago, Ill.), 2007-02, Vol.67 (4), p.1626-1635

2007

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Deficiencies in the Fanconi Anemia DNA Damage Response Pathway Increase Sensitivity to HPV-Associated Head and Neck Cancer

Cancer research (Chicago, Ill.), 2010-12, Vol.70 (23), p.9959-9968

2010

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Inactivating All Three Rb Family Pocket Proteins Is Insufficient to Initiate Cervical Cancer

Cancer research (Chicago, Ill.), 2012-10, Vol.72 (20), p.5418-5427

2012

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Development and Characterization of HPV-Positive and HPV-Negative Head and Neck Squamous Cell Carcinoma Tumorgrafts

Clinical cancer research, 2013-02, Vol.19 (4), p.855-864

2013

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Cervical Cancers Require the Continuous Expression of the Human Papillomavirus Type 16 E7 Oncoprotein Even in the Presence of the Viral E6 Oncoprotein

Cancer research (Chicago, Ill.), 2012-08, Vol.72 (16), p.4008-4016

2012

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The Human Papillomavirus Type 16 E7 Oncoprotein Activates the Fanconi Anemia (FA) Pathway and Causes Accelerated Chromosomal Instability in FA Cells

Journal of Virology, 2007-12, Vol.81 (23), p.13265-13270

2007

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Pocket Proteins Suppress Head and Neck Cancer

Cancer research (Chicago, Ill.), 2012-03, Vol.72 (5), p.1280-1289

2012

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The Fanconi Anemia Pathway Limits Human Papillomavirus Replication

Journal of Virology, 2012-08, Vol.86 (15), p.8131-8138

2012

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The Minor Capsid Protein L2 Contributes to Two Steps in the Human Papillomavirus Type 31 Life Cycle

Journal of Virology, 2005-04, Vol.79 (7), p.3938-3948

2005

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Two distinct activities contribute to human papillomavirus 16 E6's oncogenic potential

Cancer research (Chicago, Ill.), 2005-09, Vol.65 (18), p.8266-8273

2005

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Interactions with Pocket Proteins Contribute to the Role of Human Papillomavirus Type 16 E7 in the Papillomavirus Life Cycle

Journal of Virology, 2005-12, Vol.79 (23), p.14769-14780

2005

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Persistence of High-Grade Cervical Dysplasia and Cervical Cancer Requires the Continuous Expression of the Human Papillomavirus Type 16 E7 Oncogene

Cancer research (Chicago, Ill.), 2009-05, Vol.69 (10), p.4407-4414

2009

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Examination of the pRb-Dependent and pRb-Independent Functions of E7 In Vivo

Journal of Virology, 2005-09, Vol.79 (17), p.11392-11402

2005

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A Role for HPV16 E5 in Cervical Carcinogenesis

Cancer research (Chicago, Ill.), 2010-04, Vol.70 (7), p.2924-2931

2010

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E6-Associated Protein Is Required for Human Papillomavirus Type 16 E6 to Cause Cervical Cancer in Mice

Cancer research (Chicago, Ill.), 2010-06, Vol.70 (12), p.5064-5073

2010

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Phosphorylation of p53 at serine 37 is important for transcriptional activity and regulation in response to DNA damage

Oncogene, 2004-01, Vol.23 (1), p.49-57

2004

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Dissection of human papillomavirus E6 and E7 function in transgenic mouse models of cervical carcinogenesis

Cancer research (Chicago, Ill.), 2003-08, Vol.63 (16), p.4862-4871

2003

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Critical roles for non-pRb targets of human papillomavirus type 16 E7 in cervical carcinogenesis

Cancer research (Chicago, Ill.), 2006-10, Vol.66 (19), p.9393-9400

2006

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IFN-κ, a novel type I IFN, is undetectable in HPV-positive human cervical keratinocytes

Laboratory investigation, 2010-10, Vol.90 (10), p.1482-1491

2010

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Requirement of epidermal growth factor receptor for hyperplasia induced by E5, a high-risk human papillomavirus oncogene

Cancer research (Chicago, Ill.), 2005-08, Vol.65 (15), p.6534-6542

2005

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Identification of cis-Acting Elements That Mediate the Replication and Maintenance of Human Papillomavirus Type 16 Genomes in Saccharomyces cerevisiae

Journal of Virology, 2005-05, Vol.79 (10), p.5933-5942

2005

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Structural and functional consequences of glutamine tract variation in the androgen receptor

Human molecular genetics, 2004-08, Vol.13 (16), p.1677-1692

2004

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Enhanced Radiation Sensitivity in HPV-Positive Head and Neck Cancer

Fundamental differences in cell cycle deregulation in human papillomavirus-positive and human papillomavirus-negative head/neck and cervical cancers

Role of Rb-Dependent and Rb-Independent Functions of Papillomavirus E7 Oncogene in Head and Neck Cancer

The human papillomavirus E6 oncogene dysregulates the cell cycle and contributes to cervical carcinogenesis through two independent activities

Deficiencies in the Fanconi Anemia DNA Damage Response Pathway Increase Sensitivity to HPV-Associated Head and Neck Cancer

Inactivating All Three Rb Family Pocket Proteins Is Insufficient to Initiate Cervical Cancer

Development and Characterization of HPV-Positive and HPV-Negative Head and Neck Squamous Cell Carcinoma Tumorgrafts

Cervical Cancers Require the Continuous Expression of the Human Papillomavirus Type 16 E7 Oncoprotein Even in the Presence of the Viral E6 Oncoprotein

The Human Papillomavirus Type 16 E7 Oncoprotein Activates the Fanconi Anemia (FA) Pathway and Causes Accelerated Chromosomal Instability in FA Cells

Pocket Proteins Suppress Head and Neck Cancer

The Fanconi Anemia Pathway Limits Human Papillomavirus Replication

The Minor Capsid Protein L2 Contributes to Two Steps in the Human Papillomavirus Type 31 Life Cycle

Two distinct activities contribute to human papillomavirus 16 E6's oncogenic potential

Interactions with Pocket Proteins Contribute to the Role of Human Papillomavirus Type 16 E7 in the Papillomavirus Life Cycle

Persistence of High-Grade Cervical Dysplasia and Cervical Cancer Requires the Continuous Expression of the Human Papillomavirus Type 16 E7 Oncogene

Examination of the pRb-Dependent and pRb-Independent Functions of E7 In Vivo

A Role for HPV16 E5 in Cervical Carcinogenesis

E6-Associated Protein Is Required for Human Papillomavirus Type 16 E6 to Cause Cervical Cancer in Mice

Phosphorylation of p53 at serine 37 is important for transcriptional activity and regulation in response to DNA damage

Dissection of human papillomavirus E6 and E7 function in transgenic mouse models of cervical carcinogenesis

Critical roles for non-pRb targets of human papillomavirus type 16 E7 in cervical carcinogenesis

IFN-κ, a novel type I IFN, is undetectable in HPV-positive human cervical keratinocytes

Requirement of epidermal growth factor receptor for hyperplasia induced by E5, a high-risk human papillomavirus oncogene

Identification of cis-Acting Elements That Mediate the Replication and Maintenance of Human Papillomavirus Type 16 Genomes in Saccharomyces cerevisiae

Structural and functional consequences of glutamine tract variation in the androgen receptor

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