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Stress-Induced EGFR Trafficking: Mechanisms, Functions, and Therapeutic Implications

Trends in cell biology, 2016-05, Vol.26 (5), p.352-366

2016

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Key Characteristics of Carcinogens as a Basis for Organizing Data on Mechanisms of Carcinogenesis

Environmental health perspectives, 2016-06, Vol.124 (6), p.713-721

2016

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Establishment of human papillomavirus infection requires cell cycle progression

PLoS pathogens, 2009-02, Vol.5 (2), p.e1000318-e1000318

2009

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APOBEC3A functions as a restriction factor of human papillomavirus

Journal of virology, 2015-01, Vol.89 (1), p.688-702

2015

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Identifying the Target Cells and Mechanisms of Merkel Cell Polyomavirus Infection

Cell host & microbe, 2016-06, Vol.19 (6), p.775-787

2016

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Rodent Papillomaviruses

Viruses, 2017-11, Vol.9 (12), p.362

2017

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Overexpression of the human DEK oncogene reprograms cellular metabolism and promotes glycolysis

PloS one, 2017-05, Vol.12 (5), p.e0177952

2017

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Prevalence of Human Papillomavirus in Oropharyngeal Squamous Cell Carcinoma in the United States Across Time

Chemical research in toxicology, 2014-04, Vol.27 (4), p.462-469

2014

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Mouse papillomavirus type 1 (MmuPV1) DNA is frequently integrated in benign tumors by microhomology-mediated end-joining

PLoS pathogens, 2021-08, Vol.17 (8), p.e1009812-e1009812

2021

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Human papillomavirus E6 triggers upregulation of the antiviral and cancer genomic DNA deaminase APOBEC3B

mBio, 2014-12, Vol.5 (6)

2014

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Differentiation-Dependent KLF4 Expression Promotes Lytic Epstein-Barr Virus Infection in Epithelial Cells

PLoS pathogens, 2015-10, Vol.11 (10), p.e1005195-e1005195

2015

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Differentiation-Dependent LMP1 Expression Is Required for Efficient Lytic Epstein-Barr Virus Reactivation in Epithelial Cells

Journal of virology, 2017-04, Vol.91 (8)

2017

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A Human Papillomavirus-Independent Cervical Cancer Animal Model Reveals Unconventional Mechanisms of Cervical Carcinogenesis

Cell reports (Cambridge), 2019-03, Vol.26 (10), p.2636-2650.e5

2019

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Key aspects of papillomavirus infection influence the host cervicovaginal microbiome in a preclinical murine papillomavirus (MmuPV1) infection model

mBio, 2024-06, Vol.15 (6), p.e0093324

2024

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Fundamental differences in cell cycle deregulation in human papillomavirus-positive and human papillomavirus-negative head/neck and cervical cancers

Cancer research (Chicago, Ill.), 2007-05, Vol.67 (10), p.4605-4619

2007

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Current In Vitro and In Vivo Models to Study MCPyV-Associated MCC

Viruses, 2022-10, Vol.14 (10), p.2204

2022

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ΔNp63α promotes Epstein-Barr virus latency in undifferentiated epithelial cells

PLoS pathogens, 2021-11, Vol.17 (11), p.e1010045-e1010045

2021

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Inactivation of p53 rescues the maintenance of high risk HPV DNA genomes deficient in expression of E6

PLoS pathogens, 2013-10, Vol.9 (10), p.e1003717-e1003717

2013

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MmuPV1 E6 induces cell proliferation and other hallmarks of cancer

mBio, 2023-12, Vol.14 (6), p.e0245823

2023

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Type 1 and Type 2 Epstein-Barr viruses induce proliferation, and inhibit differentiation, in infected telomerase-immortalized normal oral keratinocytes

PLoS pathogens, 2022-10, Vol.18 (10), p.e1010868-e1010868

2022

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Inhibition of TGF-β and NOTCH Signaling by Cutaneous Papillomaviruses

Frontiers in microbiology, 2018-03, Vol.9, p.389-389

2018

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Emerging Prognostic and Predictive Significance of Stress Keratin 17 in HPV-Associated and Non HPV-Associated Human Cancers: A Scoping Review

Viruses, 2023-11, Vol.15 (12), p.2320

2023

Volltextzugriff (PDF)

Tumourigenesis driven by the human papillomavirus type 16 Asian-American e6 variant in a three-dimensional keratinocyte model

PloS one, 2014-07, Vol.9 (7), p.e101540

2014

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Estrogen Receptor-α Suppresses Liver Carcinogenesis and Establishes Sex-Specific Gene Expression

Cancers, 2021-05, Vol.13 (10), p.2355

2021

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Cooperation of genes in HPV16 E6/E7-dependent cervicovaginal carcinogenesis trackable by endoscopy and independent of exogenous estrogens or carcinogens

Carcinogenesis (New York), 2020-11, Vol.41 (11), p.1605-1615

2020

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1 – 25 von 647

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Stress-Induced EGFR Trafficking: Mechanisms, Functions, and Therapeutic Implications

Key Characteristics of Carcinogens as a Basis for Organizing Data on Mechanisms of Carcinogenesis

Establishment of human papillomavirus infection requires cell cycle progression

APOBEC3A functions as a restriction factor of human papillomavirus

Identifying the Target Cells and Mechanisms of Merkel Cell Polyomavirus Infection

Rodent Papillomaviruses

Overexpression of the human DEK oncogene reprograms cellular metabolism and promotes glycolysis

Prevalence of Human Papillomavirus in Oropharyngeal Squamous Cell Carcinoma in the United States Across Time

Mouse papillomavirus type 1 (MmuPV1) DNA is frequently integrated in benign tumors by microhomology-mediated end-joining

Human papillomavirus E6 triggers upregulation of the antiviral and cancer genomic DNA deaminase APOBEC3B

Differentiation-Dependent KLF4 Expression Promotes Lytic Epstein-Barr Virus Infection in Epithelial Cells

Differentiation-Dependent LMP1 Expression Is Required for Efficient Lytic Epstein-Barr Virus Reactivation in Epithelial Cells

A Human Papillomavirus-Independent Cervical Cancer Animal Model Reveals Unconventional Mechanisms of Cervical Carcinogenesis

Key aspects of papillomavirus infection influence the host cervicovaginal microbiome in a preclinical murine papillomavirus (MmuPV1) infection model

Fundamental differences in cell cycle deregulation in human papillomavirus-positive and human papillomavirus-negative head/neck and cervical cancers

Current In Vitro and In Vivo Models to Study MCPyV-Associated MCC

ΔNp63α promotes Epstein-Barr virus latency in undifferentiated epithelial cells

Inactivation of p53 rescues the maintenance of high risk HPV DNA genomes deficient in expression of E6

MmuPV1 E6 induces cell proliferation and other hallmarks of cancer

Type 1 and Type 2 Epstein-Barr viruses induce proliferation, and inhibit differentiation, in infected telomerase-immortalized normal oral keratinocytes

Inhibition of TGF-β and NOTCH Signaling by Cutaneous Papillomaviruses

Emerging Prognostic and Predictive Significance of Stress Keratin 17 in HPV-Associated and Non HPV-Associated Human Cancers: A Scoping Review

Tumourigenesis driven by the human papillomavirus type 16 Asian-American e6 variant in a three-dimensional keratinocyte model

Estrogen Receptor-α Suppresses Liver Carcinogenesis and Establishes Sex-Specific Gene Expression

Cooperation of genes in HPV16 E6/E7-dependent cervicovaginal carcinogenesis trackable by endoscopy and independent of exogenous estrogens or carcinogens

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