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Synaptic and memory dysfunction in a β-amyloid model of early Alzheimer's disease depends on increased formation of ATP-derived extracellular adenosine

Neurobiology of disease, 2019-12, Vol.132, p.104570-104570, Article 104570

2019

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Adenosine A2A Receptors in the Amygdala Control Synaptic Plasticity and Contextual Fear Memory

Neuropsychopharmacology (New York, N.Y.), 2016-11, Vol.41 (12), p.2862-2871

2016

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Inactivation of adenosine A2A receptors reverses working memory deficits at early stages of Huntington's disease models

Neurobiology of disease, 2015-07, Vol.79, p.70-80

2015

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CD73-Mediated Formation of Extracellular Adenosine Is Responsible for Adenosine A2A Receptor-Mediated Control of Fear Memory and Amygdala Plasticity

International journal of molecular sciences, 2022-10, Vol.23 (21), p.12826

2022

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The Interconnected Mechanisms of Oxidative Stress and Neuroinflammation in Epilepsy

Antioxidants, 2022-01, Vol.11 (1), p.157

2022

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Behavioral phenotyping of Parkin-deficient mice: looking for early preclinical features of Parkinson's disease

PloS one, 2014-12, Vol.9 (12), p.e114216

2014

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Adenosine A2b receptors control A1 receptor-mediated inhibition of synaptic transmission in the mouse hippocampus

The European journal of neuroscience, 2015-04, Vol.41 (7), p.878-888

2015

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Increased Synaptic ATP Release and CD73-Mediated Formation of Extracellular Adenosine in the Control of Behavioral and Electrophysiological Modifications Caused by Chronic Stress

ACS chemical neuroscience, 2023-04, Vol.14 (7), p.1299-1309

2023

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Atorvastatin prevents hippocampal cell death, neuroinflammation and oxidative stress following amyloid-β1―40 administration in mice: Evidence for dissociation between cognitive deficits and neuronal damage

Experimental neurology, 2010-12, Vol.226 (2), p.274-284

2010

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Effects of traumatic brain injury of different severities on emotional, cognitive, and oxidative stress-related parameters in mice

Journal of neurotrauma, 2010-10, Vol.27 (10), p.1883-1893

2010

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Animal models of olfactory dysfunction in neurodegenerative diseases

Handbook of Clinical Neurology, 2019, Vol.164, p.431-452

2019

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Effects of acute administration of the hydroalcoholic extract of mate tea leaves ( Ilex paraguariensis) in animal models of learning and memory

Journal of ethnopharmacology, 2008-12, Vol.120 (3), p.465-473

2008

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Ablation of striatal somatostatin interneurons affects MSN morphology and electrophysiological properties, and increases cocaine‐induced hyperlocomotion in mice

The European journal of neuroscience, 2020-03, Vol.51 (6), p.1388-1402

2020

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Optogenetic activation of intracellular adenosine A2A receptor signaling in the hippocampus is sufficient to trigger CREB phosphorylation and impair memory

Molecular psychiatry, 2015-11, Vol.20 (11), p.1339-1349

2015

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Mammalian Target of Rapamycin-RhoA Signaling Impairments in Direct Striatal Projection Neurons Induce Altered Behaviors and Striatal Physiology in Mice

Biological psychiatry (1969), 2020-12, Vol.88 (12), p.945-954

2020

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Adenosine A2A receptors modulate the dopamine D2 receptor‐mediated inhibition of synaptic transmission in the mouse prefrontal cortex

The European journal of neuroscience, 2018-05, Vol.47 (9), p.1127-1134

2018

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Risk is in the Air

Annals of the New York Academy of Sciences, 2009-07, Vol.1170 (1), p.629-636

2009

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GPRIN3 Controls Neuronal Excitability, Morphology, and Striatal-Dependent Behaviors in the Indirect Pathway of the Striatum

The Journal of neuroscience, 2019-09, Vol.39 (38), p.7513-7528

2019

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Depression as a Glial-Based Synaptic Dysfunction

Frontiers in cellular neuroscience, 2016-01, Vol.9, p.521-521

2016

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Increased ATP Release and Higher Impact of Adenosine A2A Receptors on Corticostriatal Plasticity in a Rat Model of Presymptomatic Parkinson’s Disease

Molecular neurobiology, 2023-03, Vol.60 (3), p.1659-1674

2023

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Moderate traumatic brain injury increases the vulnerability to neurotoxicity induced by systemic administration of 6-hydroxydopamine in mice

Brain research, 2017-05, Vol.1663, p.78-86

2017

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The Adenosine Neuromodulation System in Schizophrenia

International Review of Neurobiology, 2014, Vol.119, p.395-449

2014

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Corrigendum to “Atorvastatin prevents hippocampal cell death, neuroinflammation and oxidative stress following amyloid-β1–40 administration in mice: Evidence for dissociation between cognitive deficits and neuronal damage”: [Experimental Neurology, 226:2 (2010) 274–284]

Experimental neurology, 2021-11, Vol.345, p.113840-113840, Article 113840

2021

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Adenosine A 2A receptors modulate the dopamine D 2 receptor-mediated inhibition of synaptic transmission in the mouse prefrontal cortex

The European journal of neuroscience, 2018-05, Vol.47 (9), p.1127-1134

2018

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Temporal Dissociation of Striatum and Prefrontal Cortex Uncouples Anhedonia and Defense Behaviors Relevant to Depression in 6-OHDA-Lesioned Rats

Molecular neurobiology, 2016-08, Vol.53 (6), p.3891-3899

2016

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Synaptic and memory dysfunction in a β-amyloid model of early Alzheimer's disease depends on increased formation of ATP-derived extracellular adenosine

Adenosine A2A Receptors in the Amygdala Control Synaptic Plasticity and Contextual Fear Memory

Inactivation of adenosine A2A receptors reverses working memory deficits at early stages of Huntington's disease models

CD73-Mediated Formation of Extracellular Adenosine Is Responsible for Adenosine A2A Receptor-Mediated Control of Fear Memory and Amygdala Plasticity

The Interconnected Mechanisms of Oxidative Stress and Neuroinflammation in Epilepsy

Behavioral phenotyping of Parkin-deficient mice: looking for early preclinical features of Parkinson's disease

Adenosine A2b receptors control A1 receptor-mediated inhibition of synaptic transmission in the mouse hippocampus

Increased Synaptic ATP Release and CD73-Mediated Formation of Extracellular Adenosine in the Control of Behavioral and Electrophysiological Modifications Caused by Chronic Stress

Atorvastatin prevents hippocampal cell death, neuroinflammation and oxidative stress following amyloid-β1―40 administration in mice: Evidence for dissociation between cognitive deficits and neuronal damage

Effects of traumatic brain injury of different severities on emotional, cognitive, and oxidative stress-related parameters in mice

Animal models of olfactory dysfunction in neurodegenerative diseases

Effects of acute administration of the hydroalcoholic extract of mate tea leaves ( Ilex paraguariensis) in animal models of learning and memory

Ablation of striatal somatostatin interneurons affects MSN morphology and electrophysiological properties, and increases cocaine‐induced hyperlocomotion in mice

Optogenetic activation of intracellular adenosine A2A receptor signaling in the hippocampus is sufficient to trigger CREB phosphorylation and impair memory

Mammalian Target of Rapamycin-RhoA Signaling Impairments in Direct Striatal Projection Neurons Induce Altered Behaviors and Striatal Physiology in Mice

Adenosine A2A receptors modulate the dopamine D2 receptor‐mediated inhibition of synaptic transmission in the mouse prefrontal cortex

Risk is in the Air

GPRIN3 Controls Neuronal Excitability, Morphology, and Striatal-Dependent Behaviors in the Indirect Pathway of the Striatum

Depression as a Glial-Based Synaptic Dysfunction

Increased ATP Release and Higher Impact of Adenosine A2A Receptors on Corticostriatal Plasticity in a Rat Model of Presymptomatic Parkinson’s Disease

Moderate traumatic brain injury increases the vulnerability to neurotoxicity induced by systemic administration of 6-hydroxydopamine in mice

The Adenosine Neuromodulation System in Schizophrenia

Corrigendum to “Atorvastatin prevents hippocampal cell death, neuroinflammation and oxidative stress following amyloid-β1–40 administration in mice: Evidence for dissociation between cognitive deficits and neuronal damage”: [Experimental Neurology, 226:2 (2010) 274–284]

Adenosine A 2A receptors modulate the dopamine D 2 receptor-mediated inhibition of synaptic transmission in the mouse prefrontal cortex

Temporal Dissociation of Striatum and Prefrontal Cortex Uncouples Anhedonia and Defense Behaviors Relevant to Depression in 6-OHDA-Lesioned Rats

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