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The homeobox gene Hhex is essential for proper hepatoblast differentiation and bile duct morphogenesis

Developmental biology, 2007-08, Vol.308 (2), p.355-367

2007

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Biliary epithelial injury-induced regenerative response by IL-33 promotes cholangiocarcinogenesis from peribiliary glands

Proceedings of the National Academy of Sciences - PNAS, 2017-05, Vol.114 (19), p.E3806-E3815

2017

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TGR5 is essential for bile acid-dependent cholangiocyte proliferation in vivo and in vitro

Gut, 2016-03, Vol.65 (3), p.487-501

2016

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A Hippo and Fibroblast Growth Factor Receptor Autocrine Pathway in Cholangiocarcinoma

The Journal of biological chemistry, 2016-04, Vol.291 (15), p.8031-8047

2016

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Cholangiocarcinomas can originate from hepatocytes in mice

The Journal of clinical investigation, 2012-08, Vol.122 (8), p.2911-2915

2012

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Activation of β-Catenin and Yap1 in Human Hepatoblastoma and Induction of Hepatocarcinogenesis in Mice

Gastroenterology (New York, N.Y. 1943), 2014-09, Vol.147 (3), p.690-701

2014

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Foxa1 and Foxa2 regulate bile duct development in mice

The Journal of clinical investigation, 2009-06, Vol.119 (6), p.1537-1545

2009

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Oncogenic Activity of Retinoic Acid Receptor γ Is Exhibited through Activation of the Akt/NF-κB and Wnt/β-Catenin Pathways in Cholangiocarcinoma

Molecular and Cellular Biology, 2013-09, Vol.33 (17), p.3416-3425

2013

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Circ_0007534 promotes cholangiocarcinoma stemness and resistance to anoikis through DDX3X-mediated positive feedback regulation of parental gene DDX42

Cellular signalling, 2024-06, Vol.118, p.111141-111141, Article 111141

2024

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Plasmalemma vesicle-associated protein promotes angiogenesis in cholangiocarcinoma via the DKK1/CKAP4/PI3K signaling pathway

Oncogene, 2021-06, Vol.40 (25), p.4324-4337

2021

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Nobiletin suppresses cholangiocarcinoma proliferation via inhibiting GSK3β

International journal of biological sciences, 2022-01, Vol.18 (15), p.5698-5712

2022

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MUC1 induces the accumulation of Foxp3+ Treg cells in the tumor microenvironment to promote the growth and metastasis of cholangiocarcinoma through the EGFR/PI3K/Akt signaling pathway

International immunopharmacology, 2023-05, Vol.118, p.110091-110091, Article 110091

2023

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Targeting PKM2 improves the gemcitabine sensitivity of intrahepatic cholangiocarcinoma cells via inhibiting β-catenin signaling pathway

Chemico-biological interactions, 2024-01, Vol.387, p.110816-110816, Article 110816

2024

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Peroxiredoxin 6 expression is inversely correlated with nuclear factor-κB activation during Clonorchis sinensis infestation

Free radical biology & medicine, 2016-10, Vol.99, p.273-285

2016

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A Mouse Model of Cholestasis-Associated Cholangiocarcinoma and Transcription Factors Involved in Progression

Gastroenterology (New York, N.Y. 1943), 2011-07, Vol.141 (1), p.378-388.e4

2011

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miR-21 targets 15-PGDH and promotes cholangiocarcinoma growth

Molecular cancer research, 2014-06, Vol.12 (6), p.890-900

2014

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Liensinine inhibits progression of intrahepatic cholangiocarcinoma by regulating TGF‐β1 /P‐smad3 signaling through HIF‐1a

Molecular carcinogenesis, 2024-04, Vol.63 (4), p.772-784

2024

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Guanine nucleotide exchange factor T exerts the cancer-promoting function in cholangiocarcinoma by enhancing the Wnt-GSK-3β-β-catenin cascade via regulation of Rac1/Cdc42

Toxicology and applied pharmacology, 2023-05, Vol.467, p.116492-116492, Article 116492

2023

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TNF receptor-related factor 3 inactivation promotes the development of intrahepatic cholangiocarcinoma through NF-κB-inducing kinase-mediated hepatocyte transdifferentiation

Hepatology (Baltimore, Md.), 2023-02, Vol.77 (2), p.395-n/a

2023

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PLAU contributes to the development of cholangiocarcinoma via activating NF‐κB signaling pathway

Cell biology international, 2023-08, Vol.47 (8), p.1381-1391

2023

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Direct Reprogramming of Hepatocytes Into JAK/Stat-Dependent LGR5+ Liver Cells Able to Initiate Intrahepatic Cholangiocarcinoma

Stem cells (Dayton, Ohio), 2024-04, Vol.42 (4), p.301-316

2024

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MiR-605 represses PSMD10/Gankyrin and inhibits intrahepatic cholangiocarcinoma cell progression

FEBS letters, 2014-09, Vol.588 (18), p.3491-3500

2014

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Animal models of biliary injury and altered bile acid metabolism

Biochimica et biophysica acta. Molecular basis of disease, 2018-04, Vol.1864 (4), p.1254-1261

2018

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Combined targeting of AKT and mTOR using MK‐2206 and RAD001 is synergistic in the treatment of cholangiocarcinoma

International journal of cancer, 2013-11, Vol.133 (9), p.2065-2076

2013

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The homeobox gene Hhex is essential for proper hepatoblast differentiation and bile duct morphogenesis

Biliary epithelial injury-induced regenerative response by IL-33 promotes cholangiocarcinogenesis from peribiliary glands

TGR5 is essential for bile acid-dependent cholangiocyte proliferation in vivo and in vitro

A Hippo and Fibroblast Growth Factor Receptor Autocrine Pathway in Cholangiocarcinoma

Cholangiocarcinomas can originate from hepatocytes in mice

Activation of β-Catenin and Yap1 in Human Hepatoblastoma and Induction of Hepatocarcinogenesis in Mice

Foxa1 and Foxa2 regulate bile duct development in mice

Molecular portraits of patients with intrahepatic cholangiocarcinoma who diverge as rapid progressors or long survivors on chemotherapy

Oncogenic Activity of Retinoic Acid Receptor γ Is Exhibited through Activation of the Akt/NF-κB and Wnt/β-Catenin Pathways in Cholangiocarcinoma

Circ_0007534 promotes cholangiocarcinoma stemness and resistance to anoikis through DDX3X-mediated positive feedback regulation of parental gene DDX42

Plasmalemma vesicle-associated protein promotes angiogenesis in cholangiocarcinoma via the DKK1/CKAP4/PI3K signaling pathway

Nobiletin suppresses cholangiocarcinoma proliferation via inhibiting GSK3β

MUC1 induces the accumulation of Foxp3+ Treg cells in the tumor microenvironment to promote the growth and metastasis of cholangiocarcinoma through the EGFR/PI3K/Akt signaling pathway

Targeting PKM2 improves the gemcitabine sensitivity of intrahepatic cholangiocarcinoma cells via inhibiting β-catenin signaling pathway

Peroxiredoxin 6 expression is inversely correlated with nuclear factor-κB activation during Clonorchis sinensis infestation

A Mouse Model of Cholestasis-Associated Cholangiocarcinoma and Transcription Factors Involved in Progression

miR-21 targets 15-PGDH and promotes cholangiocarcinoma growth

Liensinine inhibits progression of intrahepatic cholangiocarcinoma by regulating TGF‐β1 /P‐smad3 signaling through HIF‐1a

Guanine nucleotide exchange factor T exerts the cancer-promoting function in cholangiocarcinoma by enhancing the Wnt-GSK-3β-β-catenin cascade via regulation of Rac1/Cdc42

TNF receptor-related factor 3 inactivation promotes the development of intrahepatic cholangiocarcinoma through NF-κB-inducing kinase-mediated hepatocyte transdifferentiation

PLAU contributes to the development of cholangiocarcinoma via activating NF‐κB signaling pathway

Direct Reprogramming of Hepatocytes Into JAK/Stat-Dependent LGR5+ Liver Cells Able to Initiate Intrahepatic Cholangiocarcinoma

MiR-605 represses PSMD10/Gankyrin and inhibits intrahepatic cholangiocarcinoma cell progression

Animal models of biliary injury and altered bile acid metabolism

Combined targeting of AKT and mTOR using MK‐2206 and RAD001 is synergistic in the treatment of cholangiocarcinoma

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