UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Circulating mitochondrial DAMPs cause inflammatory responses to injury

Nature, 2010-03, Vol.464 (7285), p.104-107

2010

Volltextzugriff (PDF)

Mitochondrial DAMPs increase endothelial permeability through neutrophil dependent and independent pathways

PloS one, 2013-03, Vol.8 (3), p.e59989-e59989

2013

Volltextzugriff (PDF)

Mitochondrial DNA is released by shock and activates neutrophils via p38 map kinase

Shock (Augusta, Ga.), 2010-07, Vol.34 (1), p.55-59

2010

Volltextzugriff (PDF)

Mitochondrial DNA released by trauma induces neutrophil extracellular traps

PloS one, 2015-03, Vol.10 (3), p.e0120549-e0120549

2015

Volltextzugriff (PDF)

Role of Mitochondria-Derived Danger Signals Released After Injury in Systemic Inflammation and Sepsis

Antioxidants & redox signaling, 2021-11, Vol.35 (15), p.1273-1290

2021

Volltextzugriff (PDF)

Carbon monoxide protects the kidney through the central circadian clock and CD39

Proceedings of the National Academy of Sciences - PNAS, 2018-03, Vol.115 (10), p.E2302-E2310

2018

Volltextzugriff (PDF)

Scavenging Circulating Mitochondrial DNA as a Potential Therapeutic Option for Multiple Organ Dysfunction in Trauma Hemorrhage

Frontiers in immunology, 2018-05, Vol.9, p.891-891

2018

Volltextzugriff (PDF)

Health care and socioeconomic impact of falls in the elderly

The American journal of surgery, 2012-03, Vol.203 (3), p.335-338

2012

Volltextzugriff (PDF)

Mitochondrial DAMPs Are Released During Cardiopulmonary Bypass Surgery and Are Associated With Postoperative Atrial Fibrillation

Heart, lung & circulation, 2018-01, Vol.27 (1), p.122-129

2018

Volltextzugriff (PDF)

Plasma bacterial and mitochondrial DNA distinguish bacterial sepsis from sterile systemic inflammatory response syndrome and quantify inflammatory tissue injury in nonhuman primates

Shock (Augusta, Ga.), 2013-01, Vol.39 (1), p.55-62

2013

Volltextzugriff (PDF)

Mitochondrial Damage Associated Molecular Patterns From Femoral Reamings Activate Neutrophils Through Formyl Peptide Receptors and P44/42 MAP Kinase

Journal of orthopaedic trauma, 2010-09, Vol.24 (9), p.534-538

2010

Volltextzugriff (PDF)

Heme: The Lord of the Iron Ring

Antioxidants, 2023-05, Vol.12 (5), p.1074

2023

Volltextzugriff (PDF)

Sphingosine 1-Phosphate, a Diffusible Calcium Influx Factor Mediating Store-operated Calcium Entry

The Journal of biological chemistry, 2003-07, Vol.278 (30), p.27540-27547

2003

Volltextzugriff (PDF)

Heme as a danger molecule in pathogen recognition

Free radical biology & medicine, 2015-12, Vol.89, p.651-661

2015

Volltextzugriff (PDF)

Trauma-induced heme release increases susceptibility to bacterial infection

JCI insight, 2021-10, Vol.6 (20)

2021

Volltextzugriff (PDF)

Role of Mitochondria-Derived Danger Signals Released After Injury in Systemic Inflammation and Sepsis

Antioxidants & redox signaling, 2021-11, Vol.35 (15), p.1273

2021

Volltextzugriff (PDF)

Bone marrow failure following severe injury in humans

Annals of surgery, 2003-11, Vol.238 (5), p.748-753

2003

Volltextzugriff (PDF)

Neutrophil activation is modulated by sex hormones after trauma-hemorrhagic shock and burn injuries

American journal of physiology. Heart and circulatory physiology, 2006-09, Vol.291 (3), p.H1456-H1465

2006

Volltextzugriff (PDF)

Antiplatelet agents, warfarin, and epidemic intracranial hemorrhage

Surgery, 2010-10, Vol.148 (4), p.724-730

2010

Volltextzugriff (PDF)

Removal of circulating mitochondrial N-formyl peptides via immobilized antibody therapy restores sepsis-induced neutrophil dysfunction

Journal of leukocyte biology, 2024-11, Vol.116 (5), p.1169-1183

2024

Volltextzugriff (PDF)

Extracellular mitochondria drive CD8 T cell dysfunction in trauma by upregulating CD39

Thorax, 2023-02, Vol.78 (2), p.151-159

2023

Volltextzugriff (PDF)

Circulating mitochondrial N-formyl peptides contribute to secondary nosocomial infection in patients with septic shock

Proceedings of the National Academy of Sciences - PNAS, 2021-04, Vol.118 (17), p.1-11

2021

Volltextzugriff (PDF)

DANGER Signals Activate G-Protein Receptor Kinases Suppressing Neutrophil Function and Predisposing to Infection After Tissue Trauma

Annals of surgery, 2023-12

2023

Volltextzugriff (PDF)

Cytomegalovirus durably primes neutrophil oxidative burst

Journal of leukocyte biology, 2023-10, Vol.114 (5), p.459-474

2023

Volltextzugriff (PDF)

1 – 25 von 278

Suche als RSS-Feed

Aktive Filter

Zeitschrift / SerieThe Journal Of Trauma And Acute Care Surgery

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(165)
Mit Volltextzugriff
(225)

Publikationsform

1 weiteren anzeigen...

Weniger anzeigen...

Sprache

Englisch
(271)
Deutsch
(7)

Erscheinungsjahr

n.n

Thema

Science & Technology
(162)
Life Sciences & Biomedicine
(143)
Humans
(130)
Biological And Medical Sciences
(94)
Medical Sciences
(90)
Male
(89)
Surgery
(80)
General & Internal Medicine
(66)
Critical Care Medicine
(63)
Female
(62)
Animals
(48)
Adult
(46)
Anesthesia. Intensive Care Medicine. Transfusions. Cell Therapy And Gene Therapy
(45)
Middle Aged
(36)
Aged
(32)
Traumas. Diseases Due To Physical Agents
(32)
Diseases Of The Osteoarticular System
(31)
Intensive Care Medicine
(30)
Trauma
(28)
Rats
(26)

15 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

Circulating mitochondrial DAMPs cause inflammatory responses to injury

Mitochondrial DAMPs increase endothelial permeability through neutrophil dependent and independent pathways

Mitochondrial DNA is released by shock and activates neutrophils via p38 map kinase

Mitochondrial DNA released by trauma induces neutrophil extracellular traps

Role of Mitochondria-Derived Danger Signals Released After Injury in Systemic Inflammation and Sepsis

Carbon monoxide protects the kidney through the central circadian clock and CD39

Scavenging Circulating Mitochondrial DNA as a Potential Therapeutic Option for Multiple Organ Dysfunction in Trauma Hemorrhage

Health care and socioeconomic impact of falls in the elderly

Mitochondrial DAMPs Are Released During Cardiopulmonary Bypass Surgery and Are Associated With Postoperative Atrial Fibrillation

Plasma bacterial and mitochondrial DNA distinguish bacterial sepsis from sterile systemic inflammatory response syndrome and quantify inflammatory tissue injury in nonhuman primates

Mitochondrial Damage Associated Molecular Patterns From Femoral Reamings Activate Neutrophils Through Formyl Peptide Receptors and P44/42 MAP Kinase

Heme: The Lord of the Iron Ring

Sphingosine 1-Phosphate, a Diffusible Calcium Influx Factor Mediating Store-operated Calcium Entry

Heme as a danger molecule in pathogen recognition

Trauma-induced heme release increases susceptibility to bacterial infection

Role of Mitochondria-Derived Danger Signals Released After Injury in Systemic Inflammation and Sepsis

Bone marrow failure following severe injury in humans

Neutrophil activation is modulated by sex hormones after trauma-hemorrhagic shock and burn injuries

Antiplatelet agents, warfarin, and epidemic intracranial hemorrhage

Removal of circulating mitochondrial N-formyl peptides via immobilized antibody therapy restores sepsis-induced neutrophil dysfunction

Extracellular mitochondria drive CD8 T cell dysfunction in trauma by upregulating CD39

Circulating mitochondrial N-formyl peptides contribute to secondary nosocomial infection in patients with septic shock

DANGER Signals Activate G-Protein Receptor Kinases Suppressing Neutrophil Function and Predisposing to Infection After Tissue Trauma

Mitochondrial DNA in the tumour microenvironment activates neutrophils and is associated with worse outcomes in patients with advanced epithelial ovarian cancer

Cytomegalovirus durably primes neutrophil oxidative burst

Aktive Filter

Suchergebnisse filtern