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Gene expression and functional deficits underlie TREM2-knockout microglia responses in human models of Alzheimer's disease

Nature communications, 2020-10, Vol.11 (1), p.5370-5370, Article 5370

2020

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Nicotinamide Restores Cognition in Alzheimer's Disease Transgenic Mice via a Mechanism Involving Sirtuin Inhibition and Selective Reduction of Thr231-Phosphotau

The Journal of neuroscience, 2008-11, Vol.28 (45), p.11500-11510

2008

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Ganglioside GM1 induces phosphorylation of mutant huntingtin and restores normal motor behavior in Huntington disease mice

Proceedings of the National Academy of Sciences - PNAS, 2012-02, Vol.109 (9), p.3528-3533

2012

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Inhibition of specific HDACs and sirtuins suppresses pathogenesis in a Drosophila model of Huntington’s disease

Human molecular genetics, 2008-12, Vol.17 (23), p.3767-3775

2008

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SUMO-2 and PIAS1 Modulate Insoluble Mutant Huntingtin Protein Accumulation

Cell reports (Cambridge), 2013-07, Vol.4 (2), p.362-375

2013

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Author Correction: Gene expression and functional deficits underlie TREM2-knockout microglia responses in human models of Alzheimer's disease

Nature communications, 2023-03, Vol.14 (1), p.1194-1194, Article 1194

2023

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The first 17 amino acids of Huntingtin modulate its sub-cellular localization, aggregation and effects on calcium homeostasis

Human molecular genetics, 2007-01, Vol.16 (1), p.61-77

2007

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APOE4 dysregulates autophagy in cultured cells

Autophagy reports, 2022-01, Vol.1 (1), p.29-33

2022

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Suberoylanilide Hydroxamic Acid, a Histone Deacetylase Inhibitor, Ameliorates Motor Deficits in a Mouse Model of Huntington's Disease

Proceedings of the National Academy of Sciences - PNAS, 2003-02, Vol.100 (4), p.2041-2046

2003

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Cooperation of cell adhesion and autophagy in the brain: Functional roles in development and neurodegenerative disease

Matrix biology plus, 2021-12, Vol.12, p.100089-100089, Article 100089

2021

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Selective histone deacetylase (HDAC) inhibition imparts beneficial effects in Huntington's disease mice: implications for the ubiquitin―proteasomal and autophagy systems

Human molecular genetics, 2012-12, Vol.21 (24), p.5280-5293

2012

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Serine residues 13 and 16 are key modulators of mutant huntingtin induced toxicity in Drosophila

Experimental neurology, 2021-04, Vol.338, p.113463, Article 113463

2021

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Isoform-dependent lysosomal degradation and internalization of apolipoprotein E requires autophagy proteins

Journal of cell science, 2022-01, Vol.135 (2)

2022

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PIAS1 modulates striatal transcription, DNA damage repair, and SUMOylation with relevance to Huntington's disease

Proceedings of the National Academy of Sciences - PNAS, 2021-01, Vol.118 (4)

2021

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IKKβ slows Huntington’s disease progression in R6/1 mice

Proceedings of the National Academy of Sciences - PNAS, 2019-05, Vol.116 (22), p.10952-10961

2019

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Diminished LC3-Associated Phagocytosis by Huntington's Disease Striatal Astrocytes

Journal of Huntington's disease, 2022-01, Vol.11 (1), p.25

2022

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Autophagy researchers

Autophagy, 2014-02, Vol.10 (2), p.188-191

2014

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Deletion of the huntingtin proline-rich region does not significantly affect normal huntingtin function in mice

Journal of Huntington's disease, 2012, Vol.1 (1), p.71

2012

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Targeting aggregation in the development of therapeutics for the treatment of Huntington's disease and other polyglutamine repeat diseases

Expert opinion on therapeutic targets, 2003-04, Vol.7 (2), p.201-213

2003

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Gene sequence and primary structure of mitochondrial malate dehydrogenase from Saccharomyces cerevisiae

Biochemistry (Easton), 1988-11, Vol.27 (22), p.8393-8400

1988

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Interaction of TATA-Binding Protein with Upstream Activation Factor Is Required for Activated Transcription of Ribosomal DNA by RNA Polymerase I in Saccharomyces cerevisiae In Vivo

Molecular and Cellular Biology, 1998-07, Vol.18 (7), p.3752-3761

1998

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Does Huntingtin play a role in selective macroautophagy?

Cell cycle (Georgetown, Tex.), 2010-09, Vol.9 (17), p.3401-3413

Steffan, Joan S

2010

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The role of TBP in rDNA transcription by RNA polymerase I in Saccharomyces cerevisiae: TBP is required for upstream activation factor-dependent recruitment of core factor

Genes & development, 1996-10, Vol.10 (20), p.2551-2563

1996

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RRN6 and RRN7 encode subunits of a multiprotein complex essential for the initiation of rDNA transcription by RNA polymerase I in Saccharomyces cerevisiae

Genes & development, 1994-10, Vol.8 (19), p.2349-2362

1994

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Gene expression and functional deficits underlie TREM2-knockout microglia responses in human models of Alzheimer's disease

Nicotinamide Restores Cognition in Alzheimer's Disease Transgenic Mice via a Mechanism Involving Sirtuin Inhibition and Selective Reduction of Thr231-Phosphotau

Ganglioside GM1 induces phosphorylation of mutant huntingtin and restores normal motor behavior in Huntington disease mice

Inhibition of specific HDACs and sirtuins suppresses pathogenesis in a Drosophila model of Huntington’s disease

SUMO-2 and PIAS1 Modulate Insoluble Mutant Huntingtin Protein Accumulation

Author Correction: Gene expression and functional deficits underlie TREM2-knockout microglia responses in human models of Alzheimer's disease

The first 17 amino acids of Huntingtin modulate its sub-cellular localization, aggregation and effects on calcium homeostasis

APOE4 dysregulates autophagy in cultured cells

Human Neural Stem Cell Transplantation Rescues Functional Deficits in R6/2 and Q140 Huntington's Disease Mice

Suberoylanilide Hydroxamic Acid, a Histone Deacetylase Inhibitor, Ameliorates Motor Deficits in a Mouse Model of Huntington's Disease

Cooperation of cell adhesion and autophagy in the brain: Functional roles in development and neurodegenerative disease

Selective histone deacetylase (HDAC) inhibition imparts beneficial effects in Huntington's disease mice: implications for the ubiquitin―proteasomal and autophagy systems

Serine residues 13 and 16 are key modulators of mutant huntingtin induced toxicity in Drosophila

Isoform-dependent lysosomal degradation and internalization of apolipoprotein E requires autophagy proteins

PIAS1 modulates striatal transcription, DNA damage repair, and SUMOylation with relevance to Huntington's disease

IKKβ slows Huntington’s disease progression in R6/1 mice

Diminished LC3-Associated Phagocytosis by Huntington's Disease Striatal Astrocytes

Autophagy researchers

Deletion of the huntingtin proline-rich region does not significantly affect normal huntingtin function in mice

Targeting aggregation in the development of therapeutics for the treatment of Huntington's disease and other polyglutamine repeat diseases

Gene sequence and primary structure of mitochondrial malate dehydrogenase from Saccharomyces cerevisiae

Interaction of TATA-Binding Protein with Upstream Activation Factor Is Required for Activated Transcription of Ribosomal DNA by RNA Polymerase I in Saccharomyces cerevisiae In Vivo

Does Huntingtin play a role in selective macroautophagy?

The role of TBP in rDNA transcription by RNA polymerase I in Saccharomyces cerevisiae: TBP is required for upstream activation factor-dependent recruitment of core factor

RRN6 and RRN7 encode subunits of a multiprotein complex essential for the initiation of rDNA transcription by RNA polymerase I in Saccharomyces cerevisiae

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