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Oncology Products in the European Union: An Analysis of Regulatory Approvals with a CHMP Oral Explanation

Therapeutic innovation & regulatory science, 2021-09, Vol.55 (5), p.1036-1044

2021

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Regulation of the Rat Serotonin-1A Receptor Gene by Corticosteroids

The Journal of biological chemistry, 2000-01, Vol.275 (2), p.1321-1326

2000

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Histology independent drug development – Is this the future for cancer drugs?

Cancer treatment reviews, 2024-02, Vol.123, p.102674-102674, Article 102674

2024

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Negative Interaction between the RelA(p65) Subunit of NF-κB and the Progesterone Receptor

The Journal of biological chemistry, 1996-03, Vol.271 (11), p.6217-6224

1996

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Negative cross-talk between RelA and the glucocorticoid receptor: a possible mechanism for the antiinflammatory action of glucocorticoids

Molecular endocrinology (Baltimore, Md.), 1995-04, Vol.9 (4), p.401-412

1995

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4-Hydroxytamoxifen Trans-Represses Nuclear Factor-κB Activity in Human Osteoblastic U2-OS Cells through Estrogen Receptor (ER)α, and Not through ERβ

Endocrinology (Philadelphia), 2001-03, Vol.142 (3), p.1156-1166

2001

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Distinct Domains of the RelA NF-κB Subunit Are Required for Negative Cross-talk and Direct Interaction with the Glucocorticoid Receptor

The Journal of biological chemistry, 1997-08, Vol.272 (35), p.22278-22284

1997

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Cloning and expression during development of three murine members of the COUP family of nuclear orphan receptors

Mechanisms of development, 1994-07, Vol.47 (1), p.81-97

1994

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Nuclear Factor-κ B Repression in Antiinflammation and Immunosuppression by Glucocorticoids

Trends in endocrinology and metabolism, 1997-05, Vol.8 (4), p.152-157

1997

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NF-κB/Rel Family Members Regulating the ICAM-1 Promoter in Monocytic THP-1 Cells

Immunobiology (1979), 1997-12, Vol.198 (1), p.50-64

1997

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4-Hydroxytamoxifen Trans-Represses Nuclear Factor-κB Activity in Human Osteoblastic U2-OS Cells through Estrogen Receptor (ER)α, and Not through ERβ1

Endocrinology (Philadelphia), 2001-03, Vol.142 (3), p.1156-1166

2001

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Synergistic Activation of the Serotonin-1A Receptor by Nuclear Factor-κB and Estrogen

Molecular endocrinology (Baltimore, Md.), 2001-04, Vol.15 (4), p.543-552

2001

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Isolation and Developmental Expression of Retinoic-Acid-Induced Genes

Developmental biology, 1994-02, Vol.161 (2), p.604-614

1994

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PHOSPHOLIPASE A2INHIBITORS AND LEUKOTRIENE SYNTHESIS INHIBITORS BLOCK TNF-INDUCED NF-κB ACTIVATION

Cytokine (Philadelphia, Pa.), 1999-02, Vol.11 (2), p.104-110

1999

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Negative Interaction between the RelA(p65) Subunit of NF-B and the Progesterone Receptor

The Journal of biological chemistry, 1996-03, Vol.271 (11), p.6217

1996

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Distinct Domains of the RelA NF-ÎºB Subunit Are Required for Negative Cross-talk and Direct Interaction with the Glucocorticoid Receptor

The Journal of biological chemistry, 1997-08, Vol.272 (35), p.22278

1997

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Oncology Products in the European Union: An Analysis of Regulatory Approvals with a CHMP Oral Explanation

Regulation of the Rat Serotonin-1A Receptor Gene by Corticosteroids

Histology independent drug development – Is this the future for cancer drugs?

Designing drug trials for Alzheimer’s disease: What we have learned from the release of the phase III antibody trials: A report from the EU/US/CTAD Task Force

Negative Interaction between the RelA(p65) Subunit of NF-κB and the Progesterone Receptor

Negative cross-talk between RelA and the glucocorticoid receptor: a possible mechanism for the antiinflammatory action of glucocorticoids

4-Hydroxytamoxifen Trans-Represses Nuclear Factor-κB Activity in Human Osteoblastic U2-OS Cells through Estrogen Receptor (ER)α, and Not through ERβ

Distinct Domains of the RelA NF-κB Subunit Are Required for Negative Cross-talk and Direct Interaction with the Glucocorticoid Receptor

Cloning and expression during development of three murine members of the COUP family of nuclear orphan receptors

Nuclear Factor-κ B Repression in Antiinflammation and Immunosuppression by Glucocorticoids

NF-κB/Rel Family Members Regulating the ICAM-1 Promoter in Monocytic THP-1 Cells

4-Hydroxytamoxifen Trans-Represses Nuclear Factor-κB Activity in Human Osteoblastic U2-OS Cells through Estrogen Receptor (ER)α, and Not through ERβ1

Synergistic Activation of the Serotonin-1A Receptor by Nuclear Factor-κB and Estrogen

Isolation and Developmental Expression of Retinoic-Acid-Induced Genes

PHOSPHOLIPASE A2INHIBITORS AND LEUKOTRIENE SYNTHESIS INHIBITORS BLOCK TNF-INDUCED NF-κB ACTIVATION

Negative Interaction between the RelA(p65) Subunit of NF-B and the Progesterone Receptor

Distinct Domains of the RelA NF-ÎºB Subunit Are Required for Negative Cross-talk and Direct Interaction with the Glucocorticoid Receptor

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